Clopidogrel underactivity is a common in patients with acute symptomatic severe carotid stenosis.


Journal

Journal of the neurological sciences
ISSN: 1878-5883
Titre abrégé: J Neurol Sci
Pays: Netherlands
ID NLM: 0375403

Informations de publication

Date de publication:
15 06 2021
Historique:
received: 22 11 2020
revised: 09 04 2021
accepted: 12 04 2021
pubmed: 21 4 2021
medline: 26 5 2021
entrez: 20 4 2021
Statut: ppublish

Résumé

Clopidogrel is commonly used for secondary stroke prevention in patients with large vessel stenosis. Reduced Clopidogrel high on treatment platelet reactivity (CR) can lead to Clopidogrel underactivity (CU) causing acute thrombosis. However, the prevalence of CU among patients with acute symptomatic carotid disease remains unknown. Therefore, we aimed to find the prevalence and identify the predictors for CU among patients with acutely symptomatic carotid stenosis. Over the span of 14 months, CR was measured at the time of endovascular procedure in all patients undergoing angiography and stenting because of acute symptomatic carotid stenosis. Only patients treated per institutional protocol with a combination of Clopidogrel and Aspirin were included. CR was measured with VerifyNowP2Y12 reaction units (PRU) and CU was defined as PRU > 208. Patients with CU were compared to those without CU. Thirty-five patients were included (mean age 71.3 ± 10, 76% men) and twelve (34.3%, mean age 71.8 ± 8.4, 58% men) had CU at the time of endovascular intervention. On univariate analysis more severe carotid stenosis was seen in CU patients (92.6 ± 6.5% vs 81.6 ± 13.6%, p = 0.013) and percent stenosis was independently associated with CU on multivariate analysis (p = 0.023). CU is present in 1 of every 3 patients with acutely symptomatic carotid disease. The current results suggest that CR testing should become part of routine care in patients with acutely symptomatic carotid disease.

Sections du résumé

BACKGROUND
Clopidogrel is commonly used for secondary stroke prevention in patients with large vessel stenosis. Reduced Clopidogrel high on treatment platelet reactivity (CR) can lead to Clopidogrel underactivity (CU) causing acute thrombosis. However, the prevalence of CU among patients with acute symptomatic carotid disease remains unknown. Therefore, we aimed to find the prevalence and identify the predictors for CU among patients with acutely symptomatic carotid stenosis.
PATIENTS AND METHODS
Over the span of 14 months, CR was measured at the time of endovascular procedure in all patients undergoing angiography and stenting because of acute symptomatic carotid stenosis. Only patients treated per institutional protocol with a combination of Clopidogrel and Aspirin were included. CR was measured with VerifyNowP2Y12 reaction units (PRU) and CU was defined as PRU > 208. Patients with CU were compared to those without CU.
RESULTS
Thirty-five patients were included (mean age 71.3 ± 10, 76% men) and twelve (34.3%, mean age 71.8 ± 8.4, 58% men) had CU at the time of endovascular intervention. On univariate analysis more severe carotid stenosis was seen in CU patients (92.6 ± 6.5% vs 81.6 ± 13.6%, p = 0.013) and percent stenosis was independently associated with CU on multivariate analysis (p = 0.023).
CONCLUSIONS
CU is present in 1 of every 3 patients with acutely symptomatic carotid disease. The current results suggest that CR testing should become part of routine care in patients with acutely symptomatic carotid disease.

Identifiants

pubmed: 33878658
pii: S0022-510X(21)00144-1
doi: 10.1016/j.jns.2021.117450
pii:
doi:

Substances chimiques

Platelet Aggregation Inhibitors 0
Clopidogrel A74586SNO7
Aspirin R16CO5Y76E

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

117450

Informations de copyright

Copyright © 2021 Elsevier B.V. All rights reserved.

Auteurs

A Honig (A)

Department of Neurology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel. Electronic address: Asaf.honig2@gmail.com.

T Sacagiu (T)

Department of Neurology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.

A Filioglo (A)

Department of Neurology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.

N Simaan (N)

Department of Neurology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.

Y Kalish (Y)

Department of Hematology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.

J M Gomori (JM)

Department of Radiology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.

A Horev (A)

Department of Neurology, Soroka- University Medical Center, Beer-Sheva, Israel.

R R Leker (RR)

Department of Neurology, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.

J E Cohen (JE)

Department of Neurosurgery, Hadassah-Hebrew University Medical Center, Jerusalem, Israel.

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Classifications MeSH