A case report: Amplatzer occluder device closure of an iatrogenic ventricular septal defect following radiofrequency ablation.

Occluder device Premature ventricular contractions Radiofrequency ablation Ventricular septal defect

Journal

European heart journal. Case reports
ISSN: 2514-2119
Titre abrégé: Eur Heart J Case Rep
Pays: England
ID NLM: 101730741

Informations de publication

Date de publication:
Apr 2021
Historique:
received: 19 06 2020
revised: 08 07 2020
accepted: 28 02 2021
entrez: 14 6 2021
pubmed: 15 6 2021
medline: 15 6 2021
Statut: epublish

Résumé

Interventricular septal perforation is an extremely rare complication of radiofrequency ablation (RFA), with an incidence of 1%. The most common mechanism is a 'steam pop', which can be described as 'mini-explosions' of gas bubbles. Data for percutaneous repair of cardiac perforations due to RFA are limited. A 78-year-old female patient was referred to our department for the treatment of two iatrogenic ventricular septal defects (VSDs) following radiofrequency ablation (RFA) of premature ventricular contractions. One week post-ablation, chest pain and progressive dyspnoea occurred. Transthoracic echocardiography detected a VSD, diameter 10 mm. Hence, iatrogenic, RFA-related myocardial injury was considered the most likely cause of VSD, and the patient was referred to our tertiary care centre for surgical repair. Cardiovascular magnetic resonance (CMR) imaging demonstrated border-zone oedema of the VSD only and confirmed the absence of necrotic tissue boundaries, and the patient was deemed suitable for percutaneous device closure. Laevocardiography identified an additional, smaller muscular defect that cannot be explained by analysing the Carto-Map. Both defects could be successfully closed percutaneously using two Amplatzer VSD occluder devices. In conclusion, this case demonstrates a successful percutaneous closure of a VSD resulting from RFA using an Amplatzer septal occluder device. CMR might improve tissue characterization of the VSD borders and support the decision if to opt for interventional or surgical closure.

Sections du résumé

BACKGROUND BACKGROUND
Interventricular septal perforation is an extremely rare complication of radiofrequency ablation (RFA), with an incidence of 1%. The most common mechanism is a 'steam pop', which can be described as 'mini-explosions' of gas bubbles. Data for percutaneous repair of cardiac perforations due to RFA are limited.
CASE SUMMARY METHODS
A 78-year-old female patient was referred to our department for the treatment of two iatrogenic ventricular septal defects (VSDs) following radiofrequency ablation (RFA) of premature ventricular contractions. One week post-ablation, chest pain and progressive dyspnoea occurred. Transthoracic echocardiography detected a VSD, diameter 10 mm. Hence, iatrogenic, RFA-related myocardial injury was considered the most likely cause of VSD, and the patient was referred to our tertiary care centre for surgical repair. Cardiovascular magnetic resonance (CMR) imaging demonstrated border-zone oedema of the VSD only and confirmed the absence of necrotic tissue boundaries, and the patient was deemed suitable for percutaneous device closure. Laevocardiography identified an additional, smaller muscular defect that cannot be explained by analysing the Carto-Map. Both defects could be successfully closed percutaneously using two Amplatzer VSD occluder devices.
DISCUSSION CONCLUSIONS
In conclusion, this case demonstrates a successful percutaneous closure of a VSD resulting from RFA using an Amplatzer septal occluder device. CMR might improve tissue characterization of the VSD borders and support the decision if to opt for interventional or surgical closure.

Identifiants

pubmed: 34124545
doi: 10.1093/ehjcr/ytab094
pii: ytab094
pmc: PMC8188866
doi:

Types de publication

Case Reports

Langues

eng

Pagination

ytab094

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press on behalf of the European Society of Cardiology.

Références

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pubmed: 23775197
Acta Cardiol Sin. 2016 Nov;32(6):744-747
pubmed: 27899863
Adv Ther. 2016 Oct;33(10):1782-1796
pubmed: 27554091

Auteurs

Anna Michaelis (A)

Department of Pediatric Cardiology, University of Leipzig-Heart Center, Strümpellstr. 39, 04289 Leipzig, Germany.

Ingo Dähnert (I)

Department of Pediatric Cardiology, University of Leipzig-Heart Center, Strümpellstr. 39, 04289 Leipzig, Germany.

Frank-Thomas Riede (FT)

Department of Pediatric Cardiology, University of Leipzig-Heart Center, Strümpellstr. 39, 04289 Leipzig, Germany.

Ingo Paetsch (I)

Department of Electrophysiology, University of Leipzig-Heart Center, Strümpellstr. 39, 04289 Leipzig, Germany.

Cosima Jahnke (C)

Department of Electrophysiology, University of Leipzig-Heart Center, Strümpellstr. 39, 04289 Leipzig, Germany.

Christian Paech (C)

Department of Pediatric Cardiology, University of Leipzig-Heart Center, Strümpellstr. 39, 04289 Leipzig, Germany.

Classifications MeSH