Optimal timing and criteria of interim PET in DLBCL: a comparative study of 1692 patients.


Journal

Blood advances
ISSN: 2473-9537
Titre abrégé: Blood Adv
Pays: United States
ID NLM: 101698425

Informations de publication

Date de publication:
11 05 2021
Historique:
received: 08 02 2021
accepted: 20 03 2021
entrez: 4 5 2021
pubmed: 5 5 2021
medline: 1 6 2021
Statut: ppublish

Résumé

Interim 18F-fluorodeoxyglucose positron emission tomography (Interim-18F-FDG-PET, hereafter I-PET) has the potential to guide treatment of patients with diffuse large B-cell lymphoma (DLBCL) if the prognostic value is known. The aim of this study was to determine the optimal timing and response criteria for evaluating prognosis with I-PET in DLBCL. Individual patient data from 1692 patients with de novo DLBCL were combined and scans were harmonized. I-PET was performed at various time points during treatment with rituximab plus cyclophosphamide, doxorubicin, vincristine, and prednisone (R-CHOP) therapy. Scans were interpreted using the Deauville score (DS) and change in maximum standardized uptake value (ΔSUVmax). Multilevel Cox proportional hazards models corrected for International Prognostic Index (IPI) score were used to study the effects of timing and response criteria on 2-year progression-free survival (PFS). I-PET after 2 cycles (I-PET2) and I-PET4 significantly discriminated good responders from poor responders, with the highest hazard ratios (HRs) for I-PET4. Multivariable HRs for a PET-positive result at I-PET2 and I-PET4 were 1.71 and 2.95 using DS4-5, 4.91 and 6.20 using DS5, and 2.93 and 4.65 using ΔSUVmax, respectively. ΔSUVmax identified a larger proportion of poor responders than DS5 did. For all criteria, the negative predictive value was >80%, and positive predictive values ranged from 30% to 70% at I-PET2 and I-PET4. Unlike I-PET1, I-PET3 discriminated good responders from poor responders using DS4-5 and DS5 thresholds (HRs, 2.94 and 4.67, respectively). I-PET2 and I-PET4 predict good response equally during R-CHOP therapy in DLBCL. Optimal timing and response criteria depend on the clinical context. Good response at I-PET2 is suggested for de-escalation trials, and poor response using ΔSUVmax at I-PET4 is suggested for randomized trials that are evaluating new therapies.

Identifiants

pubmed: 33944897
pii: S2473-9529(21)00302-5
doi: 10.1182/bloodadvances.2021004467
pmc: PMC8114547
doi:

Substances chimiques

Fluorodeoxyglucose F18 0Z5B2CJX4D
Vincristine 5J49Q6B70F

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2375-2384

Subventions

Organisme : Wellcome Trust
ID : WT 203148/Z/16/Z
Pays : United Kingdom
Organisme : Medical Research Council
Pays : United Kingdom

Informations de copyright

© 2021 by The American Society of Hematology.

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Auteurs

J J Eertink (JJ)

Department of Hematology, Cancer Center Amsterdam, and.

C N Burggraaff (CN)

Department of Hematology, Cancer Center Amsterdam, and.

M W Heymans (MW)

Department of Epidemiology and Data Science, Amsterdam Public Health Research Institute, Amsterdam UMC, Vrije Universeit Amsterdam, Amsterdam, The Netherlands.

U Dührsen (U)

Department of Hematology, West German Cancer Center, and.

A Hüttmann (A)

Department of Hematology, West German Cancer Center, and.

C Schmitz (C)

Department of Hematology, West German Cancer Center, and.

S Müller (S)

Department of Nuclear Medicine, University Hospital Essen, University of Duisburg-Essen, Essen, Germany.

P J Lugtenburg (PJ)

Department of Hematology, Erasmus Medical Center Cancer Institute, Rotterdam, The Netherlands.

S F Barrington (SF)

King's College London and Guy's and St Thomas' PET Centre, School of Biomedical Engineering and Imaging Sciences, King's Health Partners, and.

N G Mikhaeel (NG)

Department of Clinical Oncology, Guy's Cancer Centre and School of Cancer and Pharmaceutical Sciences, King's College London, London, United Kingdom.

R Carr (R)

Department of Haematology, Guy's and St Thomas' National Health Service Foundation Trust and Cancer Division, King's College London, London, United Kingdom.

S Czibor (S)

Department of Nuclear Medicine, Medical Imaging Centre, Semmelweis University, Budapest, Hungary.

T Györke (T)

Department of Nuclear Medicine, Medical Imaging Centre, Semmelweis University, Budapest, Hungary.

L Ceriani (L)

Department of Nuclear Medicine and PET/CT Centre, Imaging Institute of Southern Switzerland, Bellinzona, Switzerland.
SAKK-Swiss Group for Clinical Cancer Research, Bern, Switzerland.

E Zucca (E)

SAKK-Swiss Group for Clinical Cancer Research, Bern, Switzerland.
Medical Oncology Clinics, Oncology Institute of Southern Switzerland, Università della Svizzera Italiana, Bellinzona, Switzerland.

M Hutchings (M)

Department of Hematology, Rigshospitalet, Copenhagen University Hospital, Copenhagen, Denmark.

L Kostakoglu (L)

Department of Radiology and Medical Imaging, University of Virginia Health System, Charlottesville, VA.

A Loft (A)

Department of Clinical Physiology, Nuclear Medicine and PET, Rigshospitalet, Copenhagen University Hospital, Copenhagen, Denmark.

S Fanti (S)

Department of Nuclear Medicine, Sant'Orsola-Malpighi Hospital, Bologna, Italy; and.

S E Wiegers (SE)

Department of Hematology, Cancer Center Amsterdam, and.

S Pieplenbosch (S)

Department of Hematology, Cancer Center Amsterdam, and.

R Boellaard (R)

Department of Radiology and Nuclear Medicine, Cancer Center Amsterdam, Amsterdam UMC, Vrije Universiteit Amsterdam, Amsterdam, The Netherlands.

O S Hoekstra (OS)

Department of Radiology and Nuclear Medicine, Cancer Center Amsterdam, Amsterdam UMC, Vrije Universiteit Amsterdam, Amsterdam, The Netherlands.

J M Zijlstra (JM)

Department of Hematology, Cancer Center Amsterdam, and.

H C W de Vet (HCW)

Department of Epidemiology and Data Science, Amsterdam Public Health Research Institute, Amsterdam UMC, Vrije Universeit Amsterdam, Amsterdam, The Netherlands.

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