Clonal hematopoiesis and its emerging effects on cellular therapies.


Journal

Leukemia
ISSN: 1476-5551
Titre abrégé: Leukemia
Pays: England
ID NLM: 8704895

Informations de publication

Date de publication:
10 2021
Historique:
received: 27 03 2021
accepted: 23 06 2021
revised: 22 06 2021
pubmed: 4 7 2021
medline: 18 11 2021
entrez: 3 7 2021
Statut: ppublish

Résumé

The accumulation of somatic mutations in hematopoietic stem cells during aging, leading to clonal expansion, is linked to a higher risk of cardiovascular mortality and hematologic malignancies. Clinically, clonal hematopoiesis is associated with a pro-inflammatory phenotype of hematopoietic cells and their progeny, inflammatory conditions and a poor outcome for patients with hematologic neoplasms and solid tumors. Here, we review the relevance and complications of clonal hematopoiesis for the treatment of hematologic malignancies with cell therapeutic approaches. In autologous and allogeneic hematopoietic stem cell transplantation native hematopoietic and immune effector cells of clonal origin are transferred, which may affect outcome of the procedure. In chimeric antigen receptor modified T-cell therapy, the effectiveness may be altered by preexisting somatic mutations in genetically modified effector cells or by unmodified bystander cells harboring clonal hematopoiesis. Registry studies and carefully designed prospective trials will be required to assess the relative roles of donor- and recipient-derived individual clonal events for autologous and allogeneic cell therapies and to incorporate novel insights into therapeutic strategies.

Identifiants

pubmed: 34215849
doi: 10.1038/s41375-021-01337-8
pii: 10.1038/s41375-021-01337-8
pmc: PMC8249428
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

2752-2758

Informations de copyright

© 2021. The Author(s).

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Auteurs

Malte von Bonin (M)

Medizinische Klinik und Poliklinik 1, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany.

Helena Klara Jambor (HK)

Medizinische Klinik und Poliklinik 1, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany.

Raphael Teipel (R)

Medizinische Klinik und Poliklinik 1, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany.

Friedrich Stölzel (F)

Medizinische Klinik und Poliklinik 1, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany.

Christian Thiede (C)

Medizinische Klinik und Poliklinik 1, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany.
AgenDix, Angewandte molekulare Diagnostik mbH, Dresden, Germany.

Frederik Damm (F)

Department of Hematology, Oncology, and Tumor Immunology, Charitè-Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, and Berlin Institute of Health, Berlin, Germany.

Frank Kroschinsky (F)

Medizinische Klinik und Poliklinik 1, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany.

Johannes Schetelig (J)

Medizinische Klinik und Poliklinik 1, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany.
DKMS Clinical Trials Unit, Dresden, Germany.

Triantafyllos Chavakis (T)

Institut für Klinische Chemie und Laboratoriumsmedizin, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany.

Martin Bornhäuser (M)

Medizinische Klinik und Poliklinik 1, Universitätsklinikum Carl Gustav Carus an der TU Dresden, Dresden, Germany. martin.bornhaeuser@ukdd.de.
Nationales Centrum für Tumorerkrankungen (NCT), Partnerstandort Dresden, Dresden, Germany. martin.bornhaeuser@ukdd.de.

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