Evidence of the pathogenic HERV-W envelope expression in T lymphocytes in association with the respiratory outcome of COVID-19 patients.


Journal

EBioMedicine
ISSN: 2352-3964
Titre abrégé: EBioMedicine
Pays: Netherlands
ID NLM: 101647039

Informations de publication

Date de publication:
Apr 2021
Historique:
received: 18 01 2021
revised: 26 03 2021
accepted: 30 03 2021
pubmed: 20 4 2021
medline: 18 5 2021
entrez: 19 4 2021
Statut: ppublish

Résumé

Despite an impressive effort in clinical research, no standard therapeutic approach for coronavirus disease 2019 (COVID-19) patients has been established, highlighting the need to identify early biomarkers for predicting disease progression and new therapeutic interventions for patient management. The present study aimed to evaluate the involvement of the human endogenous retrovirus -W envelope (HERV-W ENV) in severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection considering recent findings that HERVs are activated in response to infectious agents and lead to various immunopathological effects. We analysed HERV-W ENV expression in blood cells of COVID-19 patients in correlation with clinical characteristics and have discussed its potential role in the outcome of the disease. We analysed HERV-W ENV expression in blood samples of COVID-19 patients and healthy donors by flow cytometry and quantitative reverse transcriptase PCR analysis, and evaluated its correlation with clinical signs, inflammatory markers, cytokine expression, and disease progression. HERV-W ENV was highly expressed in the leukocytes of COVID-19 patients but not in those of healthy donors. Its expression correlated with the markers of T-cell differentiation and exhaustion and blood cytokine levels. The percentage of HERV-W ENV-positive lymphocytes correlated with inflammatory markers and pneumonia severity in COVID-19 patients. Notably, HERV-W ENV expression reflects the respiratory outcome of patients during hospitalization. Given the known immuno- and neuro-pathogenicity of HERV-W ENV protein, it could promote certain pathogenic features of COVID-19 and therefore serve as a biomarker to predict clinical progression of disease and open to further studies for therapeutic intervention. Information available at the end of the manuscript.

Sections du résumé

BACKGROUND BACKGROUND
Despite an impressive effort in clinical research, no standard therapeutic approach for coronavirus disease 2019 (COVID-19) patients has been established, highlighting the need to identify early biomarkers for predicting disease progression and new therapeutic interventions for patient management. The present study aimed to evaluate the involvement of the human endogenous retrovirus -W envelope (HERV-W ENV) in severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection considering recent findings that HERVs are activated in response to infectious agents and lead to various immunopathological effects. We analysed HERV-W ENV expression in blood cells of COVID-19 patients in correlation with clinical characteristics and have discussed its potential role in the outcome of the disease.
METHODS METHODS
We analysed HERV-W ENV expression in blood samples of COVID-19 patients and healthy donors by flow cytometry and quantitative reverse transcriptase PCR analysis, and evaluated its correlation with clinical signs, inflammatory markers, cytokine expression, and disease progression.
FINDINGS RESULTS
HERV-W ENV was highly expressed in the leukocytes of COVID-19 patients but not in those of healthy donors. Its expression correlated with the markers of T-cell differentiation and exhaustion and blood cytokine levels. The percentage of HERV-W ENV-positive lymphocytes correlated with inflammatory markers and pneumonia severity in COVID-19 patients. Notably, HERV-W ENV expression reflects the respiratory outcome of patients during hospitalization.
INTERPRETATION CONCLUSIONS
Given the known immuno- and neuro-pathogenicity of HERV-W ENV protein, it could promote certain pathogenic features of COVID-19 and therefore serve as a biomarker to predict clinical progression of disease and open to further studies for therapeutic intervention.
FUNDING BACKGROUND
Information available at the end of the manuscript.

Identifiants

pubmed: 33867312
pii: S2352-3964(21)00134-1
doi: 10.1016/j.ebiom.2021.103341
pmc: PMC8082064
pii:
doi:

Substances chimiques

Antiviral Agents 0
Cytokines 0
Gene Products, env 0
IL6 protein, human 0
Interleukin-6 0
Pregnancy Proteins 0
syncytin 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

103341

Informations de copyright

Copyright © 2021 The Author(s). Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interests C.M. reports grant from Gilead, outside the submitted work; M.I. reports personal fees from Biogen srl, personal fees from Becton, Dickinson and Company, outside the submitted work; HP and BC receive compensation for their work by Geneuro-Innovation. The other authors have nothing to disclose.

Auteurs

Emanuela Balestrieri (E)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy.

Antonella Minutolo (A)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy.

Vita Petrone (V)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy.

Marialaura Fanelli (M)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy.

Marco Iannetta (M)

Department of Systems Medicine, University of Rome Tor Vergata, Rome 00133, Italy; Infectious Diseases Clinic, Policlinic of Tor Vergata, Rome 00133, Italy.

Vincenzo Malagnino (V)

Department of Systems Medicine, University of Rome Tor Vergata, Rome 00133, Italy; Infectious Diseases Clinic, Policlinic of Tor Vergata, Rome 00133, Italy.

Marta Zordan (M)

Department of Systems Medicine, University of Rome Tor Vergata, Rome 00133, Italy; Infectious Diseases Clinic, Policlinic of Tor Vergata, Rome 00133, Italy.

Pietro Vitale (P)

Infectious Diseases Clinic, Policlinic of Tor Vergata, Rome 00133, Italy.

Benjamin Charvet (B)

International Center for Infectiology Research (CIRI), INSERM U1111, CNRS UMR5308, Ecole Normale Supérieure de Lyon, University of Lyon, Lyon, France; Geneuro - Innovation, Lyon 69008, France.

Branka Horvat (B)

International Center for Infectiology Research (CIRI), INSERM U1111, CNRS UMR5308, Ecole Normale Supérieure de Lyon, University of Lyon, Lyon, France.

Sergio Bernardini (S)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy.

Enrico Garaci (E)

IRCCS San Raffaele Pisana, Rome 00163, Italy.

Paolo di Francesco (P)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy.

Paola Sinibaldi Vallebona (P)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy; Institute of Translational Pharmacology, National Research Council, Rome 00133, Italy.

Loredana Sarmati (L)

Department of Systems Medicine, University of Rome Tor Vergata, Rome 00133, Italy; Infectious Diseases Clinic, Policlinic of Tor Vergata, Rome 00133, Italy.

Sandro Grelli (S)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy; Virology Unit, Policlinic of Tor Vergata, Rome 00133, Italy.

Massimo Andreoni (M)

Department of Systems Medicine, University of Rome Tor Vergata, Rome 00133, Italy; Infectious Diseases Clinic, Policlinic of Tor Vergata, Rome 00133, Italy.

Hervé Perron (H)

Geneuro - Innovation, Lyon 69008, France; University of Lyon, Lyon 69007, France.

Claudia Matteucci (C)

Department of Experimental Medicine, University of Rome Tor Vergata, Rome 00133, Italy. Electronic address: matteucci@med.uniroma2.it.

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Classifications MeSH