A
A2B receptors
K+ channels
adenosine
oligodendrocyte differentiation
oligodendrocyte progenitor cells (OPCs)
remyelination
sphingosine kinase (SphK)
sphingosine-1-phosphate
Journal
Frontiers in neuroscience
ISSN: 1662-4548
Titre abrégé: Front Neurosci
Pays: Switzerland
ID NLM: 101478481
Informations de publication
Date de publication:
2021
2021
Historique:
received:
08
03
2021
accepted:
22
04
2021
entrez:
17
6
2021
pubmed:
18
6
2021
medline:
18
6
2021
Statut:
epublish
Résumé
Oligodendrocyte-formed myelin sheaths allow fast synaptic transmission in the brain. Impairments in the process of myelination, or demyelinating insults, might cause chronic diseases such as multiple sclerosis (MS). Under physiological conditions, remyelination is an ongoing process throughout adult life consisting in the differentiation of oligodendrocyte progenitor cells (OPCs) into mature oligodendrocytes (OLs). During pathological events, this process fails due to unfavorable environment. Adenosine and sphingosine kinase/sphingosine 1-phosphate signaling axes (SphK/S1P) play important roles in remyelination processes. Remarkably, fingolimod (FTY720), a sphingosine analog recently approved for MS treatment, plays important roles in OPC maturation. We recently demonstrated that the selective stimulation of A
Identifiants
pubmed: 34135730
doi: 10.3389/fnins.2021.677988
pmc: PMC8202686
doi:
Types de publication
Journal Article
Review
Langues
eng
Pagination
677988Informations de copyright
Copyright © 2021 Coppi, Cencetti, Cherchi, Venturini, Donati, Bruni, Pedata and Pugliese.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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