The ATF6β-calreticulin axis promotes neuronal survival under endoplasmic reticulum stress and excitotoxicity.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
22 06 2021
Historique:
received: 01 03 2021
accepted: 09 06 2021
entrez: 23 6 2021
pubmed: 24 6 2021
medline: 9 11 2021
Statut: epublish

Résumé

While ATF6α plays a central role in the endoplasmic reticulum (ER) stress response, the function of its paralogue ATF6β remains elusive, especially in the central nervous system (CNS). Here, we demonstrate that ATF6β is highly expressed in the hippocampus of the brain, and specifically regulates the expression of calreticulin (CRT), a molecular chaperone in the ER with a high Ca

Identifiants

pubmed: 34158584
doi: 10.1038/s41598-021-92529-w
pii: 10.1038/s41598-021-92529-w
pmc: PMC8219835
doi:

Substances chimiques

Activating Transcription Factor 6 0
Atf6b protein, mouse 0
Calreticulin 0
Kainic Acid SIV03811UC

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

13086

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Auteurs

Dinh Thi Nguyen (DT)

Department of Neuroanatomy, Graduate School of Medical Sciences, Kanazawa University, 13-1 Takara-Machi, Kanazawa City, Ishikawa, 920-8640, Japan.

Thuong Manh Le (TM)

Department of Neuroanatomy, Graduate School of Medical Sciences, Kanazawa University, 13-1 Takara-Machi, Kanazawa City, Ishikawa, 920-8640, Japan.
Department of Human Anatomy, Hanoi Medical University, Hanoi, Vietnam.

Tsuyoshi Hattori (T)

Department of Neuroanatomy, Graduate School of Medical Sciences, Kanazawa University, 13-1 Takara-Machi, Kanazawa City, Ishikawa, 920-8640, Japan.

Mika Takarada-Iemata (M)

Department of Neuroanatomy, Graduate School of Medical Sciences, Kanazawa University, 13-1 Takara-Machi, Kanazawa City, Ishikawa, 920-8640, Japan.

Hiroshi Ishii (H)

Department of Neuroanatomy, Graduate School of Medical Sciences, Kanazawa University, 13-1 Takara-Machi, Kanazawa City, Ishikawa, 920-8640, Japan.

Jureepon Roboon (J)

Department of Neuroanatomy, Graduate School of Medical Sciences, Kanazawa University, 13-1 Takara-Machi, Kanazawa City, Ishikawa, 920-8640, Japan.

Takashi Tamatani (T)

Department of Neuroanatomy, Graduate School of Medical Sciences, Kanazawa University, 13-1 Takara-Machi, Kanazawa City, Ishikawa, 920-8640, Japan.

Takayuki Kannon (T)

Department of Bioinformatics and Genomics, Graduate School of Advanced Preventive Medical Sciences, Kanazawa University, Kanazawa, Japan.

Kazuyoshi Hosomichi (K)

Department of Bioinformatics and Genomics, Graduate School of Advanced Preventive Medical Sciences, Kanazawa University, Kanazawa, Japan.

Atsushi Tajima (A)

Department of Bioinformatics and Genomics, Graduate School of Advanced Preventive Medical Sciences, Kanazawa University, Kanazawa, Japan.

Shusuke Taniuchi (S)

Division of Molecular Biology, Institute for Genome Research, Institute of Advanced Medical Sciences, Tokushima University, Tokushima, Japan.

Masato Miyake (M)

Division of Molecular Biology, Institute for Genome Research, Institute of Advanced Medical Sciences, Tokushima University, Tokushima, Japan.

Seiichi Oyadomari (S)

Division of Molecular Biology, Institute for Genome Research, Institute of Advanced Medical Sciences, Tokushima University, Tokushima, Japan.

Takashi Tanaka (T)

Department of Anatomy II, Kanazawa Medical University, Kahoku, Japan.

Nobuo Kato (N)

Department of Physiology I, Kanazawa Medical University, Kahoku, Japan.

Shunsuke Saito (S)

Department of Biophysics, Graduate School of Science, Kyoto University, Kyoto, Japan.

Kazutoshi Mori (K)

Department of Biophysics, Graduate School of Science, Kyoto University, Kyoto, Japan.

Osamu Hori (O)

Department of Neuroanatomy, Graduate School of Medical Sciences, Kanazawa University, 13-1 Takara-Machi, Kanazawa City, Ishikawa, 920-8640, Japan. osamuh3@staff.kanazawa-u.ac.jp.

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Classifications MeSH