KCNE4-dependent functional consequences of Kv1.3-related leukocyte physiology.
Animals
Cell Line
Cell Membrane
/ metabolism
Dendritic Cells
/ metabolism
Gene Knockdown Techniques
Humans
Immunity
Immunological Synapses
/ physiology
Interleukin-2
/ metabolism
Ion Channel Gating
Jurkat Cells
Kv1.3 Potassium Channel
/ physiology
Leukocytes
/ physiology
Mice
Potassium Channels, Voltage-Gated
/ physiology
Journal
Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288
Informations de publication
Date de publication:
16 07 2021
16 07 2021
Historique:
received:
09
03
2021
accepted:
28
06
2021
entrez:
17
7
2021
pubmed:
18
7
2021
medline:
9
11
2021
Statut:
epublish
Résumé
The voltage-dependent potassium channel Kv1.3 plays essential roles in the immune system, participating in leukocyte activation, proliferation and apoptosis. The regulatory subunit KCNE4 acts as an ancillary peptide of Kv1.3, modulates K
Identifiants
pubmed: 34272451
doi: 10.1038/s41598-021-94015-9
pii: 10.1038/s41598-021-94015-9
pmc: PMC8285421
doi:
Substances chimiques
Interleukin-2
0
KCNE4 protein, human
0
KCNE4 protein, mouse
0
Kv1.3 Potassium Channel
0
Potassium Channels, Voltage-Gated
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
14632Informations de copyright
© 2021. The Author(s).
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