Confirming Genetic Abnormalities of Hypokalemic Periodic Paralysis Using Next-Generation Sequencing: A Case Report and Literature Review.

Arrhythmia Genetic diseases Hypokalemic periodic paralysis

Journal

Electrolyte & blood pressure : E & BP
ISSN: 1738-5997
Titre abrégé: Electrolyte Blood Press
Pays: Korea (South)
ID NLM: 101249217

Informations de publication

Date de publication:
Jun 2021
Historique:
received: 23 03 2021
revised: 12 05 2021
accepted: 13 05 2021
entrez: 22 7 2021
pubmed: 23 7 2021
medline: 23 7 2021
Statut: ppublish

Résumé

Hypokalemic periodic paralysis (hypoPP) is a disorder characterized by episodic, short-lived, and hypo-reflexive skeletal muscle weakness. HypoPP is a rare disease caused by genetic mutations related to expression of sodium or calcium ion channels. Most mutations are associated with autosomal dominant inheritance, but some are found in patients with no relevant family history. A 28-year-old man who visited the emergency room for paralytic attack was assessed in this study. He exhibited motor weakness in four limbs. There was no previous medical history or family history. The initial electrocardiogram showed a flat T wave and QT prolongation. His blood test was delayed, and sudden hypotension and bradycardia were observed. The blood test showed severe hypokalemia. After correcting hypokalemia, his muscle paralysis recovered without any neurological deficits. The patient's thyroid function and long exercise test results were normal. However, because of the history of high carbohydrate diet and exercise, hypoPP was suspected. Hence, next-generation sequencing (NGS) was performed, and a mutation of Arg669His was noted in the

Identifiants

pubmed: 34290819
doi: 10.5049/EBP.2021.19.1.10
pmc: PMC8267070
doi:

Types de publication

Case Reports

Langues

eng

Pagination

10-14

Informations de copyright

Copyright © 2021 Korean Society for Electrolyte and Blood Pressure Research.

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Auteurs

Hae Ri Kim (HR)

Department of Internal Medicine, Division of Nephrology, Chungnam National University Sejong Hospital, Sejong, Republic of Korea.

Jae Wan Jeon (JW)

Department of Internal Medicine, Division of Nephrology, Chungnam National University Sejong Hospital, Sejong, Republic of Korea.

Eu Jin Lee (EJ)

Department of Internal Medicine, Division of Nephrology, Chungnam National University Hospital, Daejeon, Republic of Korea.

Young Rok Ham (YR)

Department of Internal Medicine, Division of Nephrology, Chungnam National University Hospital, Daejeon, Republic of Korea.

Ki Ryang Na (KR)

Department of Internal Medicine, Division of Nephrology, Chungnam National University Hospital, Daejeon, Republic of Korea.

Kang Wook Lee (KW)

Department of Internal Medicine, Division of Nephrology, Chungnam National University Hospital, Daejeon, Republic of Korea.

Kee Hong Park (KH)

Department of Neurology, Seoul Medical Center, Seoul, Republic of Korea.

Seon Young Kim (SY)

Department of Laboratory Medicine, Chungnam National University Hospital, Daejeon, Republic of Korea.

Dae Eun Choi (DE)

Department of Internal Medicine, Division of Nephrology, Chungnam National University Hospital, Daejeon, Republic of Korea.

Classifications MeSH