Breaking the therapeutic ceiling in drug development in ulcerative colitis.


Journal

The lancet. Gastroenterology & hepatology
ISSN: 2468-1253
Titre abrégé: Lancet Gastroenterol Hepatol
Pays: Netherlands
ID NLM: 101690683

Informations de publication

Date de publication:
07 2021
Historique:
received: 03 01 2021
revised: 15 02 2021
accepted: 18 02 2021
pubmed: 22 5 2021
medline: 17 8 2021
entrez: 21 5 2021
Statut: ppublish

Résumé

Increased knowledge of the intricate pathogenesis of ulcerative colitis has triggered an advance in drug development during the past two decades, resulting in the advent of several biological agents and small-molecule therapies. Although the increase in therapeutic options is positive, remission rates of patients with ulcerative colitis given new therapeutic agents in induction trials remain at a modest 20-30%, seemingly facing a so-called therapeutic ceiling. This therapeutic ceiling requires a critical appraisal and highlights the need for alternative strategies for drug development. In this Review, we objectively itemise the boundaries of therapeutic efficacy in ulcerative colitis, provide possible explanations for the shortcomings of current strategies, and propose solutions to achieve better therapeutic outcomes in ulcerative colitis.

Identifiants

pubmed: 34019798
pii: S2468-1253(21)00065-0
doi: 10.1016/S2468-1253(21)00065-0
pii:
doi:

Substances chimiques

Anti-Inflammatory Agents, Non-Steroidal 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

589-595

Subventions

Organisme : NIDDK NIH HHS
ID : P30 DK097948
Pays : United States

Informations de copyright

Copyright © 2021 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests BV reports financial support for research from Pfizer; lecture fees from Abbvie, Biogen, Chiesi, Falk, Ferring, Galapagos, Janssen, MSD, Pfizer, R-Biopharm, Takeda, and Truvion; and consultancy fees from Janssen, Guidepont, and Sandoz. SV reports financial support for research from MSD, AbbVie, Takeda, Pfizer, Johnson & Johnson; lecture fees from MSD, AbbVie, Takeda, Ferring, Centocor, Hospira, Pfizer, Johnson & Johnson, and Genentech and Roche; and consultancy fees from MSD, AbbVie, Takeda, Ferring, Centocor, Hospira, Pfizer, Johnson & Johnson, Genentech and Roche, Celgene, Mundipharma, Celltrion, SecondGenome, Prometheus, Shire, Prodigest, Gilead, and Galapagos. CF received speaker fees from UCB, Sandoz, Janssen, and Genentech; consultancy fees from Athos Therapeutics; and grants from US National Institutes of Health (P30 DK097948-06 and T32DK083251-11). DA declares no competing interests.

Auteurs

Dahham Alsoud (D)

Translational Research in Gastrointestinal Disorders, Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium.

Bram Verstockt (B)

Translational Research in Gastrointestinal Disorders, Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium; Department of Gastroenterology and Hepatology, University Hospitals Leuven, KU Leuven, Leuven, Belgium.

Claudio Fiocchi (C)

Department of Inflammation and Immunity, Lerner Research Institute, Cleveland, OH, USA; Department of Gastroenterology, Hepatology and Nutrition, Digestive Disease and Surgery Institute, Cleveland Clinic, Cleveland, OH, USA.

Séverine Vermeire (S)

Translational Research in Gastrointestinal Disorders, Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium; Department of Gastroenterology and Hepatology, University Hospitals Leuven, KU Leuven, Leuven, Belgium. Electronic address: severine.vermeire@uzleuven.be.

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Classifications MeSH