COVID-19 in DMARD-treated patients with inflammatory rheumatic diseases: Insights from an analysis of the World Health Organization pharmacovigilance database.


Journal

Fundamental & clinical pharmacology
ISSN: 1472-8206
Titre abrégé: Fundam Clin Pharmacol
Pays: England
ID NLM: 8710411

Informations de publication

Date de publication:
Feb 2022
Historique:
revised: 09 04 2021
received: 16 12 2020
accepted: 06 05 2021
pubmed: 12 5 2021
medline: 18 1 2022
entrez: 11 5 2021
Statut: ppublish

Résumé

To determine whether the use of disease-modifying antirheumatic drugs (DMARDs) is linked to the risk of COVID-19 among patients with inflammatory rheumatic diseases (IRDs). We performed a disproportionality analysis of the World Health Organization pharmacovigilance database between January 1, 2020, and June 10, 2020. The frequency of COVID-19 reports for all DMARD classes identified was compared with that for all other reports for all other drugs and quoted as the reporting odds ratio (ROR) (95% confidence interval [CI]). Among 980,446 individual case-safety reports voluntarily recorded in the database, 398 identified COVID-19 in DMARD-treated patients with IRDs. There were 177 (44.5%) patients with rheumatoid arthritis (RA), 120 (30.1%) with ankylosing spondylitis (AS), 93 (23.4%) with psoriatic arthritis (PsA), and 8 (2.0%) with juvenile idiopathic arthritis. Most of the cases of COVID-19 occurred in patients taking anti-TNF agents (84.2%), resulting in a significant disproportionality signal (ROR [95% CI]: 8.31 [7.48-9.23]) - particularly in patients with RA, AS or PsA. A significant inverse disproportionality was found for the anti-IL-6 agent tocilizumab (ROR [95% CI]: 0.12 [0.02-0.88]) and JAK inhibitors (ROR [95% CI]: 0.33 [0.19-0.58]) in patients with RA - suggesting that these two drug classes are safer in the context of RA. Our results are in line with the literature on a potentially better safety profile for anti-IL-6 agents and JAK inhibitors. The WHO pharmacovigilance data suggest that COVID-19 is significantly more frequent in patients with IRDs treated with TNF inhibitors.

Sections du résumé

BACKGROUND BACKGROUND
To determine whether the use of disease-modifying antirheumatic drugs (DMARDs) is linked to the risk of COVID-19 among patients with inflammatory rheumatic diseases (IRDs).
METHODS METHODS
We performed a disproportionality analysis of the World Health Organization pharmacovigilance database between January 1, 2020, and June 10, 2020. The frequency of COVID-19 reports for all DMARD classes identified was compared with that for all other reports for all other drugs and quoted as the reporting odds ratio (ROR) (95% confidence interval [CI]).
RESULTS RESULTS
Among 980,446 individual case-safety reports voluntarily recorded in the database, 398 identified COVID-19 in DMARD-treated patients with IRDs. There were 177 (44.5%) patients with rheumatoid arthritis (RA), 120 (30.1%) with ankylosing spondylitis (AS), 93 (23.4%) with psoriatic arthritis (PsA), and 8 (2.0%) with juvenile idiopathic arthritis. Most of the cases of COVID-19 occurred in patients taking anti-TNF agents (84.2%), resulting in a significant disproportionality signal (ROR [95% CI]: 8.31 [7.48-9.23]) - particularly in patients with RA, AS or PsA. A significant inverse disproportionality was found for the anti-IL-6 agent tocilizumab (ROR [95% CI]: 0.12 [0.02-0.88]) and JAK inhibitors (ROR [95% CI]: 0.33 [0.19-0.58]) in patients with RA - suggesting that these two drug classes are safer in the context of RA.
CONCLUSION CONCLUSIONS
Our results are in line with the literature on a potentially better safety profile for anti-IL-6 agents and JAK inhibitors. The WHO pharmacovigilance data suggest that COVID-19 is significantly more frequent in patients with IRDs treated with TNF inhibitors.

Identifiants

pubmed: 33973280
doi: 10.1111/fcp.12695
pmc: PMC8239613
doi:

Substances chimiques

Antirheumatic Agents 0
Tumor Necrosis Factor Inhibitors 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

199-209

Informations de copyright

© 2021 Société Française de Pharmacologie et de Thérapeutique.

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Auteurs

Amandine Dernoncourt (A)

Department of Internal Medicine, Amiens-Picardie University Medical Center, Amiens, France.
RECIF, Amiens-Picardie University Medical Center, Amiens, France.

Jean Schmidt (J)

Department of Internal Medicine, Amiens-Picardie University Medical Center, Amiens, France.
RECIF, Amiens-Picardie University Medical Center, Amiens, France.

Pierre Duhaut (P)

Department of Internal Medicine, Amiens-Picardie University Medical Center, Amiens, France.
RECIF, Amiens-Picardie University Medical Center, Amiens, France.

Sophie Liabeuf (S)

Department of Clinical Pharmacology, Amiens University Medical Center, Amiens, France.
MP3CV Laboratory, Jules Verne University of Picardie, Amiens, France.

Valérie Gras-Champel (V)

Department of Clinical Pharmacology, Amiens University Medical Center, Amiens, France.
MP3CV Laboratory, Jules Verne University of Picardie, Amiens, France.

Kamel Masmoudi (K)

Department of Clinical Pharmacology, Amiens University Medical Center, Amiens, France.

Youssef Bennis (Y)

Department of Clinical Pharmacology, Amiens University Medical Center, Amiens, France.
MP3CV Laboratory, Jules Verne University of Picardie, Amiens, France.

Benjamin Batteux (B)

RECIF, Amiens-Picardie University Medical Center, Amiens, France.
Department of Clinical Pharmacology, Amiens University Medical Center, Amiens, France.
MP3CV Laboratory, Jules Verne University of Picardie, Amiens, France.
Department of Rheumatology, Saint-Quentin Medical Center, Saint-Quentin, France.

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