Basophil reactivity to BNT162b2 is mediated by PEGylated lipid nanoparticles in patients with PEG allergy.


Journal

The Journal of allergy and clinical immunology
ISSN: 1097-6825
Titre abrégé: J Allergy Clin Immunol
Pays: United States
ID NLM: 1275002

Informations de publication

Date de publication:
07 2021
Historique:
received: 13 04 2021
revised: 22 04 2021
accepted: 28 04 2021
pubmed: 16 5 2021
medline: 20 7 2021
entrez: 15 5 2021
Statut: ppublish

Résumé

The mechanisms underpinning allergic reactions to the BNT162b2 (Pfizer) COVID-19 vaccine remain unknown, with polyethylene glycol (PEG) contained in the lipid nanoparticle suspected as being the cause. Our aim was to evaluate the performance of skin testing and basophil activation testing to PEG, polysorbate 80, and the BNT162b2 (Pfizer) and AZD1222 (AstraZeneca) COVID-19 vaccines in patients with a history of PEG allergy. Three known individuals with PEG allergy and 3 healthy controls were recruited and evaluated for hypersensitivity to the BNT162b2 and AZD1222 vaccines, and to related compounds by skin testing and basophil activation, as measured by CD63 upregulation using flow cytometry. We found that the BNT162b2 vaccine induced positive skin test results in patients with PEG allergy, whereas the result of traditional PEG skin testing was negative in 2 of 3 patients. One patient was found to be cosensitized to both the BNT162b2 and AZD1222 vaccines because of cross-reactive PEG and polysorbate allergy. The BNT162b2 vaccine, but not PEG alone, induced dose-dependent activation of all patients' basophils ex vivo. Similar basophil activation could be induced by PEGylated liposomal doxorubicin, suggesting that PEGylated lipids within nanoparticles, but not PEG in its native state, are able to efficiently induce degranulation. Our findings implicate PEG, as covalently modified and arranged on the vaccine lipid nanoparticle, as a potential trigger of anaphylaxis in response to BNT162b2, and highlight shortcomings of current skin testing protocols for allergy to PEGylated liposomal drugs.

Sections du résumé

BACKGROUND
The mechanisms underpinning allergic reactions to the BNT162b2 (Pfizer) COVID-19 vaccine remain unknown, with polyethylene glycol (PEG) contained in the lipid nanoparticle suspected as being the cause.
OBJECTIVE
Our aim was to evaluate the performance of skin testing and basophil activation testing to PEG, polysorbate 80, and the BNT162b2 (Pfizer) and AZD1222 (AstraZeneca) COVID-19 vaccines in patients with a history of PEG allergy.
METHODS
Three known individuals with PEG allergy and 3 healthy controls were recruited and evaluated for hypersensitivity to the BNT162b2 and AZD1222 vaccines, and to related compounds by skin testing and basophil activation, as measured by CD63 upregulation using flow cytometry.
RESULTS
We found that the BNT162b2 vaccine induced positive skin test results in patients with PEG allergy, whereas the result of traditional PEG skin testing was negative in 2 of 3 patients. One patient was found to be cosensitized to both the BNT162b2 and AZD1222 vaccines because of cross-reactive PEG and polysorbate allergy. The BNT162b2 vaccine, but not PEG alone, induced dose-dependent activation of all patients' basophils ex vivo. Similar basophil activation could be induced by PEGylated liposomal doxorubicin, suggesting that PEGylated lipids within nanoparticles, but not PEG in its native state, are able to efficiently induce degranulation.
CONCLUSIONS
Our findings implicate PEG, as covalently modified and arranged on the vaccine lipid nanoparticle, as a potential trigger of anaphylaxis in response to BNT162b2, and highlight shortcomings of current skin testing protocols for allergy to PEGylated liposomal drugs.

Identifiants

pubmed: 33991580
pii: S0091-6749(21)00731-4
doi: 10.1016/j.jaci.2021.04.032
pii:
doi:

Substances chimiques

COVID-19 Vaccines 0
Lipids 0
liposomal doxorubicin 0
Polyethylene Glycols 3WJQ0SDW1A
Doxorubicin 80168379AG
ChAdOx1 nCoV-19 B5S3K2V0G8
BNT162 Vaccine N38TVC63NU

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

91-95

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

Copyright © 2021 American Academy of Allergy, Asthma & Immunology. All rights reserved.

Auteurs

Alexander Troelnikov (A)

SA Pathology, Adelaide, Australia.

Griffith Perkins (G)

SA Pathology, Adelaide, Australia; School of Biological Sciences, University of Adelaide, Adelaide, Australia.

Chino Yuson (C)

Immunology Department, Royal Adelaide Hospital, Adelaide, Australia.

Aida Ahamdie (A)

Immunology Department, Royal Adelaide Hospital, Adelaide, Australia.

Summaya Balouch (S)

Immunology Department, Royal Adelaide Hospital, Adelaide, Australia.

Plinio R Hurtado (PR)

Department of Renal Medicine, Royal Adelaide Hospital, Adelaide, Australia; School of Medicine, University of Adelaide, Adelaide, Australia.

Pravin Hissaria (P)

SA Pathology, Adelaide, Australia; Immunology Department, Royal Adelaide Hospital, Adelaide, Australia. Electronic address: pravin.hissaria@sa.gov.au.

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Classifications MeSH