Hyperemic hemodynamic characteristics of serial coronary lesions assessed by pullback pressure gradients.


Journal

Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions
ISSN: 1522-726X
Titre abrégé: Catheter Cardiovasc Interv
Pays: United States
ID NLM: 100884139

Informations de publication

Date de publication:
01 11 2021
Historique:
revised: 08 06 2021
received: 30 03 2021
accepted: 01 07 2021
pubmed: 16 7 2021
medline: 15 12 2021
entrez: 15 7 2021
Statut: ppublish

Résumé

To characterize hemodynamics of serial coronary stenoses using fractional flow reserve (FFR) pullbacks and the pullback pressure gradients (PPG) index. The cross-talk between stenoses within the same coronary artery makes the prediction of the functional contribution of each lesion challenging. One-hundred seventeen patients undergoing coronary angiography for stable angina were prospectively recruited. Serial lesions were defined as two or more narrowings with visual diameter stenosis >50% on conventional angiography. Motorized FFR pullback tracings were obtained at 1 mm/s. Pullbacks were visually adjudicated as presenting two, one, and no focal pressure drops. The pattern of disease (i.e., focal or diffuse) was quantified using the PPG index. Twenty-five vessels presented serial lesions (mean PPG 0.48 ± 0.17). Two, one or no focal pressure drops were observed in 40% (n = 10; PPG 0.59 ± 0.17), 52% (n = 13; PPG 0.44 ± 0.12) and 8% of cases (n = 2; PPG 0.27 ± 0.01; p-value = 0.01). Distal FFR was similar between vessels with two, one and no focal pressure drops in the pullback curve (p-value = 0.27). The PPG index independently predicted the presence of two focal pressure drops in the pullback curve (p = 0.04). FFR pullbacks in serial coronary lesions exhibit three distinct functional patterns. High PPG was associated with pullback curves presenting two pressure drops. The PPG provides a quantitative assessment of the pattern of coronary artery disease in cases with serial lesions and might be useful to assess the appropriateness of percutaneous revascularization.

Sections du résumé

OBJECTIVES
To characterize hemodynamics of serial coronary stenoses using fractional flow reserve (FFR) pullbacks and the pullback pressure gradients (PPG) index.
BACKGROUND
The cross-talk between stenoses within the same coronary artery makes the prediction of the functional contribution of each lesion challenging.
METHODS AND RESULTS
One-hundred seventeen patients undergoing coronary angiography for stable angina were prospectively recruited. Serial lesions were defined as two or more narrowings with visual diameter stenosis >50% on conventional angiography. Motorized FFR pullback tracings were obtained at 1 mm/s. Pullbacks were visually adjudicated as presenting two, one, and no focal pressure drops. The pattern of disease (i.e., focal or diffuse) was quantified using the PPG index. Twenty-five vessels presented serial lesions (mean PPG 0.48 ± 0.17). Two, one or no focal pressure drops were observed in 40% (n = 10; PPG 0.59 ± 0.17), 52% (n = 13; PPG 0.44 ± 0.12) and 8% of cases (n = 2; PPG 0.27 ± 0.01; p-value = 0.01). Distal FFR was similar between vessels with two, one and no focal pressure drops in the pullback curve (p-value = 0.27). The PPG index independently predicted the presence of two focal pressure drops in the pullback curve (p = 0.04).
CONCLUSIONS
FFR pullbacks in serial coronary lesions exhibit three distinct functional patterns. High PPG was associated with pullback curves presenting two pressure drops. The PPG provides a quantitative assessment of the pattern of coronary artery disease in cases with serial lesions and might be useful to assess the appropriateness of percutaneous revascularization.

Identifiants

pubmed: 34264014
doi: 10.1002/ccd.29868
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

E647-E654

Subventions

Organisme : STAR program of the Compagnia di San Paolo
Organisme : CardioPaTh PhD program

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

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Siyeong J, Gu L. Hemodynamic interference of serial Stenoses and its impact on FFR and iFR measurements. Appl Sci. 2019;9:279.
Modi BN, Rahman H, Ryan M, et al. Comparison of fractional flow reserve, instantaneous wave-free ratio and a novel technique for assessing coronary arteries with serial lesions. EuroIntervention. 2020;16(7):577-583.
Sianos G, Morel MA, Kappetein AP, et al. The SYNTAX score: an angiographic tool grading the complexity of coronary artery disease. EuroIntervention. 2005;1(2):219-227.
De Bruyne B, Pijls NH, Heyndrickx GR, Hodeige D, Kirkeeide R, Gould KL. Pressure-derived fractional flow reserve to assess serial epicardial stenoses: theoretical basis and animal validation. Circulation. 2000;101(15):1840-1847.
Collet C, Sonck J, Vandeloo B, et al. Measurement of hyperemic pullback pressure gradients to characterize patterns of coronary atherosclerosis. J Am Coll Cardiol. 2019;74(14):1772-1784.
Toth GG, Johnson NP, Jeremias A, et al. Standardization of fractional flow reserve measurements. J Am Coll Cardiol. 2016;68(7):742-753.
Kim HL, Koo BK, Nam CW, et al. Clinical and physiological outcomes of fractional flow reserve-guided percutaneous coronary intervention in patients with serial stenoses within one coronary artery. JACC Cardiovasc Interv. 2012;5(10):1013-1018.
Pijls NH, De Bruyne B, Bech GJ, et al. Coronary pressure measurement to assess the hemodynamic significance of serial stenoses within one coronary artery: validation in humans. Circulation. 2000;102(19):2371-2377.
Modi BN, De Silva K, Rajani R, Curzen N, Perera D. Physiology-guided Management of Serial Coronary Artery Disease: a review. JAMA Cardiol. 2018;3(5):432-438.
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Auteurs

Alessandro Candreva (A)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.
Department of Cardiology, University Heart Centre, University Hospital Zurich, Zurich, Switzerland.

Takuya Mizukami (T)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.
Division of Clinical Pharmacology, Show University School of Medicine, Tokyo, Japan.

Jeroen Sonck (J)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.
Department of Advanced Biomedical Sciences, University of Naples Federico II, Naples, Italy.

Daniel Munhoz (D)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.

Sakura Nagumo (S)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.
Division of Clinical Pharmacology, Show University School of Medicine, Tokyo, Japan.

Giuseppe Di Gioia (G)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.
Department of Advanced Biomedical Sciences, University of Naples Federico II, Naples, Italy.

Emanuele Gallinoro (E)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.
Department of Translational Medical Sciences, University of Campania Luigi Vanvitelli, Naples, Italy.

Niya Mileva (N)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.

Jozef Bartunek (J)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.

Eric Wyffels (E)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.

Emanuele Barbato (E)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.
Department of Advanced Biomedical Sciences, University of Naples Federico II, Naples, Italy.

Bernard De Bruyne (B)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.
Department of Cardiology, University Hospital Lausanne, Lausanne, Switzerland.

Divaka Perera (D)

Cardiovascular Division, St. Thomas' Hospital Campus, King's College London, London, UK.

Carlos Collet (C)

Cardiovascular Center Aalst, OLV-Clinic, Aalst, Belgium.

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