The Endocannabinoid Anandamide Attenuates Acute Respiratory Distress Syndrome by Downregulating miRNA that Target Inflammatory Pathways.
T regulatory cells (T regs)
acute respiratory distress syndrome (ARDS)
anandamide (AEA)
miRNA-23a-3p
miRNA-34a-5p
micro-RNA (miRNA/miR)
myeloid derived suppressor cells (MDSC)
staphylococcus enterotoxin B (SEB)
Journal
Frontiers in pharmacology
ISSN: 1663-9812
Titre abrégé: Front Pharmacol
Pays: Switzerland
ID NLM: 101548923
Informations de publication
Date de publication:
2021
2021
Historique:
received:
20
12
2020
accepted:
22
03
2021
entrez:
17
5
2021
pubmed:
18
5
2021
medline:
18
5
2021
Statut:
epublish
Résumé
Acute respiratory distress syndrome (ARDS) is defined as a type of respiratory failure that is caused by a variety of insults such as pneumonia, sepsis, trauma and certain viral infections. In this study, we investigated the effect of an endocannabinoid, anandamide (AEA), on ARDS induced in the mouse by
Identifiants
pubmed: 33995054
doi: 10.3389/fphar.2021.644281
pii: 644281
pmc: PMC8113864
doi:
Types de publication
Journal Article
Langues
eng
Pagination
644281Informations de copyright
Copyright © 2021 Sultan, Alghetaa, Mohammed, Abdulla, Wisniewski, Singh, Nagarkatti and Nagarkatti.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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