Viral Sequencing to Investigate Sources of SARS-CoV-2 Infection in US Healthcare Personnel.

SARS-CoV-2 healthcare personnel infection control precision epidemiology viral sequencing

Journal

Clinical infectious diseases : an official publication of the Infectious Diseases Society of America
ISSN: 1537-6591
Titre abrégé: Clin Infect Dis
Pays: United States
ID NLM: 9203213

Informations de publication

Date de publication:
15 09 2021
Historique:
received: 15 02 2021
pubmed: 16 4 2021
medline: 24 9 2021
entrez: 15 4 2021
Statut: ppublish

Résumé

Healthcare personnel (HCP) are at increased risk of infection with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). We posit that current infection control guidelines generally protect HCP from SARS-CoV-2 infection in a healthcare setting. In this retrospective case series, we used viral genomics to investigate the likely source of SARS-CoV-2 infection in HCP at a major academic medical institution in the Upper Midwest of the United States between 25 March and 27 December 2020. We obtained limited epidemiological data through informal interviews and review of the electronic health record and combined this information with healthcare-associated viral sequences and viral sequences collected in the broader community to infer the most likely source of infection in HCP. We investigated SARS-CoV-2 infection clusters involving 95 HCP and 137 possible patient contact sequences. The majority of HCP infections could not be linked to a patient or coworker (55 of 95 [57.9%]) and were genetically similar to viruses circulating concurrently in the community. We found that 10.5% of HCP infections (10 of 95) could be traced to a coworker. Strikingly, only 4.2% (4 of 95) could be traced to a patient source. Infections among HCP add further strain to the healthcare system and put patients, HCP, and communities at risk. We found no evidence for healthcare-associated transmission in the majority of HCP infections evaluated. Although we cannot rule out the possibility of cryptic healthcare-associated transmission, it appears that HCP most commonly become infected with SARS-CoV-2 via community exposure. This emphasizes the ongoing importance of mask wearing, physical distancing, robust testing programs, and rapid distribution of vaccines.

Sections du résumé

BACKGROUND
Healthcare personnel (HCP) are at increased risk of infection with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). We posit that current infection control guidelines generally protect HCP from SARS-CoV-2 infection in a healthcare setting.
METHODS
In this retrospective case series, we used viral genomics to investigate the likely source of SARS-CoV-2 infection in HCP at a major academic medical institution in the Upper Midwest of the United States between 25 March and 27 December 2020. We obtained limited epidemiological data through informal interviews and review of the electronic health record and combined this information with healthcare-associated viral sequences and viral sequences collected in the broader community to infer the most likely source of infection in HCP.
RESULTS
We investigated SARS-CoV-2 infection clusters involving 95 HCP and 137 possible patient contact sequences. The majority of HCP infections could not be linked to a patient or coworker (55 of 95 [57.9%]) and were genetically similar to viruses circulating concurrently in the community. We found that 10.5% of HCP infections (10 of 95) could be traced to a coworker. Strikingly, only 4.2% (4 of 95) could be traced to a patient source.
CONCLUSIONS
Infections among HCP add further strain to the healthcare system and put patients, HCP, and communities at risk. We found no evidence for healthcare-associated transmission in the majority of HCP infections evaluated. Although we cannot rule out the possibility of cryptic healthcare-associated transmission, it appears that HCP most commonly become infected with SARS-CoV-2 via community exposure. This emphasizes the ongoing importance of mask wearing, physical distancing, robust testing programs, and rapid distribution of vaccines.

Identifiants

pubmed: 33857303
pii: 6226897
doi: 10.1093/cid/ciab281
pmc: PMC8083259
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e1329-e1336

Subventions

Organisme : NIAID NIH HHS
ID : DP2 AI144244
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM008692
Pays : United States
Organisme : NLM NIH HHS
ID : T15 LM007359
Pays : United States
Organisme : National Institute of Allergy and Infectious Diseases
ID : 1DP2AI144244-01
Organisme : NIAID NIH HHS
ID : F30 AI145182
Pays : United States

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press for the Infectious Diseases Society of America. All rights reserved. For permissions, e-mail: journals.permissions@oup.com.

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Auteurs

Katarina M Braun (KM)

Department of Pathobiological Sciences, University of Wisconsin-Madison, Madison, Wisconsin, USA.

Gage K Moreno (GK)

Department of Pathology and Laboratory Medicine, University of Wisconsin-Madison, Madison, Wisconsin, USA.

Ashley Buys (A)

University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.

Elizabeth D Somsen (ED)

Department of Pathology and Laboratory Medicine, University of Wisconsin-Madison, Madison, Wisconsin, USA.

Max Bobholz (M)

Department of Pathology and Laboratory Medicine, University of Wisconsin-Madison, Madison, Wisconsin, USA.

Molly A Accola (MA)

University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.
William S. Middleton Memorial Veterans Hospital, Madison, Wisconsin, USA.

Laura Anderson (L)

University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.
William S. Middleton Memorial Veterans Hospital, Madison, Wisconsin, USA.

William M Rehrauer (WM)

University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.
William S. Middleton Memorial Veterans Hospital, Madison, Wisconsin, USA.

David A Baker (DA)

Department of Pathology and Laboratory Medicine, University of Wisconsin-Madison, Madison, Wisconsin, USA.

Nasia Safdar (N)

Department of Medicine, Division of Infectious Diseases, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.

Alexander J Lepak (AJ)

Department of Medicine, Division of Infectious Diseases, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin, USA.

David H O'Connor (DH)

Department of Pathology and Laboratory Medicine, University of Wisconsin-Madison, Madison, Wisconsin, USA.
Wisconsin National Primate Research Center, University of Wisconsin-Madison, Madison, Wisconsin, USA.

Thomas C Friedrich (TC)

Department of Pathobiological Sciences, University of Wisconsin-Madison, Madison, Wisconsin, USA.
Wisconsin National Primate Research Center, University of Wisconsin-Madison, Madison, Wisconsin, USA.

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