Efficacy and safety of switching from reference adalimumab to CT-P17 (100 mg/ml): 52-week randomized, double-blind study in rheumatoid arthritis.


Journal

Rheumatology (Oxford, England)
ISSN: 1462-0332
Titre abrégé: Rheumatology (Oxford)
Pays: England
ID NLM: 100883501

Informations de publication

Date de publication:
11 04 2022
Historique:
received: 08 03 2021
revised: 17 05 2021
pubmed: 19 6 2021
medline: 14 4 2022
entrez: 18 6 2021
Statut: ppublish

Résumé

To compare the safety and efficacy of switching from reference adalimumab to adalimumab biosimilar CT-P17 with continuing reference adalimumab/CT-P17 in active RA. This double-blind, phase III study randomized (1:1) subjects with active RA to receive 40 mg (100 mg/ml) CT-P17 or European Union-sourced reference adalimumab subcutaneously every 2 weeks (Q2W) until week (W) 24 [treatment period (TP) 1]. Thereafter, subjects receiving reference adalimumab were randomized (1:1) to continue reference adalimumab or switch to CT-P17 from W26 (both Q2W until W48; TP2). Subjects receiving CT-P17 in TP1 continued CT-P17. W0-W24 results were previously reported; we present W26-W52 findings. End points were efficacy (including joint damage progression), pharmacokinetics, safety and immunogenicity. Of 607 subjects who initiated TP2 treatment, 303 continued CT-P17, 153 continued reference adalimumab and 151 switched to CT-P17. Efficacy improvements up to W24 were maintained during TP2; efficacy was comparable among groups. At W52, 20% improvement in ACR response rates were 80.5% (continued CT-P17), 77.8% (continued reference adalimumab) and 82.2% (switched to CT-P17). Joint damage progression was minimal. Mean trough serum adalimumab concentrations were similar among groups. CT-P17 and reference adalimumab safety profiles were numerically similar and switching did not affect immunogenicity. At W52, 28.4% (continued CT-P17), 27.0% (continued reference adalimumab) and 28.3% (switched to CT-P17) of subjects were anti-drug antibody-positive. Efficacy, pharmacokinetics, safety and immunogenicity of CT-P17 and reference adalimumab were comparable after 1 year of treatment, including after switching from reference adalimumab to CT-P17. ClinicalTrials.gov, http://clinicaltrials.gov, NCT03789292.

Identifiants

pubmed: 34142111
pii: 6303620
doi: 10.1093/rheumatology/keab460
pmc: PMC8996790
doi:

Substances chimiques

Antirheumatic Agents 0
Biosimilar Pharmaceuticals 0
Adalimumab FYS6T7F842

Banques de données

ClinicalTrials.gov
['NCT03789292']

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1385-1395

Commentaires et corrections

Type : CommentIn

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press on behalf of the British Society for Rheumatology.

Références

Arthritis Rheumatol. 2018 Jun;70(6):832-840
pubmed: 29439289
Clin Transl Sci. 2021 Jul;14(4):1280-1291
pubmed: 33503313
Arthritis Res Ther. 2019 Dec 12;21(1):281
pubmed: 31831079
Ann Rheum Dis. 2015 Mar;74(3):513-8
pubmed: 24326008
Arthritis Res Ther. 2021 Feb 5;23(1):51
pubmed: 33546755
BioDrugs. 2020 Dec;34(6):809-823
pubmed: 33119861
Ann Rheum Dis. 2018 Jun;77(6):914-921
pubmed: 29514803
Arthritis Res Ther. 2019 Mar 29;21(1):84
pubmed: 30922373
Ann Rheum Dis. 2017 Oct;76(10):1679-1687
pubmed: 28584187
Br J Clin Pharmacol. 2021 Nov;87(11):4323-4333
pubmed: 33822406
Br J Clin Pharmacol. 2020 Nov;86(11):2274-2285
pubmed: 32363771

Auteurs

Daniel E Furst (DE)

Division of Rheumatology, Department of Medicine, University of California, Los Angeles, Los Angeles, CA.
Division of Rheumatology, Department of Medicine, University of Washington, Seattle, WA, USA.
Department of Medicine, University of Florence, Florence, Italy.

Janusz Jaworski (J)

Department of Rheumatology, Reumatika-Centrum Reumatologii, Warsaw.

Rafal Wojciechowski (R)

Department of Rheumatology and Connective Tissue Diseases, University Hospital No 2, Bydgoszcz.

Piotr Wiland (P)

Department of Rheumatology and Internal Diseases, Medical University, Wrocław.

Anna Dudek (A)

Department of Rheumatology, Centrum Medyczne AMED, Warsaw.

Marek Krogulec (M)

Rheumatology Clinic, NZOZ Lecznica MAK-MED, Nadarzyn.

Slawomir Jeka (S)

Department of Rheumatology and Connective Tissue Diseases, Nasz Lekarz Przychodnie Medyczne, Toruń.

Agnieszka Zielinska (A)

Medycyna Kliniczna.

Jakub Trefler (J)

Reuma Centrum, Warsaw.

Katarzyna Bartnicka-Maslowska (K)

Department of Rheumatology, Centrum Medyczne Amed, Łódź.

Magdalena Krajewska-Wlodarczyk (M)

Department of Internal Medicine, University of Warmia and Mazury, Olsztyn.

Piotr A Klimiuk (PA)

Department of Rheumatology and Internal Diseases, Medical University of Bialystok and Gabinet Internistyczno-Reumatologiczny Piotr Adrian Klimiuk, Białystok, Poland.

Sang Joon Lee (SJ)

Clinical Development Division, Celltrion, Inc., Incheon, Republic of Korea.

Sung Hyun Kim (SH)

Clinical Development Division, Celltrion, Inc., Incheon, Republic of Korea.

Yun Ju Bae (YJ)

Clinical Development Division, Celltrion, Inc., Incheon, Republic of Korea.

Go Eun Yang (GE)

Clinical Development Division, Celltrion, Inc., Incheon, Republic of Korea.

Jae Kyoung Yoo (JK)

Clinical Development Division, Celltrion, Inc., Incheon, Republic of Korea.

Jonathan Kay (J)

Division of Rheumatology, Department of Medicine, University of Massachusetts Medical School and UMass Memorial Medical Center.
Division of Epidemiology, Department of Population and Quantitative Health Sciences, University of Massachusetts Medical School, Worcester, MA, USA.

Edward Keystone (E)

Department of Rheumatology, University of Toronto, Toronto, ON, Canada.

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Classifications MeSH