Reduced adult neurogenesis is associated with increased macrophages in the subependymal zone in schizophrenia.


Journal

Molecular psychiatry
ISSN: 1476-5578
Titre abrégé: Mol Psychiatry
Pays: England
ID NLM: 9607835

Informations de publication

Date de publication:
11 2021
Historique:
received: 10 12 2020
accepted: 26 04 2021
revised: 17 04 2021
pubmed: 2 6 2021
medline: 15 3 2022
entrez: 1 6 2021
Statut: ppublish

Résumé

Neural stem cells in the human subependymal zone (SEZ) generate neuronal progenitor cells that can differentiate and integrate as inhibitory interneurons into cortical and subcortical brain regions; yet the extent of adult neurogenesis remains unexplored in schizophrenia and bipolar disorder. We verified the existence of neurogenesis across the lifespan by chartering transcriptional alterations (2 days-103 years, n = 70) and identifying cells indicative of different stages of neurogenesis in the human SEZ. Expression of most neural stem and neuronal progenitor cell markers decreased during the first postnatal years and remained stable from childhood into ageing. We next discovered reduced neural stem and neuronal progenitor cell marker expression in the adult SEZ in schizophrenia and bipolar disorder compared to controls (n = 29-32 per group). RNA sequencing identified increased expression of the macrophage marker CD163 as the most significant molecular change in schizophrenia. CD163

Identifiants

pubmed: 34059796
doi: 10.1038/s41380-021-01149-3
pii: 10.1038/s41380-021-01149-3
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

6880-6895

Subventions

Organisme : NIAAA NIH HHS
ID : R28 AA012725
Pays : United States

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer Nature Limited.

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Auteurs

Christin Weissleder (C)

Schizophrenia Research Laboratory, Neuroscience Research Australia, Randwick, NSW, Australia.

Hayley F North (HF)

Schizophrenia Research Laboratory, Neuroscience Research Australia, Randwick, NSW, Australia.
School of Psychiatry, Faculty of Medicine, University of New South Wales, Sydney, NSW, Australia.

Maina Bitar (M)

QIMR Berghofer Medical Research Institute, Herston, QLD, Australia.

Janice M Fullerton (JM)

Neuroscience Research Australia, Randwick, NSW, Australia.
School of Medical Sciences, Faculty of Medicine, University of New South Wales, Sydney, NSW, Australia.

Rachel Sager (R)

Department of Neuroscience and Physiology, Upstate Medical University, Syracuse, NY, USA.

Guy Barry (G)

QIMR Berghofer Medical Research Institute, Herston, QLD, Australia.

Michael Piper (M)

School of Biomedical Sciences, The University of Queensland, Brisbane, QLD, Australia.
Queensland Brain Institute, The University of Queensland, Brisbane, QLD, Australia.

Glenda M Halliday (GM)

Neuroscience Research Australia, Randwick, NSW, Australia.
School of Medical Sciences, Faculty of Medicine, University of New South Wales, Sydney, NSW, Australia.
Brain and Mind Centre and Faculty of Medicine and Health, School of Medical Sciences, University of Sydney, Sydney, NSW, Australia.

Maree J Webster (MJ)

Laboratory of Brain Research, Stanley Medical Research Institute, Rockville, MD, USA.

Cynthia Shannon Weickert (C)

Schizophrenia Research Laboratory, Neuroscience Research Australia, Randwick, NSW, Australia. c.weickert@neura.edu.au.
School of Psychiatry, Faculty of Medicine, University of New South Wales, Sydney, NSW, Australia. c.weickert@neura.edu.au.
Department of Neuroscience and Physiology, Upstate Medical University, Syracuse, NY, USA. c.weickert@neura.edu.au.

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