Pain Progression at Initiation of Cabazitaxel in Metastatic Castration-Resistant Prostate Cancer (mCRPC): A Post Hoc Analysis of the PROSELICA Study.

cabazitaxel chemotherapy clinical progression metastatic castration-resistant prostate cancer pain taxanes type of progression

Journal

Cancers
ISSN: 2072-6694
Titre abrégé: Cancers (Basel)
Pays: Switzerland
ID NLM: 101526829

Informations de publication

Date de publication:
13 Mar 2021
Historique:
received: 08 02 2021
revised: 03 03 2021
accepted: 07 03 2021
entrez: 3 4 2021
pubmed: 4 4 2021
medline: 4 4 2021
Statut: epublish

Résumé

In the PROSELICA phase III trial (NCT01308580), cabazitaxel 20 mg/m Progression type at randomization was defined as follows: PSA progression only (PSA-p; no radiological progression (RADIO-p), no pain), RADIO-p (±PSA-p, no pain), or pain progression (PAIN-p, ±PSA-p, ±RADIO-p). Relationships between progression type and overall survival (OS), radiological progression-free survival (rPFS), and PSA response (confirmed PSA decrease ≥ 50%) were analyzed. All randomized patients ( This post hoc analysis of the PROSELICA phase III study shows that pain progression at initiation of CABA in mCRPC patients previously treated with DOC is associated with a poor prognosis. Disease progression should be carefully monitored, even in the absence of PSA rise.

Sections du résumé

BACKGROUND BACKGROUND
In the PROSELICA phase III trial (NCT01308580), cabazitaxel 20 mg/m
METHODS METHODS
Progression type at randomization was defined as follows: PSA progression only (PSA-p; no radiological progression (RADIO-p), no pain), RADIO-p (±PSA-p, no pain), or pain progression (PAIN-p, ±PSA-p, ±RADIO-p). Relationships between progression type and overall survival (OS), radiological progression-free survival (rPFS), and PSA response (confirmed PSA decrease ≥ 50%) were analyzed.
RESULTS RESULTS
All randomized patients (
CONCLUSIONS CONCLUSIONS
This post hoc analysis of the PROSELICA phase III study shows that pain progression at initiation of CABA in mCRPC patients previously treated with DOC is associated with a poor prognosis. Disease progression should be carefully monitored, even in the absence of PSA rise.

Identifiants

pubmed: 33805793
pii: cancers13061284
doi: 10.3390/cancers13061284
pmc: PMC8002173
pii:
doi:

Types de publication

Journal Article

Langues

eng

Subventions

Organisme : Sanofi
ID : NA

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Auteurs

Nicolas Delanoy (N)

Medical Oncology, Université de Paris, 75015 Paris, France.
Medical Oncology, AP-HP Paris, Centre, Georges Pompidou European Hospital, 75015 Paris, France.

Debbie Robbrecht (D)

Medical Oncology, Erasmus University Medical Center, 3000 CA Rotterdam, The Netherlands.

Mario Eisenberger (M)

Medical Oncology, The Sidney Kimmel Comprehensive Cancer Center, Johns Hopkins University, Baltimore, MD 21231, USA.

Oliver Sartor (O)

Medicine and Urology, Tulane Cancer Center, New Orleans, LA 70112, USA.

Ronald de Wit (R)

Medical Oncology, Erasmus University Medical Center, 3000 CA Rotterdam, The Netherlands.

Florence Mercier (F)

Stat Process, Boulevard de Sébastopol, 75003 Paris, France.

Christine Geffriaud-Ricouard (C)

Sanofi, Europe Medical Oncology, 75008 Paris, France.

Johann de Bono (J)

The Institute of Cancer Research, London SM2 5NG, UK.
Royal Marsden NHS Foundation Trust, London SW3 6JJ, UK.

Stéphane Oudard (S)

Medical Oncology, Université de Paris, 75015 Paris, France.
Medical Oncology, AP-HP Paris, Centre, Georges Pompidou European Hospital, 75015 Paris, France.

Classifications MeSH