Microtubule destabilization is a critical checkpoint of chemotaxis and transendothelial migration in melanoma cells but not in T cells.


Journal

Cell adhesion & migration
ISSN: 1933-6926
Titre abrégé: Cell Adh Migr
Pays: United States
ID NLM: 101469464

Informations de publication

Date de publication:
12 2021
Historique:
entrez: 21 6 2021
pubmed: 22 6 2021
medline: 15 12 2021
Statut: ppublish

Résumé

Microtubules (MTs) control cell shape and intracellular cargo transport. The role of MT turnover in the migration of slow-moving cells through endothelial barriers remains unclear. To irreversibly interfere with MT disassembly, we have used the MT-stabilizing agent zampanolide (ZMP) in Β16F10 melanoma as amodel of slow-moving cells. ZMP-treated B16 cells failed to follow chemotactic gradients across rigid confinements and could not generate stable sub-endothelial pseudopodia under endothelial monolayers. In vivo, ZMP-treated Β16 cells failed to extravasate though lung capillaries. In contrast to melanoma cells, the chemotaxis and transendothelial migration of ZMP-treated Tcells were largely conserved. This is afirst demonstration that MT disassembly is akey checkpoint in the directional migration of cancer cells but not of lymphocytes.

Identifiants

pubmed: 34152257
doi: 10.1080/19336918.2021.1934958
pmc: PMC8218694
doi:

Substances chimiques

Macrolides 0
zampanolide 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

166-179

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Auteurs

Francesco Roncato (F)

Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.

Ofer Regev (O)

Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.

Sandeep Kumar Yadav (SK)

Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.

Ronen Alon (R)

Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.

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