Pathogenic and therapeutic roles of cytokines in acute myeloid leukemia.


Journal

Cytokine
ISSN: 1096-0023
Titre abrégé: Cytokine
Pays: England
ID NLM: 9005353

Informations de publication

Date de publication:
06 2021
Historique:
received: 28 10 2020
revised: 14 03 2021
accepted: 15 03 2021
pubmed: 4 4 2021
medline: 19 1 2022
entrez: 3 4 2021
Statut: ppublish

Résumé

Acute myeloid leukemia (AML) is a heterogeneous disease with high mortality that accounts for the most common acute leukemia in adults. Despite all progress in the therapeutic strategies and increased rate of complete remission, many patients will eventually relapse and die from the disease. Cytokines as molecular messengers play a pivotal role in the immune system. The imbalance release of cytokine has been shown to exert a significant influence on the progression of hematopoietic malignancies including acute myeloid leukemia. This article aimed to summarize current knowledge about cytokines and their critical roles in the pathogenesis, treatment, and survival of AML patients.

Identifiants

pubmed: 33810945
pii: S1043-4666(21)00088-0
doi: 10.1016/j.cyto.2021.155508
pii:
doi:

Substances chimiques

Cytokines 0
Immune Checkpoint Inhibitors 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

155508

Informations de copyright

Copyright © 2021 Elsevier Ltd. All rights reserved.

Auteurs

Omid Karimdadi Sariani (O)

Department of Genetics, College of Science, Islamic Azad University, Kazerun Branch, Kazerun, Iran.

Sara Eghbalpour (S)

School of Medicine, Iran University of Medical Science, Tehran, Iran.

Elahe Kazemi (E)

Biosensor Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Kimia Rafiei Buzhani (K)

School of Medicine, Kermanshah University of Medical Science, Kermanshah, Iran.

Farhad Zaker (F)

Department of Hematology and Blood Banking, Faculty of Allied Medicine, Iran University of Medical Sciences, Tehran, Iran. Electronic address: farhadz20@yahoo.co.uk.

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Classifications MeSH