Significance of Oil-Red-O positive macrophages in bronchoalveolar lavage in diagnosing E-cigarettes or vaping product use-associated lung injury: A case series.


Journal

Diagnostic cytopathology
ISSN: 1097-0339
Titre abrégé: Diagn Cytopathol
Pays: United States
ID NLM: 8506895

Informations de publication

Date de publication:
Jul 2021
Historique:
revised: 12 04 2021
received: 01 12 2020
accepted: 19 04 2021
pubmed: 27 4 2021
medline: 15 12 2021
entrez: 26 4 2021
Statut: ppublish

Résumé

Lipid-laden macrophages detected by Oil-Red-O (ORO) stain in fresh bronchoalveolar lavage (BAL) specimens have been proposed as a potential diagnostic marker for E-cigarettes or vaping product use-associated lung injury (EVALI). However, studies are few, and the sensitivity and specificity of the test have not been thoroughly investigated. We performed ORO stain on fresh BAL specimens from six confirmed EVALI and 36 non-EVALI patients. After semi-quantitative analysis, the sensitivity and specificity of ORO-positive macrophages (OPM) for detection of EVALI were calculated. No significant difference in cytomorphology or raw macrophage count was observed between EVALI and non-EVALI groups (49% vs 55% of all nucleated cells). However, with ORO stain, all EVALI specimens (6/6) showed a high percentage (≥50% of all macrophages) of OPM (mean 87%), and large (≥25% of host macrophage nuclear size) lipid droplets (mean 42%), while the majority of non-EVALI specimens showed a low percentage of OPM (32/36, mean 10%), and small lipid droplets (34/36, mean 6%). The differences between the two groups in both high OPM and large lipid droplet rates are statistically significant (P < .0001 for both comparisons). The combined sensitivity and specificity of high OPM and large lipid droplets for diagnosing EVALI were 100% and 94%, respectively. In BAL specimens obtained from patients with clinically suspected EVALI, a high percentage of OPM with large lipid droplets showed high sensitivity and specificity for the diagnosis of EVALI and may serve as a potentially useful tool in the evaluation of vaping-related lung injury, improving diagnostic accuracy.

Sections du résumé

BACKGROUND BACKGROUND
Lipid-laden macrophages detected by Oil-Red-O (ORO) stain in fresh bronchoalveolar lavage (BAL) specimens have been proposed as a potential diagnostic marker for E-cigarettes or vaping product use-associated lung injury (EVALI). However, studies are few, and the sensitivity and specificity of the test have not been thoroughly investigated.
METHODS METHODS
We performed ORO stain on fresh BAL specimens from six confirmed EVALI and 36 non-EVALI patients. After semi-quantitative analysis, the sensitivity and specificity of ORO-positive macrophages (OPM) for detection of EVALI were calculated.
RESULTS RESULTS
No significant difference in cytomorphology or raw macrophage count was observed between EVALI and non-EVALI groups (49% vs 55% of all nucleated cells). However, with ORO stain, all EVALI specimens (6/6) showed a high percentage (≥50% of all macrophages) of OPM (mean 87%), and large (≥25% of host macrophage nuclear size) lipid droplets (mean 42%), while the majority of non-EVALI specimens showed a low percentage of OPM (32/36, mean 10%), and small lipid droplets (34/36, mean 6%). The differences between the two groups in both high OPM and large lipid droplet rates are statistically significant (P < .0001 for both comparisons). The combined sensitivity and specificity of high OPM and large lipid droplets for diagnosing EVALI were 100% and 94%, respectively.
CONCLUSION CONCLUSIONS
In BAL specimens obtained from patients with clinically suspected EVALI, a high percentage of OPM with large lipid droplets showed high sensitivity and specificity for the diagnosis of EVALI and may serve as a potentially useful tool in the evaluation of vaping-related lung injury, improving diagnostic accuracy.

Identifiants

pubmed: 33900686
doi: 10.1002/dc.24760
doi:

Substances chimiques

Azo Compounds 0
Coloring Agents 0
E-Cigarette Vapor 0
oil red O G7S71FND9B

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

876-884

Subventions

Organisme : University of Texas Southwestern Medical Center

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

Outbreak of Lung Injury Associated with the Use of E-Cigarette, or Vaping Products February 2019. https://www.cdc.gov/tobacco/basic_information/e-cigarettes/severe-lung-disease.html.
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Butt YM, Smith ML, Tazelaar HD, et al. Pathology of vaping-associated lung injury. N Engl J Med. 2019;381(18):1780-1781.
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Mukhopadhyay S, Mehrad M, Dammert P, et al. Lung biopsy findings in severe pulmonary illness associated with E-cigarette use (vaping). Am J Clin Pathol. 2020;153(1):30-39.
Davidson K, Brancato A, Heetderks P, et al. Outbreak of electronic-cigarette-associated acute lipoid pneumonia - North Carolina, July-august 2019. MMWR Morb Mortal Wkly Rep. 2019;68(36):784-786.
Layden JE, Ghinai I, Pray I, et al. Pulmonary illness related to E-cigarette use in Illinois and Wisconsin - final report. N Engl J Med. 2020;382(10):903-916.
Modi SSR, Alhajhusain A. Acute lipoid pneumonia secondary to e-cigarette use: an unlikely replacement for cigarettes [abstract]. Chest. 2015;148:382A.
Maddock SD, Cirulis MM, Callahan SJ, et al. Pulmonary lipid-laden macrophages and vaping. N Engl J Med. 2019;381(15):1488-1489.
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Pambuccian SE. Testing for lipid-laden macrophages in bronchoalveolar lavage fluid to diagnose vaping-associated pulmonary injury. Are we there yet? J Am Soc Cytopathol. 2020;9(1):1-8.
Bhat TA, Kalathil SG, Bogner PN, Blount BC, Goniewicz ML, Thanavala YM. An animal model of inhaled vitamin E acetate and EVALI-like lung injury. N Engl J Med. 2020;382(12):1175-1177.
Corwin RW, Irwin RS. The lipid-laden alveolar macrophage as a marker of aspiration in parenchymal lung disease. Am Rev Respir Dis. 1985;132(3):576-581.
Shabbir A, Das A, Sehgal S, Lau C, Highland KB. A 40-year-old woman with progressive shortness of breath, cough, and recurrent "pneumonia". Ann Am Thorac Soc. 2016;13(5):746-750.

Auteurs

Shuang Niu (S)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Pathology, Parkland Hospital, Dallas, Texas, USA.

Glorimar Rivera Colon (GR)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Pathology, Parkland Hospital, Dallas, Texas, USA.

Kyle Molberg (K)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Pathology, Parkland Hospital, Dallas, Texas, USA.

Hao Chen (H)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Pathology, Parkland Hospital, Dallas, Texas, USA.

Kelley Carrick (K)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Pathology, Parkland Hospital, Dallas, Texas, USA.

Shirley Yan (S)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Venetia Sarode (V)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Elena Lucas (E)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Pathology, Parkland Hospital, Dallas, Texas, USA.
Harold C. Simmons Comprehensive Cancer Center, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

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