A novel gene associated with small bowel bleeding in patients taking low-dose aspirin.


Journal

Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
ISSN: 1878-3562
Titre abrégé: Dig Liver Dis
Pays: Netherlands
ID NLM: 100958385

Informations de publication

Date de publication:
07 2021
Historique:
received: 08 03 2021
revised: 30 04 2021
accepted: 30 04 2021
pubmed: 2 6 2021
medline: 2 2 2022
entrez: 1 6 2021
Statut: ppublish

Résumé

We have previously revealed the clinical factors and genetic polymorphisms associated with gastrointestinal mucosal injury and bleeding, induced by low-dose aspirin (LDA). After performing genome-wide analysis of single nucleotide polymorphisms (SNPs) using the Drug Metabolizing Enzymes and Transporters (DMET) system among drug metabolism and transporter genes, certain SNPs were found to increase the risk for LDA-induced small bowel bleeding. The aim of this study was to identify the SNPs involved in LDA-induced small bowel bleeding. Subjects were patients taking LDA, with small bowel bleeding diagnosed using capsule endoscopy. We investigated the clinical characteristics and the previously identified SNPs, that were examined by the DNA direct sequence method. 56 patients with bleeding and 410 controls taking LDA were enrolled. The risk factors associated with small bowel bleeding included smoking, cerebrovascular diseases, chronic renal failure, non-steroidal anti-inflammatory drug (NSAID) or anticoagulants combination, and two SNPs (CYP4F11 20043G>A (D446N) rs1060463, GSTP1 313A>G rs1695). After propensity score matching, GSTP1 rs1695 was significantly associated with small bowel bleeding. The GSTP1 SNP may be a predictive marker for small bowel bleeding among patients taking LDA.

Identifiants

pubmed: 34059446
pii: S1590-8658(21)00243-7
doi: 10.1016/j.dld.2021.04.038
pii:
doi:

Substances chimiques

Cytochrome P450 Family 4 EC 1.14.14.1
CYP4F11 protein, human EC 1.14.14.78
GSTP1 protein, human EC 2.5.1.18
Glutathione S-Transferase pi EC 2.5.1.18
Aspirin R16CO5Y76E

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

841-845

Informations de copyright

Copyright © 2021 Editrice Gastroenterologica Italiana S.r.l. Published by Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest None.

Auteurs

Yukiko Handa (Y)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan. Electronic address: yukko0903handaclinic@gmail.com.

Shinya Fukushima (S)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan.

Shogen Yo (S)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan.

Motoyasu Osawa (M)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan.

Takahisa Murao (T)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan.

Osamu Handa (O)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan.

Hiroshi Matsumoto (H)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan.

Eiji Umegaki (E)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan.

Takashi Sakakibara (T)

Department of Gastroenterology, The Sakakibara Heart Institute of Okayama, Okayama City, Okayama, Japan.

Akiko Shiotani (A)

Division of Gastroenterology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki City, Okayama, Japan.

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Classifications MeSH