Crocetin suppresses the growth and migration in HCT-116 human colorectal cancer cells by activating the p-38 MAPK signaling pathway.

Crocetin HCT-116 cells Matrix metalloproteinase 9 Vascular endothelial growth factor p38-mitogen activated protein kinase

Journal

Research in pharmaceutical sciences
ISSN: 1735-5362
Titre abrégé: Res Pharm Sci
Pays: Iran
ID NLM: 101516968

Informations de publication

Date de publication:
Dec 2020
Historique:
received: 29 11 2019
revised: 09 05 2020
accepted: 14 11 2020
entrez: 8 4 2021
pubmed: 9 4 2021
medline: 9 4 2021
Statut: epublish

Résumé

Crocetin is a natural antioxidant that is found in the crocus flower and HCT-116 cells were treated with different concentrations (0, 200, 400, 600, and 800 μM) of crocetin for 24 h. The cell survival rate was measured by MTT assay. Cell migration capacity was evaluated using the wound healing assay. The expression levels of vascular endothelial growth factor (VEGF) and matrix metalloproteinase (MMP-9) was monitored by RT-PCR. Phosphorylation of focal adhesion kinase (FAK) and p38 mitogen-activated protein kinase (MAPK) was determined using western blot. The proliferation of HCT-116 was inhibited by crocetin at 800 μM ( These data suggested that crocetin-induced growth- and migration- suppressing effects on HCT-116 cells may partially depend on the regulation of the p38 (MAPK) signaling pathway.

Sections du résumé

BACKGROUND AND PURPOSE OBJECTIVE
Crocetin is a natural antioxidant that is found in the crocus flower and
EXPERIMENTAL APPROACH METHODS
HCT-116 cells were treated with different concentrations (0, 200, 400, 600, and 800 μM) of crocetin for 24 h. The cell survival rate was measured by MTT assay. Cell migration capacity was evaluated using the wound healing assay. The expression levels of vascular endothelial growth factor (VEGF) and matrix metalloproteinase (MMP-9) was monitored by RT-PCR. Phosphorylation of focal adhesion kinase (FAK) and p38 mitogen-activated protein kinase (MAPK) was determined using western blot.
FINDINGS/RESULTS RESULTS
The proliferation of HCT-116 was inhibited by crocetin at 800 μM (
CONCLUSION AND IMPLICATION CONCLUSIONS
These data suggested that crocetin-induced growth- and migration- suppressing effects on HCT-116 cells may partially depend on the regulation of the p38 (MAPK) signaling pathway.

Identifiants

pubmed: 33828602
doi: 10.4103/1735-5362.301344
pii: RPS-15-592
pmc: PMC8020854
doi:

Types de publication

Journal Article

Langues

eng

Pagination

592-601

Informations de copyright

Copyright: © 2020 Research in Pharmaceutical Sciences.

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Auteurs

Esmaeil Khajeh (E)

Department of Biochemistry, Faculty of Medicine, Urmia University of Medical Sciences, Urmia, I.R. Iran.

Yousef Rasmi (Y)

Department of Biochemistry, Faculty of Medicine, Urmia University of Medical Sciences, Urmia, I.R. Iran.
Cellular and Molecular Research Center, Urmia University of Medical Sciences, Urmia, I.R. Iran.

Fatemeh Kheradmand (F)

Department of Biochemistry, Faculty of Medicine, Urmia University of Medical Sciences, Urmia, I.R. Iran.

Hassan Malekinejad (H)

Department of Pharmacology and Toxicology, Faculty of Pharmacy, Urmia University of Medical Sciences, Urmia, I.R. Iran.

Pornanong Aramwit (P)

Department of Pharmacy Practice, Faculty of Pharmaceutical Sciences and Center of Excellence in Bioactive Resources for Innovative Clinical Applications, Chulalongkorn University, Bangkok, 10330 Thailand.

Ehsan Saboory (E)

Zanjan Metabolic Diseases Research Center, Zanjan University of Medical Sciences, Zanjan, I.R. Iran.

Behrokh Daeihassani (B)

Department of Biology, Payame Noor University, Tehran, I.R. Iran.

Mahdieh Nasirzadeh (M)

Department of Biochemistry, Faculty of Medicine, Urmia University of Medical Sciences, Urmia, I.R. Iran.

Classifications MeSH