7-Ketocholesterol: Effects on viral infections and hypothetical contribution in COVID-19.


Journal

The Journal of steroid biochemistry and molecular biology
ISSN: 1879-1220
Titre abrégé: J Steroid Biochem Mol Biol
Pays: England
ID NLM: 9015483

Informations de publication

Date de publication:
09 2021
Historique:
received: 29 04 2021
revised: 04 06 2021
accepted: 07 06 2021
pubmed: 13 6 2021
medline: 10 9 2021
entrez: 12 6 2021
Statut: ppublish

Résumé

7-Ketocholesterol, which is one of the earliest cholesterol oxidization products identified, is essentially formed by the auto-oxidation of cholesterol. In the body, 7-ketocholesterol is both provided by food and produced endogenously. This pro-oxidant and pro-inflammatory molecule, which can activate apoptosis and autophagy at high concentrations, is an abundant component of oxidized Low Density Lipoproteins. 7-Ketocholesterol appears to significantly contribute to the development of age-related diseases (cardiovascular diseases, age-related macular degeneration, and Alzheimer's disease), chronic inflammatory bowel diseases and to certain cancers. Recent studies have also shown that 7-ketocholesterol has anti-viral activities, including on SARS-CoV-2, which are, however, lower than those of oxysterols resulting from the oxidation of cholesterol on the side chain. Furthermore, 7-ketocholesterol is increased in the serum of moderately and severely affected COVID-19 patients. In the case of COVID-19, it can be assumed that the antiviral activity of 7-ketocholesterol could be counterbalanced by its toxic effects, including pro-oxidant, pro-inflammatory and pro-coagulant activities that might promote the induction of cell death in alveolar cells. It is therefore suggested that this oxysterol might be involved in the pathophysiology of COVID-19 by contributing to the acute respiratory distress syndrome and promoting a deleterious, even fatal outcome. Thus, 7-ketocholesterol could possibly constitute a lipid biomarker of COVID-19 outcome and counteracting its toxic effects with adjuvant therapies might have beneficial effects in COVID-19 patients.

Identifiants

pubmed: 34118414
pii: S0960-0760(21)00132-1
doi: 10.1016/j.jsbmb.2021.105939
pmc: PMC8188774
pii:
doi:

Substances chimiques

Antiviral Agents 0
Biomarkers 0
Ketocholesterols 0
7-ketocholesterol O7676FE78M

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

105939

Informations de copyright

Copyright © 2021 Elsevier Ltd. All rights reserved.

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Auteurs

Imen Ghzaiel (I)

University Bourgogne Franche-Comté, Team 'Biochemistry of the Peroxisome, Inflammation and Lipid Metabolism' EA 7270 / Inserm, 21000 Dijon, France; University of Monastir, Faculty of Medicine, LR12ES05, Lab-NAFS 'Nutrition - Functional Food & Vascular Health', 5000 Monastir, Tunisia; University Tunis-El Manar, Faculty of Sciences of Tunis, 2092 Tunis, Tunisia. Electronic address: imenghzaiel93@gmail.com.

Khouloud Sassi (K)

University Bourgogne Franche-Comté, Team 'Biochemistry of the Peroxisome, Inflammation and Lipid Metabolism' EA 7270 / Inserm, 21000 Dijon, France; University Tunis El Manar, Laboratory of Onco-Hematology (LR05ES05), Faculty of Medicine, 1007 Tunis, Tunisia. Electronic address: sassikhouloud@hotmail.com.

Amira Zarrouk (A)

University of Monastir, Faculty of Medicine, LR12ES05, Lab-NAFS 'Nutrition - Functional Food & Vascular Health', 5000 Monastir, Tunisia; University of Sousse, Faculty of Medicine, Sousse, Tunisia. Electronic address: zarroukamira@gmail.com.

Thomas Nury (T)

University Bourgogne Franche-Comté, Team 'Biochemistry of the Peroxisome, Inflammation and Lipid Metabolism' EA 7270 / Inserm, 21000 Dijon, France. Electronic address: thomas.nury@u-bourgogne.fr.

Mohamed Ksila (M)

University Bourgogne Franche-Comté, Team 'Biochemistry of the Peroxisome, Inflammation and Lipid Metabolism' EA 7270 / Inserm, 21000 Dijon, France; University Tunis-El Manar, Loboratory of Neurophysiology, Cellular Physiopathology and Valorisation of BioMoleecules, LR18ES03, Department of Biologie, Faculty of Sciences, 2092 Tunis, Tunisia. Electronic address: mohamedksila44@gmail.com.

Valerio Leoni (V)

Laboratory of Clinical Chemistry, Hospitals of Desio, ASST-Brianza and Department of Medicine and Surgery, University of Milano-Bicocca, 20900 Monza, Italy. Electronic address: valerio.leoni@unimib.it.

Balkiss Bouhaouala-Zahar (B)

Laboratory of Venoms and Therapeutic Molecules, Pasteur Institute of Tunis & University of Tunis El Manar, 1002 Tunis, Tunisia. Electronic address: balkiss.bouhaouala@pasteur.rns.tn.

Sonia Hammami (S)

University of Monastir, Faculty of Medicine, LR12ES05, Lab-NAFS 'Nutrition - Functional Food & Vascular Health', 5000 Monastir, Tunisia. Electronic address: sonia.hammami@fmm.rnu.tn.

Mohamed Hammami (M)

University of Monastir, Faculty of Medicine, LR12ES05, Lab-NAFS 'Nutrition - Functional Food & Vascular Health', 5000 Monastir, Tunisia. Electronic address: mohamed.hammami@fmm.rnu.tn.

John J Mackrill (JJ)

Department of Physiology, School of Medicine, University College Cork, Cork, Ireland. Electronic address: J.Mackrill@ucc.ie.

Mohammad Samadi (M)

LCPMC-A2, ICPM, Dept of Chemistry, Univ. Lorraine, Metz Technopôle, Metz, France. Electronic address: mohammad.samadi@univ-lorraine.fr.

Taoufik Ghrairi (T)

University Tunis-El Manar, Loboratory of Neurophysiology, Cellular Physiopathology and Valorisation of BioMoleecules, LR18ES03, Department of Biologie, Faculty of Sciences, 2092 Tunis, Tunisia. Electronic address: taoufik.ghrairi@fst.utm.tn.

Anne Vejux (A)

University Bourgogne Franche-Comté, Team 'Biochemistry of the Peroxisome, Inflammation and Lipid Metabolism' EA 7270 / Inserm, 21000 Dijon, France. Electronic address: anne.vejux@u-bourgogne.fr.

Gérard Lizard (G)

University Bourgogne Franche-Comté, Team 'Biochemistry of the Peroxisome, Inflammation and Lipid Metabolism' EA 7270 / Inserm, 21000 Dijon, France. Electronic address: gerard.lizard@u-bourgogne.fr.

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