Reduced T cell immunity in unmedicated, comorbidity-free obsessive-compulsive disorder: An immunophenotyping study.

Autoimmunity Immunophenotyping Inflammation Obsessive-compulsive disorder T cells T regulatory cells

Journal

Journal of psychiatric research
ISSN: 1879-1379
Titre abrégé: J Psychiatr Res
Pays: England
ID NLM: 0376331

Informations de publication

Date de publication:
05 2021
Historique:
received: 14 10 2020
revised: 11 02 2021
accepted: 19 03 2021
pubmed: 5 4 2021
medline: 21 5 2021
entrez: 4 4 2021
Statut: ppublish

Résumé

Immune system aberrations have been postulated to play a role in the pathophysiology of Obsessive-compulsive disorder (OCD). This study was aimed to examine the profile of immune cell subsets in peripheral blood of un-medicated OCD patients. Thirteen drug-naïve/free OCD patients and twenty-six age & sex matched healthy controls were recruited. Immunophenotyping was carried out by staining the whole blood specimens with fluorescent monoclonal antibodies against the cell surface markers such as CD45, CD3, CD16, CD56, CD8, CD4, CD28, CD25 and CD127, followed by data acquisition on BD FACSVerse™ flow cytometer. The proportions of CD4 and CD8 T cells; T regulatory (Tregs), Natural Killer (NK) cells and NK-T cells were compared between patients with OCD and healthy control subjects. Significantly reduced percentage of T regulatory (Treg) cells was observed in individuals with OCD compared to healthy control subjects [1.0 ± 0.7 vs. 1.9 ± 1.4; p = 0.03, r = 0.33]. Treg cells play a crucial role in regulating the immune response, especially by suppressing the functional activities of T cells. In this study, decreased population of Treg cells essentially indicates a dysregulated T cell and/or T cell mediated immune activation in drug-naïve OCD patients. This preliminary observation might form the basis of further studies examining the immuno-inflammatory/autoimmune origin of OCD.

Sections du résumé

BACKGROUND
Immune system aberrations have been postulated to play a role in the pathophysiology of Obsessive-compulsive disorder (OCD). This study was aimed to examine the profile of immune cell subsets in peripheral blood of un-medicated OCD patients.
METHOD
Thirteen drug-naïve/free OCD patients and twenty-six age & sex matched healthy controls were recruited. Immunophenotyping was carried out by staining the whole blood specimens with fluorescent monoclonal antibodies against the cell surface markers such as CD45, CD3, CD16, CD56, CD8, CD4, CD28, CD25 and CD127, followed by data acquisition on BD FACSVerse™ flow cytometer. The proportions of CD4 and CD8 T cells; T regulatory (Tregs), Natural Killer (NK) cells and NK-T cells were compared between patients with OCD and healthy control subjects.
RESULTS
Significantly reduced percentage of T regulatory (Treg) cells was observed in individuals with OCD compared to healthy control subjects [1.0 ± 0.7 vs. 1.9 ± 1.4; p = 0.03, r = 0.33].
CONCLUSION
Treg cells play a crucial role in regulating the immune response, especially by suppressing the functional activities of T cells. In this study, decreased population of Treg cells essentially indicates a dysregulated T cell and/or T cell mediated immune activation in drug-naïve OCD patients. This preliminary observation might form the basis of further studies examining the immuno-inflammatory/autoimmune origin of OCD.

Identifiants

pubmed: 33813311
pii: S0022-3956(21)00185-0
doi: 10.1016/j.jpsychires.2021.03.035
pii:
doi:

Substances chimiques

Pharmaceutical Preparations 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

521-524

Informations de copyright

Copyright © 2021 Elsevier Ltd. All rights reserved.

Auteurs

Manjula Subbanna (M)

Department of Human Genetics, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India; Translational Psychiatry Laboratory, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India.

Venkataram Shivakumar (V)

Department of Integrative Medicine, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India; Translational Psychiatry Laboratory, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India. Electronic address: drshiv.nimhans@gmail.com.

Dania Jose (D)

Translational Psychiatry Laboratory, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India.

Manjunath Venkataswamy (M)

Department of Neurovirology, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India.

Monojit Debnath (M)

Department of Human Genetics, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India.

Vasanthapuram Ravi (V)

Department of Neurovirology, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India.

Y C Janardhan Reddy (YCJ)

OCD Clinic, Department of Psychiatry, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India.

Ganesan Venkatasubramanian (G)

Translational Psychiatry Laboratory, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India; OCD Clinic, Department of Psychiatry, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India.

Janardhanan C Narayanaswamy (JC)

Translational Psychiatry Laboratory, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India; OCD Clinic, Department of Psychiatry, National Institute of Mental Health and Neurosciences (NIMHANS), Bengaluru-560029, India.

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