Gastric epithelial neoplasm of fundic-gland mucosa lineage: proposal for a new classification in association with gastric adenocarcinoma of fundic-gland type.

Gastric adenocarcinoma of fundic-gland mucosa type Gastric adenocarcinoma of fundic-gland type Gastric epithelial neoplasm of fundic-gland mucosa lineage Oxyntic gland adenoma

Journal

Journal of gastroenterology
ISSN: 1435-5922
Titre abrégé: J Gastroenterol
Pays: Japan
ID NLM: 9430794

Informations de publication

Date de publication:
09 2021
Historique:
received: 24 04 2021
accepted: 10 07 2021
pubmed: 17 7 2021
medline: 15 12 2021
entrez: 16 7 2021
Statut: ppublish

Résumé

Gastric adenocarcinoma of fundic-gland type (GA-FG) is a rare variant of gastric neoplasia. However, the etiology, classification, and clinicopathological features of gastric epithelial neoplasm of fundic-gland mucosa lineage (GEN-FGML; generic term of GA-FG related neoplasm) are not fully elucidated. We performed a large, multicenter, retrospective study to establish a new classification and clarify the clinicopathological features of GEN-FGML. One hundred GEN-FGML lesions in 94 patients were collected from 35 institutions between 2008 and 2019. We designed a new histopathological classification of GEN-FGML using immunohistochemical analysis and analyzed via clinicopathological, immunohistochemical, and genetic evaluation. GEN-FGML was classified into 3 major types; oxyntic gland adenoma (OGA), GA-FG, and gastric adenocarcinoma of fundic-gland mucosa type (GA-FGM). In addition, GA-FGM was classified into 3 subtypes; Type 1 (organized with exposure type), Type 2 (disorganized with exposure type), and Type 3 (disorganized with non-exposure type). OGA and GA-FG demonstrated low-grade epithelial neoplasm, and GA-FGM should be categorized as an aggressive variant of GEN-FGML that demonstrated high-grade epithelial neoplasm (Type 2 > 1, 3). The frequent presence of GNAS mutation was a characteristic genetic feature of GEN-FGML (7/34, 20.6%; OGA 1/3, 33.3%; GA-FG 3/24, 12.5%; GA-FGM 3/7, 42.9%) in mutation analysis using next-generation sequencing. We have established a new histopathological classification of GEN-FGML and propose a new lineage of gastric epithelial neoplasm that harbors recurrent GNAS mutation. This classification will be useful to estimate the malignant potential of GEN-FGML and establish an appropriate standard therapeutic approach.

Sections du résumé

BACKGROUND
Gastric adenocarcinoma of fundic-gland type (GA-FG) is a rare variant of gastric neoplasia. However, the etiology, classification, and clinicopathological features of gastric epithelial neoplasm of fundic-gland mucosa lineage (GEN-FGML; generic term of GA-FG related neoplasm) are not fully elucidated. We performed a large, multicenter, retrospective study to establish a new classification and clarify the clinicopathological features of GEN-FGML.
METHODS
One hundred GEN-FGML lesions in 94 patients were collected from 35 institutions between 2008 and 2019. We designed a new histopathological classification of GEN-FGML using immunohistochemical analysis and analyzed via clinicopathological, immunohistochemical, and genetic evaluation.
RESULTS
GEN-FGML was classified into 3 major types; oxyntic gland adenoma (OGA), GA-FG, and gastric adenocarcinoma of fundic-gland mucosa type (GA-FGM). In addition, GA-FGM was classified into 3 subtypes; Type 1 (organized with exposure type), Type 2 (disorganized with exposure type), and Type 3 (disorganized with non-exposure type). OGA and GA-FG demonstrated low-grade epithelial neoplasm, and GA-FGM should be categorized as an aggressive variant of GEN-FGML that demonstrated high-grade epithelial neoplasm (Type 2 > 1, 3). The frequent presence of GNAS mutation was a characteristic genetic feature of GEN-FGML (7/34, 20.6%; OGA 1/3, 33.3%; GA-FG 3/24, 12.5%; GA-FGM 3/7, 42.9%) in mutation analysis using next-generation sequencing.
CONCLUSIONS
We have established a new histopathological classification of GEN-FGML and propose a new lineage of gastric epithelial neoplasm that harbors recurrent GNAS mutation. This classification will be useful to estimate the malignant potential of GEN-FGML and establish an appropriate standard therapeutic approach.

Identifiants

pubmed: 34268625
doi: 10.1007/s00535-021-01813-z
pii: 10.1007/s00535-021-01813-z
pmc: PMC8370942
doi:

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

814-828

Informations de copyright

© 2021. The Author(s).

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Auteurs

Hiroya Ueyama (H)

Department of Gastroenterology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-Ku, Tokyo, 113-8421, Japan. psyro@juntendo.ac.jp.

Takashi Yao (T)

Department of Human Pathology, Juntendo University Graduate School of Medicine, Tokyo, Japan.

Yoichi Akazawa (Y)

Department of Gastroenterology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-Ku, Tokyo, 113-8421, Japan.

Takuo Hayashi (T)

Department of Human Pathology, Juntendo University School of Medicine, Tokyo, Japan.

Koichi Kurahara (K)

Department of Gastroenterology, Matsuyama Red Cross Hospital, Ehime, Japan.

Yumi Oshiro (Y)

Department of Pathology, Matsuyama Red Cross Hospital, Ehime, Japan.

Masayoshi Yamada (M)

Endoscopy Division, National Cancer Center Hospital, Tokyo, Japan.

Ichiro Oda (I)

Endoscopy Division, National Cancer Center Hospital, Tokyo, Japan.

Shin Fujioka (S)

Division of Gastroenterology, Fukuoka Red Cross Hospital, Fukuoka, Japan.

Chiaki Kusumoto (C)

Department of Gastroenterology, Nippon Kokan Fukuyama Hospital, Hiroshima, Japan.

Masayoshi Fukuda (M)

Department of Gastroenterology and Hepatology, Tokyo Medical and Dental University, Tokyo, Japan.

Kunihisa Uchita (K)

Department of Gastroenterology, Kochi Red Cross Hospital, Kochi, Japan.

Tomohiro Kadota (T)

Department of Gastroenterology and Endoscopy, National Cancer Center Hospital East, Chiba, Japan.

Yasuhiro Oono (Y)

Department of Gastroenterology and Endoscopy, National Cancer Center Hospital East, Chiba, Japan.

Kazuhisa Okamoto (K)

Department of Gastroenterology, Faculty of Medicine, Oita University, Oita, Japan.

Kazunari Murakami (K)

Department of Gastroenterology, Faculty of Medicine, Oita University, Oita, Japan.

Yasumasa Matsuo (Y)

Division of Gastroenterology and Hepatology, Department of Internal Medicine, St. Marianna University School of Medicine, Kanagawa, Japan.

Motohiko Kato (M)

Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo, Japan.
Division of Research and Development for Minimally Invasive Treatment, Cancer Center, Keio University School of Medicine, Tokyo, Japan.

Tadateru Maehata (T)

Division of Research and Development for Minimally Invasive Treatment, Cancer Center, Keio University School of Medicine, Tokyo, Japan.

Naohisa Yahagi (N)

Division of Research and Development for Minimally Invasive Treatment, Cancer Center, Keio University School of Medicine, Tokyo, Japan.

Yumiko Yasuhara (Y)

Department of Diagnostic Pathology, Kyoto-Katsura Hospital, Kyoto, Japan.

Tomoyuki Yada (T)

Department of Gastroenterology & Hepatology, National Center for Global Health and Medicine, Kohnodai Hospital, Chiba, Japan.

Koji Uraushihara (K)

Department of Gastroenterology and Hepatology, Showa General Hospital, Tokyo, Japan.

Tetsumi Yamane (T)

Department of Diagnostic Pathology, Tottori Red Cross Hospital, Tottori, Japan.

Taiji Matsuo (T)

Department of Endoscopy, Hiroshima University Hospital, Hiroshima, Japan.

Masanori Ito (M)

Department of General Internal Medicine, Hiroshima University Hospital, Hiroshima, Japan.

Yasuhiko Maruyama (Y)

Department of Gastroenterology, Fujieda Municipal General Hospital, Shizuoka, Japan.

Ayumi Osako (A)

Department of Gastroenterology, Tottori Seikyo Hospital, Tottori, Japan.

Shoko Ono (S)

Department of Gastroenterology, Hokkaido University Hospital, Hokkaido, Japan.

Mototsugu Kato (M)

Department of Gastroenterology, National Hospital Organization Hakodate National Hospital, Hokkaido, Japan.

Kazuyoshi Yagi (K)

Department of Gastroenterology and Hepatology, Uonuma Institute of Community Medicine, Niigata University Medical and Dental Hospital, Niigata, Japan.

Takashi Hashimoto (T)

Department of Esophageal and Gastroenterological Surgery, Juntendo University School of Medicine, Tokyo, Japan.

Natsumi Tomita (N)

Department of Esophageal and Gastroenterological Surgery, Juntendo University School of Medicine, Tokyo, Japan.

Sho Tsuyama (S)

Department of Human Pathology, Juntendo University School of Medicine, Tokyo, Japan.

Tsuyoshi Saito (T)

Department of Human Pathology, Juntendo University School of Medicine, Tokyo, Japan.

Kohei Matsumoto (K)

Department of Gastroenterology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-Ku, Tokyo, 113-8421, Japan.

Kenshi Matsumoto (K)

Department of Gastroenterology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-Ku, Tokyo, 113-8421, Japan.

Sumio Watanabe (S)

Department of Gastroenterology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-Ku, Tokyo, 113-8421, Japan.

Naomi Uemura (N)

Department of Gastroenterology & Hepatology, National Center for Global Health and Medicine, Kohnodai Hospital, Chiba, Japan.

Tsutomu Chiba (T)

Department of Gastroenterology and Hepatology, Kyoto University Graduate School of Medicine, Kyoto, Japan.
Kansai Electric Power Hospital, Osaka, Japan.

Akihito Nagahara (A)

Department of Gastroenterology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-Ku, Tokyo, 113-8421, Japan.

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