Small Airway Dysfunction Links Asthma Severity with Physical Activity and Symptom Control.

Asthma control Physical activity Small airway dysfunction Structural equation modeling

Journal

The journal of allergy and clinical immunology. In practice
ISSN: 2213-2201
Titre abrégé: J Allergy Clin Immunol Pract
Pays: United States
ID NLM: 101597220

Informations de publication

Date de publication:
09 2021
Historique:
received: 04 12 2020
revised: 12 04 2021
accepted: 12 04 2021
pubmed: 1 5 2021
medline: 28 10 2021
entrez: 30 4 2021
Statut: ppublish

Résumé

Little is known about the role of small airway dysfunction (SAD) and its complex relation with asthma control and physical activity (PA). To investigate the interrelations among SAD, risk factors for asthma severity, symptom control, and PA. We assessed SAD by impulse oscillometry and other sophisticated lung function measures including inert gas washout in adults with asthma (mild to moderate, n = 140; severe, n = 128) and 69 healthy controls from the All Age Asthma Cohort. We evaluated SAD prevalence and its interrelation with risk factors for asthma severity (older age, obesity, and smoking), type 2 inflammation (sputum and blood eosinophils, fractional exhaled nitric oxide), systemic inflammation (high-sensitivity C-reactive protein), asthma control (AC), and PA (accelerometer for 1 week). We applied a clinical model based on structural equation modeling that integrated causal pathways among these clinical variables. The prevalence of SAD ranged from 75% to 90% in patients with severe asthma and from 53% to 64% in mild to moderate asthma. Severe SAD was associated with poor AC and low PA. Structural equation modeling indicated that age, obesity, obesity-related systemic inflammation, T2 inflammation, and smoking are independent predictors of SAD. Small airway dysfunction was the main determinant factor of AC, which in turn affected PA. Obesity affected AC directly and through its contribution to SAD and low PA. In addition, PA had bidirectional associations with obesity, SAD, and AC. Structural equation modeling also indicated interrelations among distal airflow limitation, air trapping, and ventilation heterogeneity. Small airway dysfunction is a highly prevalent key feature of asthma that interrelates a spectrum of distal lung function abnormalities with risk factors for asthma severity, asthma control, and physical activity.

Sections du résumé

BACKGROUND
Little is known about the role of small airway dysfunction (SAD) and its complex relation with asthma control and physical activity (PA).
OBJECTIVE
To investigate the interrelations among SAD, risk factors for asthma severity, symptom control, and PA.
METHODS
We assessed SAD by impulse oscillometry and other sophisticated lung function measures including inert gas washout in adults with asthma (mild to moderate, n = 140; severe, n = 128) and 69 healthy controls from the All Age Asthma Cohort. We evaluated SAD prevalence and its interrelation with risk factors for asthma severity (older age, obesity, and smoking), type 2 inflammation (sputum and blood eosinophils, fractional exhaled nitric oxide), systemic inflammation (high-sensitivity C-reactive protein), asthma control (AC), and PA (accelerometer for 1 week). We applied a clinical model based on structural equation modeling that integrated causal pathways among these clinical variables.
RESULTS
The prevalence of SAD ranged from 75% to 90% in patients with severe asthma and from 53% to 64% in mild to moderate asthma. Severe SAD was associated with poor AC and low PA. Structural equation modeling indicated that age, obesity, obesity-related systemic inflammation, T2 inflammation, and smoking are independent predictors of SAD. Small airway dysfunction was the main determinant factor of AC, which in turn affected PA. Obesity affected AC directly and through its contribution to SAD and low PA. In addition, PA had bidirectional associations with obesity, SAD, and AC. Structural equation modeling also indicated interrelations among distal airflow limitation, air trapping, and ventilation heterogeneity.
CONCLUSIONS
Small airway dysfunction is a highly prevalent key feature of asthma that interrelates a spectrum of distal lung function abnormalities with risk factors for asthma severity, asthma control, and physical activity.

Identifiants

pubmed: 33930619
pii: S2213-2198(21)00497-9
doi: 10.1016/j.jaip.2021.04.035
pii:
doi:

Substances chimiques

Nitric Oxide 31C4KY9ESH

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3359-3368.e1

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2021 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Mustafa Abdo (M)

LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany. Electronic address: m.abdo@lungenclinic.de.

Frederik Trinkmann (F)

Department of Pneumology and Critical Care Medicine, Thoraxklinik, University of Heidelberg, Translational Lung Research Center Heidelberg, German Center for Lung Research, Heidelberg, Germany; Department of Biomedical Informatics, Heinrich-Lanz-Center, University Medical Center Mannheim, Mannheim, Germany.

Anne-Marie Kirsten (AM)

Pulmonary Research Institute at the LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany.

Frauke Pedersen (F)

LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany; Pulmonary Research Institute at the LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany.

Christian Herzmann (C)

Research Center Borstel, Airway Research Center North, German Center for Lung Research, Borstel, Germany.

Erika von Mutius (E)

Dr von Hauner Children's Hospital, Ludwig Maximilians University of Munich, Comprehensive Pneumology Center Munich, German Center for Lung Research, Munich, Germany.

Matthias V Kopp (MV)

Division of Pediatric Pulmonology and Allergology, University Children's Hospital, Luebeck, Germany, Airway Research Center North, German Center for Lung Research, Luebeck, Germany.

Gesine Hansen (G)

Department of Paediatric Pneumology, Allergology, and Neonatology, Hannover Medical School, Biomedical Research in Endstage and Obstructive Lung Disease, German Center for Lung Research, Hannover, Germany.

Benjamin Waschki (B)

LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany; Department of General and Interventional Cardiology, University Heart Center Hamburg, Hamburg, Germany.

Klaus F Rabe (KF)

LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany.

Henrik Watz (H)

Pulmonary Research Institute at the LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany.

Thomas Bahmer (T)

LungenClinic Grosshansdorf, Airway Research Center North, German Center for Lung Research, Grosshansdorf, Germany; University Hospital Schleswig-Holstein-Campus Kiel, Department for Internal Medicine I, Airway Research Center North, German Center for Lung Research, Kiel, Germany.

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Classifications MeSH