Serum neurofilament levels reflect outer retinal layer changes in multiple sclerosis.

multiple sclerosis neuroimmunology optic neuritis optical coherence tomography serum neurofilament translation

Journal

Therapeutic advances in neurological disorders
ISSN: 1756-2856
Titre abrégé: Ther Adv Neurol Disord
Pays: England
ID NLM: 101480242

Informations de publication

Date de publication:
2021
Historique:
received: 22 10 2020
accepted: 28 02 2021
entrez: 9 6 2021
pubmed: 10 6 2021
medline: 10 6 2021
Statut: epublish

Résumé

Serum neurofilament light chain (sNfL) and distinct intra-retinal layers are both promising biomarkers of neuro-axonal injury in multiple sclerosis (MS). We aimed to unravel the association of both markers in early MS, having identified that neurofilament has a distinct immunohistochemical expression pattern among intra-retinal layers. Three-dimensional (3D) spectral domain macular optical coherence tomography scans and sNfL levels were investigated in 156 early MS patients (female/male: 109/47, mean age: 33.3 ± 9.5 years, mean disease duration: 2.0 ± 3.3 years). Out of the whole cohort, 110 patients had no history of optic neuritis (NHON) and 46 patients had a previous history of optic neuritis (HON). In addition, a subgroup of patients ( In our cohort, HON patients had a thinner outer plexiform layer (OPL) volume compared to NHON patients ( In summary, sNfL levels were a good predictor of future outer retinal thinning in MS. Changes within the neurofilament-rich OPL could be considered as an additional retinal marker linked to MS neurodegeneration.

Sections du résumé

BACKGROUND BACKGROUND
Serum neurofilament light chain (sNfL) and distinct intra-retinal layers are both promising biomarkers of neuro-axonal injury in multiple sclerosis (MS). We aimed to unravel the association of both markers in early MS, having identified that neurofilament has a distinct immunohistochemical expression pattern among intra-retinal layers.
METHODS METHODS
Three-dimensional (3D) spectral domain macular optical coherence tomography scans and sNfL levels were investigated in 156 early MS patients (female/male: 109/47, mean age: 33.3 ± 9.5 years, mean disease duration: 2.0 ± 3.3 years). Out of the whole cohort, 110 patients had no history of optic neuritis (NHON) and 46 patients had a previous history of optic neuritis (HON). In addition, a subgroup of patients (
RESULTS RESULTS
In our cohort, HON patients had a thinner outer plexiform layer (OPL) volume compared to NHON patients (
CONCLUSIONS CONCLUSIONS
In summary, sNfL levels were a good predictor of future outer retinal thinning in MS. Changes within the neurofilament-rich OPL could be considered as an additional retinal marker linked to MS neurodegeneration.

Identifiants

pubmed: 34104217
doi: 10.1177/17562864211003478
pii: 10.1177_17562864211003478
pmc: PMC8155762
doi:

Types de publication

Journal Article

Langues

eng

Pagination

17562864211003478

Informations de copyright

© The Author(s), 2021.

Déclaration de conflit d'intérêts

Conflict of interest statement: The authors declare that there is no conflict of interest.

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Auteurs

Caspar B Seitz (CB)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network (rmn), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.

Falk Steffen (F)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network (rmn), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.

Muthuraman Muthuraman (M)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network (rmn), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.

Timo Uphaus (T)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network (rmn), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.

Julia Krämer (J)

Department of Neurology with Institute of Translational Neurology, University of Münster, Albert-Schweizer-Campus, Münster, Germany.

Sven G Meuth (SG)

Department of Neurology with Institute of Translational Neurology, University of Münster, Albert-Schweizer-Campus, Münster, Germany.

Philipp Albrecht (P)

Department of Neurology, Medical Faculty, Heinrich-Heine University, Düsseldorf, Germany.

Sergiu Groppa (S)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network (rmn), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.

Frauke Zipp (F)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network (rmn), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.

Stefan Bittner (S)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network (rmn), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.

Vinzenz Fleischer (V)

Department of Neurology, Focus Program Translational Neuroscience (FTN), Rhine Main Neuroscience Network (rmn), University Medical Center of the Johannes Gutenberg University Mainz, Langenbeckstr. 1, Mainz 55131, Germany.

Classifications MeSH