Autistic traits are associated with faster pace of aging: Evidence from the Dunedin study at age 45.

aging autism spectrum disorder autistic traits intelligence physical health socioeconomic status

Journal

Autism research : official journal of the International Society for Autism Research
ISSN: 1939-3806
Titre abrégé: Autism Res
Pays: United States
ID NLM: 101461858

Informations de publication

Date de publication:
08 2021
Historique:
revised: 31 03 2021
received: 11 12 2020
accepted: 03 05 2021
pubmed: 28 5 2021
medline: 13 8 2021
entrez: 27 5 2021
Statut: ppublish

Résumé

Growing evidence indicates that the defining characteristics of autism spectrum disorder (ASD) are distributed throughout the general population; hence, understanding the correlates of aging in people with high autistic traits could shed light on ASD and aging. 915 members of the Dunedin longitudinal birth cohort completed a measure of autistic traits at age 45. A composite measure of the "pace of aging" was derived by tracking the decline in 19 biomarkers across ages 26, 32, 38, and 45 years. Facial age was also assessed. Reports of perceived health were collected from participants themselves, informants, and interviewers. Higher self-reported autistic traits significantly correlated with a faster pace of aging, older facial age, and poorer self-, informant-, and interviewer-rated health. After control for sex, SES and IQ, autistic traits were significantly associated with each variable: pace of aging (β = 0.09), facial age (β = 0.08), self- (β = -0.15), informant (β = -0.12), and interviewer-rated (β = -0.17) health. Autistic traits measured at age 45 are associated with faster aging. Participants with high autistic traits appear to be more vulnerable to poor health outcomes, as previously reported for those clinically diagnosed with ASD. Therefore, autistic traits may have important health implications. Replicating these findings in samples of autistic people is needed to identify the mechanism of their effect on aging and physical health to improve outcomes for those with ASD diagnoses or high autistic traits. LAY SUMMARY: The role that autistic traits have in relation to health outcomes has not been investigated. We looked at how physical health and aging (measured with self-reported questions and decline in multiple biological measures) were related to autistic traits (measured with a questionnaire, at age 45). We found that higher autistic traits were associated with poorer reports of physical health, and a faster pace of aging. This suggests that both those with autism and those with higher autistic traits may be more likely to experience poorer health outcomes.

Identifiants

pubmed: 34042279
doi: 10.1002/aur.2534
pmc: PMC8328948
mid: NIHMS1719906
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1684-1694

Subventions

Organisme : NIA NIH HHS
ID : R01 AG032282
Pays : United States
Organisme : NIA NIH HHS
ID : R01 AG049789
Pays : United States

Informations de copyright

© 2021 The Authors. Autism Research published by International Society for Autism Research and Wiley Periodicals LLC.

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Auteurs

David Mason (D)

Social, Genetic, and Developmental Psychiatry Centre, Institute of Psychiatry, Psychology, and Neuroscience, King's College London, London, United Kingdom.

Angelica Ronald (A)

Centre for Brain and Cognitive Development, Department of Psychological Sciences, Birkbeck University of London, London, United Kingdom.

Antony Ambler (A)

Social, Genetic, and Developmental Psychiatry Centre, Institute of Psychiatry, Psychology, and Neuroscience, King's College London, London, United Kingdom.
Dunedin Multidisciplinary Health and Development Research Unit, University of Otago, Dunedin, New Zealand.

Avshalom Caspi (A)

Social, Genetic, and Developmental Psychiatry Centre, Institute of Psychiatry, Psychology, and Neuroscience, King's College London, London, United Kingdom.
Department of Psychology and Neuroscience, Duke University, Durham, North Carolina, USA.
Department of Psychiatry and Behavioral Sciences, Duke University School of Medicine, Durham, North Carolina, USA.
Center for Genomic and Computational Biology, Duke University, Durham, North Carolina, USA.
PROMENTA Center, University of Oslo, Oslo, Norway.

Renate Houts (R)

Department of Psychology and Neuroscience, Duke University, Durham, North Carolina, USA.

Richie Poulton (R)

Dunedin Multidisciplinary Health and Development Research Unit, University of Otago, Dunedin, New Zealand.

Sandhya Ramrakha (S)

Dunedin Multidisciplinary Health and Development Research Unit, University of Otago, Dunedin, New Zealand.

Jasmin Wertz (J)

Department of Psychology and Neuroscience, Duke University, Durham, North Carolina, USA.

Terrie E Moffitt (TE)

Social, Genetic, and Developmental Psychiatry Centre, Institute of Psychiatry, Psychology, and Neuroscience, King's College London, London, United Kingdom.
Department of Psychology and Neuroscience, Duke University, Durham, North Carolina, USA.
Department of Psychiatry and Behavioral Sciences, Duke University School of Medicine, Durham, North Carolina, USA.
Center for Genomic and Computational Biology, Duke University, Durham, North Carolina, USA.
PROMENTA Center, University of Oslo, Oslo, Norway.

Francesca Happé (F)

Social, Genetic, and Developmental Psychiatry Centre, Institute of Psychiatry, Psychology, and Neuroscience, King's College London, London, United Kingdom.

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