Association of Maternal Viral Load and CD4 Count With Perinatal HIV-1 Transmission Risk During Breastfeeding in the PROMISE Postpartum Component.


Journal

Journal of acquired immune deficiency syndromes (1999)
ISSN: 1944-7884
Titre abrégé: J Acquir Immune Defic Syndr
Pays: United States
ID NLM: 100892005

Informations de publication

Date de publication:
01 10 2021
Historique:
received: 27 11 2020
accepted: 11 03 2021
pubmed: 11 6 2021
medline: 31 12 2021
entrez: 10 6 2021
Statut: ppublish

Résumé

Breastfeeding mothers with HIV infection not qualifying for antiretroviral therapy (ART) based on country-specific guidelines at the time of the Promoting Maternal-Infant Survival Everywhere trial and their uninfected neonates were randomized to maternal ART (mART) or infant nevirapine prophylaxis (iNVP) postpartum. HIV transmission proportions were similar (<1%) in the 2 arms. We assessed whether maternal viral load (MVL) and CD4 cell counts were associated with breastfeeding HIV transmission. MVL was collected at entry (7-14 days postpartum) and at weeks 6, 14, 26, and 50 postpartum. CD4 cell counts were collected at entry and weeks 14, 26, 38, and 50 postpartum. Infant HIV-1 nucleic acid test was performed at weeks 1 and 6, every 4 weeks until week 26, and then every 12 weeks. The associations of baseline and time-varying MVL and CD4 cell counts with transmission risk were assessed using time-to-event analyses by randomized treatment arm. Two thousand four hundred thirty-one mother-infant pairs were enrolled in the study. Baseline MVL (P = 0.11) and CD4 cell counts (P = 0.51) were not significantly associated with infant HIV-1 infection. Time-varying MVL was significantly associated with infant HIV-1 infection {hazard ratio [95% confidence interval (CI)]: 13.96 (3.12 to 62.45)} in the mART arm but not in the iNVP arm [hazard ratio (95% CI): 1.04 (0.20 to 5.39)]. Time-varying CD4 cell counts were also significantly associated with infant HIV-1 infection [hazard ratio (95% CI): 0.18 (0.03 to 0.93)] in the mART arm but not in the iNVP arm [hazard ratio (95% CI): 0.38 (0.08 to 1.77)]. In women receiving mART, increased MVL and decreased CD4 cell counts during breastfeeding were associated with increased risk of infant HIV-1 infection.

Sections du résumé

BACKGROUND
Breastfeeding mothers with HIV infection not qualifying for antiretroviral therapy (ART) based on country-specific guidelines at the time of the Promoting Maternal-Infant Survival Everywhere trial and their uninfected neonates were randomized to maternal ART (mART) or infant nevirapine prophylaxis (iNVP) postpartum. HIV transmission proportions were similar (<1%) in the 2 arms. We assessed whether maternal viral load (MVL) and CD4 cell counts were associated with breastfeeding HIV transmission.
METHODS
MVL was collected at entry (7-14 days postpartum) and at weeks 6, 14, 26, and 50 postpartum. CD4 cell counts were collected at entry and weeks 14, 26, 38, and 50 postpartum. Infant HIV-1 nucleic acid test was performed at weeks 1 and 6, every 4 weeks until week 26, and then every 12 weeks. The associations of baseline and time-varying MVL and CD4 cell counts with transmission risk were assessed using time-to-event analyses by randomized treatment arm.
RESULTS
Two thousand four hundred thirty-one mother-infant pairs were enrolled in the study. Baseline MVL (P = 0.11) and CD4 cell counts (P = 0.51) were not significantly associated with infant HIV-1 infection. Time-varying MVL was significantly associated with infant HIV-1 infection {hazard ratio [95% confidence interval (CI)]: 13.96 (3.12 to 62.45)} in the mART arm but not in the iNVP arm [hazard ratio (95% CI): 1.04 (0.20 to 5.39)]. Time-varying CD4 cell counts were also significantly associated with infant HIV-1 infection [hazard ratio (95% CI): 0.18 (0.03 to 0.93)] in the mART arm but not in the iNVP arm [hazard ratio (95% CI): 0.38 (0.08 to 1.77)].
CONCLUSIONS
In women receiving mART, increased MVL and decreased CD4 cell counts during breastfeeding were associated with increased risk of infant HIV-1 infection.

Identifiants

pubmed: 34108383
doi: 10.1097/QAI.0000000000002744
pii: 00126334-202110010-00013
pmc: PMC8434954
mid: NIHMS1711531
doi:

Substances chimiques

Anti-HIV Agents 0
Nevirapine 99DK7FVK1H

Types de publication

Journal Article Randomized Controlled Trial Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

206-213

Subventions

Organisme : NIAID NIH HHS
ID : UM1 AI068632
Pays : United States
Organisme : NIAID NIH HHS
ID : UM1 AI069469
Pays : United States
Organisme : NIAID NIH HHS
ID : UM1 AI069518
Pays : United States
Organisme : NIAID NIH HHS
ID : P30 AI027757
Pays : United States
Organisme : NIAID NIH HHS
ID : UM1 AI069436
Pays : United States
Organisme : NIAID NIH HHS
ID : U01 AI068632
Pays : United States
Organisme : NICHD NIH HHS
ID : HHSN275201800001C
Pays : United States
Organisme : NIAID NIH HHS
ID : UM1 AI069463
Pays : United States
Organisme : NIAID NIH HHS
ID : UM1 AI068616
Pays : United States
Organisme : NIAID NIH HHS
ID : UM1 AI106716
Pays : United States
Organisme : NIAID NIH HHS
ID : U01 AI069436
Pays : United States
Organisme : NIAID NIH HHS
ID : UM1 AI069536
Pays : United States
Organisme : NICHD NIH HHS
ID : HHSN275201800001I
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2021 Wolters Kluwer Health, Inc. All rights reserved.

Déclaration de conflit d'intérêts

P.M.F. is a consultant for Merck. The remaining authors have no conflicts of interest to disclose.

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Auteurs

Patricia M Flynn (PM)

Department of Infectious Diseases, St. Jude Children's Research Hospital, Memphis, TN.

Taha E Taha (TE)

Department of Epidemiology, Johns Hopkins Bloomberg School of Public Health, Baltimore, MD.

Mae Cababasay (M)

Center for Biostatistics in AIDS Research, Department of Biostatistics, Harvard T. H. Chan School of Public Health, Boston, MA.

Kevin Butler (K)

Center for Biostatistics in AIDS Research, Department of Biostatistics, Harvard T. H. Chan School of Public Health, Boston, MA.

Mary G Fowler (MG)

Department of Pathology, Johns Hopkins University School of Medicine, Baltimore, MD.

Lynne M Mofenson (LM)

Elizabeth Glaser Pediatric AIDS Foundation, Washington, DC.

Maxensia Owor (M)

Makerere University-Johns Hopkins University Research Collaboration, Kampala, Uganda.

Susan Fiscus (S)

Department of Microbiology and Immunology, University of North Carolina School of Medicine, Chapel Hill, NC.

Lynda Stranix-Chibanda (L)

Department of Paediatrics and Child Health, Faculty of Medicine and Health Sciences, University of Zimbabwe, Harare, Zimbabwe.
University of Zimbabwe Clinical Trials Research Centre, Harare, Zimbabwe.

Anna Coutsoudis (A)

Department of Pediatrics and Child Health, University of KwaZulu-Natal, Durban, South Africa.

Devasena Gnanashanmugam (D)

Division of AIDS, National Institute of Allergy and Immunology, National Institutes of Health, Bethesda, MD.

Nahida Chakhtoura (N)

Maternal and Pediatric Infectious Disease Branch, Division of Extramural Research, Eunice Kennedy Shriver Institute of Child Health and Human Development, National Institutes of Health, Rockville, MD.

Katie McCarthy (K)

FHI 360, Durham, NC.

Lisa Frenkel (L)

Department of Pediatrics and Department of Laboratory Medicine and Pathology, University of Washington, Seattle, WA.
Center for Global Infectious Disease Research, Seattle Children's Research Institute, Seattle, WA.

Ingrid Beck (I)

Center for Global Infectious Disease Research, Seattle Children's Research Institute, Seattle, WA.

Cornelius Mukuzunga (C)

University of North Carolina Project-Malawi, Kamuzu Central Hospital, Lilongwe, Malawi.

Bonus Makanani (B)

Department of Obstetrics and Gynecology, College of Medicine, University of Malawi, Blantyre, Malawi.

Dhayendre Moodley (D)

Department of Obstetrics and Gynaecology, Centre for the AIDS Programme of Research in South Africa and School of Clinical Medicine, College of Health Sciences, University of KwaZulu Natal, Durban, South Africa.

Teacler Nematadzira (T)

University of Zimbabwe Clinical Trials Research Centre, Harare, Zimbabwe.

Bangani Kusakara (B)

University of Zimbabwe Clinical Trials Research Centre, Harare, Zimbabwe.

Sandesh Patil (S)

Department of Obstetrics and Gynecology, Byramjee Jeejeebhoy Government Medical College and Johns Hopkins Clinical Trials Unit, Pune, India.

Tichaona Vhembo (T)

University of Zimbabwe Clinical Trials Research Centre, Harare, Zimbabwe.

Raziya Bobat (R)

Department of Pediatrics and Child Health, Nelson R. Mandela School of Medicine, University of KwaZulu-Natal, Durban, South Africa.

Blandina T Mmbaga (BT)

Department of Pediatrics, Kilimanjaro Christian Medical Centre and Kilimanjaro Christian Medical University College, Moshi, Tanzania.

Maysseb Masenya (M)

Wits Reproductive Health and HIV Institute, Johannesburg, South Africa.

Mandisa Nyati (M)

Perinatal HIV Research Unit, Chris Baragwanath Hospital, Johannesburg, South Africa.

Gerhard Theron (G)

Department of Obstetrics and Gynecology, Faculty of Medicine and Health Sciences, Stellenbosch University, Cape Town, South Africa; and.

Helen Mulenga (H)

Centre for Infectious Disease Research in Zambia, Lusaka, Zambia.

David E Shapiro (DE)

Center for Biostatistics in AIDS Research, Department of Biostatistics, Harvard T. H. Chan School of Public Health, Boston, MA.

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