Transcranial Magnetic Stimulation in Succinic Semialdehyde Dehydrogenase Deficiency: A Measure of Maturational Trajectory of Cortical Excitability.

Succinic semialdehyde dehydrogenase deficiency (SSADHD) cortical excitability cortical silent period (CSP) excitation: inhibition ratio long interval intracortical inhibtion (LICI) maturational trajectory resting motor threshold (rMT) transcranial magnetic stimulation (TMS)

Journal

Journal of child neurology
ISSN: 1708-8283
Titre abrégé: J Child Neurol
Pays: United States
ID NLM: 8606714

Informations de publication

Date de publication:
11 2021
Historique:
pubmed: 2 6 2021
medline: 15 3 2022
entrez: 1 6 2021
Statut: ppublish

Résumé

Succinic semialdehyde dehydrogenase deficiency (SSADHD) is a disorder of GABA degradation with use-dependent downregulation of postsynaptic GABA In this single-center observational study, 18 subjects with SSADHD and 8 healthy controls underwent transcranial magnetic stimulation. Resting motor threshold, cortical silent period, and long-interval intracortical inhibition were measured in both groups. Resting motor threshold in focal epilepsy patients from an institutional transcranial magnetic stimulation database were also included. SSADHD subjects had higher resting motor threshold than healthy controls but lower relative to focal epilepsy patients. Resting motor threshold decreased with age in all groups. Cortical silent period was longer in SSADHD subjects than in healthy controls. No difference was detected in long-interval intracortical inhibition between the 2 groups. Findings suggest abnormal corticospinal tract physiology in SSADHD, but with preserved developmental trajectory for corticospinal tract maturation. Defining features of these transcranial magnetic stimulation metrics in SSADHD will be better elucidated through this ongoing longitudinal study.

Sections du résumé

BACKGROUND
Succinic semialdehyde dehydrogenase deficiency (SSADHD) is a disorder of GABA degradation with use-dependent downregulation of postsynaptic GABA
METHODS
In this single-center observational study, 18 subjects with SSADHD and 8 healthy controls underwent transcranial magnetic stimulation. Resting motor threshold, cortical silent period, and long-interval intracortical inhibition were measured in both groups. Resting motor threshold in focal epilepsy patients from an institutional transcranial magnetic stimulation database were also included.
RESULTS
SSADHD subjects had higher resting motor threshold than healthy controls but lower relative to focal epilepsy patients. Resting motor threshold decreased with age in all groups. Cortical silent period was longer in SSADHD subjects than in healthy controls. No difference was detected in long-interval intracortical inhibition between the 2 groups.
CONCLUSION
Findings suggest abnormal corticospinal tract physiology in SSADHD, but with preserved developmental trajectory for corticospinal tract maturation. Defining features of these transcranial magnetic stimulation metrics in SSADHD will be better elucidated through this ongoing longitudinal study.

Identifiants

pubmed: 34058900
doi: 10.1177/08830738211008735
pmc: PMC8630082
mid: NIHMS1686399
doi:

Substances chimiques

Succinate-Semialdehyde Dehydrogenase EC 1.2.1.24

Types de publication

Journal Article Observational Study Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1169-1176

Subventions

Organisme : NICHD NIH HHS
ID : P50 HD105351
Pays : United States
Organisme : NICHD NIH HHS
ID : R01 HD091142
Pays : United States

Auteurs

Melissa Tsuboyama (M)

Division of Epilepsy and Clinical Neurophysiology, Department of Neurology, 1862Boston Children's Hospital, Boston, MA, USA.

Jingjing Liu (J)

Division of Epilepsy and Clinical Neurophysiology, Department of Neurology, 1862Boston Children's Hospital, Boston, MA, USA.
F.M. Kirby Neurobiology Center, 1862Boston Children's Hospital, Boston, MA, USA.

Harper Kaye (H)

12259Boston University School of Medicine, Behavioral Neuroscience Program, Boston, MA, USA.

Melissa DiBacco (M)

Division of Epilepsy and Clinical Neurophysiology, Department of Neurology, 1862Boston Children's Hospital, Boston, MA, USA.

Phillip L Pearl (PL)

Division of Epilepsy and Clinical Neurophysiology, Department of Neurology, 1862Boston Children's Hospital, Boston, MA, USA.

Alexander Rotenberg (A)

Division of Epilepsy and Clinical Neurophysiology, Department of Neurology, 1862Boston Children's Hospital, Boston, MA, USA.
F.M. Kirby Neurobiology Center, 1862Boston Children's Hospital, Boston, MA, USA.
Berenson-Allen Center for Noninvasive Brain Stimulation, Department of Neurology, Beth Israel Deaconess Medical Center, Boston, MA, USA.

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Classifications MeSH