Cardiac tamponade: Better prognosis in association with pulmonary embolism: Case report.

Prognosis Pulmonary arterial hypertension Pulmonary embolism Tamponade Tuberculosis

Journal

Annals of medicine and surgery (2012)
ISSN: 2049-0801
Titre abrégé: Ann Med Surg (Lond)
Pays: England
ID NLM: 101616869

Informations de publication

Date de publication:
Jun 2021
Historique:
received: 18 04 2021
revised: 15 05 2021
accepted: 16 05 2021
entrez: 7 6 2021
pubmed: 8 6 2021
medline: 8 6 2021
Statut: epublish

Résumé

The tamponade leads to an increase in intrapericardial pressure, which impairs the diastolic filling of the ventricles and reduces ejection. However, the association with pulmonary arterial hypertension, which in turn leads to an intracardiac hyper-pressure, constitutes a compensatory mechanism. We report the case of a 23 year old patient followed for anemia due to martial deficiency for 2 years, who consulted us with right heart failure evolving for 5 months, a hemoptoic cough and chest tightness. The entire workup revealed a pericardial and pleural tamponade of tuberculous origin associated with a pulmonary embolism evaluated at 15% according to the Qanadli score. After stabilization of her hemodynamic state, the patient was put on anti-bacillary and anticoagulant treatment with a good improvement of her cardiopulmonary state. She was discharged after 1 month (satisfactory check-up), regularly followed up in outpatient clinic with check-up of hemostasis and cardiac echography every 2 weeks. She was declared cured of her tuberculosis after 6 months of treatment. There was no recurrence after two years of follow-up. The combination of tamponade and pulmonary hypertension is synergistic in that it improves the patient's prognosis.

Identifiants

pubmed: 34094528
doi: 10.1016/j.amsu.2021.102410
pii: S2049-0801(21)00360-5
pmc: PMC8166641
doi:

Types de publication

Case Reports

Langues

eng

Pagination

102410

Informations de copyright

© 2021 The Authors.

Déclaration de conflit d'intérêts

Authors of this article have no conflict or competing interests. All of the authors approved the final version of the manuscript.

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Auteurs

A Baragé (A)

Department of Cardiology, IBN ROCHD University Hospital, Casablanca, B.P, 5696, Casablanca, Morocco.

S Harouna Idrissa (S)

Department of Cardiology, IBN ROCHD University Hospital, Casablanca, B.P, 5696, Casablanca, Morocco.

N Mahoungou-Mackonia (N)

Department of Cardiology, IBN ROCHD University Hospital, Casablanca, B.P, 5696, Casablanca, Morocco.

S Arous (S)

Faculty of Medicine and Pharmacy, Hassan II University of Casablanca, B.P, 5696, Casablanca, Morocco.
Department of Cardiology, IBN ROCHD University Hospital, Casablanca, B.P, 5696, Casablanca, Morocco.

G Bennouna (G)

Faculty of Medicine and Pharmacy, Hassan II University of Casablanca, B.P, 5696, Casablanca, Morocco.
Department of Cardiology, IBN ROCHD University Hospital, Casablanca, B.P, 5696, Casablanca, Morocco.

L Azzouzi (L)

Faculty of Medicine and Pharmacy, Hassan II University of Casablanca, B.P, 5696, Casablanca, Morocco.
Department of Cardiology, IBN ROCHD University Hospital, Casablanca, B.P, 5696, Casablanca, Morocco.

R Habbal (R)

Faculty of Medicine and Pharmacy, Hassan II University of Casablanca, B.P, 5696, Casablanca, Morocco.
Department of Cardiology, IBN ROCHD University Hospital, Casablanca, B.P, 5696, Casablanca, Morocco.

Classifications MeSH