The Interaction of Organic Cation Transporters 1-3 and PMAT with Psychoactive Substances.


Journal

Handbook of experimental pharmacology
ISSN: 0171-2004
Titre abrégé: Handb Exp Pharmacol
Pays: Germany
ID NLM: 7902231

Informations de publication

Date de publication:
2021
Historique:
pubmed: 17 5 2021
medline: 29 10 2021
entrez: 16 5 2021
Statut: ppublish

Résumé

Organic cation transporters 1-3 (OCT1-3, SLC22A1-3) and the plasma membrane monoamine transporter (PMAT, SLC29A4) play a major role in maintaining monoaminergic equilibrium in the central nervous system. With many psychoactive substances interacting with OCT1-3 and PMAT, a growing literature focuses on characterizing their properties via in vitro and in vivo studies. In vitro studies mainly aim at characterizing compounds as inhibitors or substrates of murine, rat, and human isoforms. The preponderance of studies has put emphasis on phenylalkylamine derivatives, but ketamine and opioids have also been investigated. Studies employing in vivo (knockout) models mostly concentrate on the interaction of psychoactive substances and OCT3, with an emphasis on stress and addiction, pharmacokinetics, and sensitization to psychoactive drugs. The results highlight the importance of OCT3 in the mechanism of action of psychoactive compounds. Concerning in vivo studies, a veritable research gap concerning OCT1, 2, and PMAT exists. This review provides an overview and summary of research conducted in this field of research.

Identifiants

pubmed: 33993413
doi: 10.1007/164_2021_469
doi:

Substances chimiques

Cations 0
Organic Cation Transport Proteins 0
Organic Cation Transporter 1 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

199-214

Subventions

Organisme : Austrian Science Fund FWF
ID : DOC 33
Pays : Austria

Informations de copyright

© 2021. The Author(s), under exclusive license to Springer Nature Switzerland AG.

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Auteurs

Julian Maier (J)

Center for Physiology and Pharmacology, Institute of Pharmacology, Medical University of Vienna, Vienna, Austria.

Marco Niello (M)

Center for Physiology and Pharmacology, Institute of Pharmacology, Medical University of Vienna, Vienna, Austria.

Deborah Rudin (D)

Center for Physiology and Pharmacology, Institute of Pharmacology, Medical University of Vienna, Vienna, Austria.

Lynette C Daws (LC)

Department of Cellular and Integrative Physiology, University of Texas Health, San Antonio, TX, USA.
Department of Pharmacology, University of Texas Health, San Antonio, TX, USA.

Harald H Sitte (HH)

Center for Physiology and Pharmacology, Institute of Pharmacology, Medical University of Vienna, Vienna, Austria. harald.sitte@meduniwien.ac.at.

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