HUWE1 employs a giant substrate-binding ring to feed and regulate its HECT E3 domain.


Journal

Nature chemical biology
ISSN: 1552-4469
Titre abrégé: Nat Chem Biol
Pays: United States
ID NLM: 101231976

Informations de publication

Date de publication:
10 2021
Historique:
received: 14 02 2021
accepted: 08 06 2021
pubmed: 24 7 2021
medline: 13 10 2021
entrez: 23 7 2021
Statut: ppublish

Résumé

HUWE1 is a universal quality-control E3 ligase that marks diverse client proteins for proteasomal degradation. Although the giant HECT enzyme is an essential component of the ubiquitin-proteasome system closely linked with severe human diseases, its molecular mechanism is little understood. Here, we present the crystal structure of Nematocida HUWE1, revealing how a single E3 enzyme has specificity for a multitude of unrelated substrates. The protein adopts a remarkable snake-like structure, where the C-terminal HECT domain heads an extended alpha-solenoid body that coils in on itself and houses various protein-protein interaction modules. Our integrative structural analysis shows that this ring structure is highly dynamic, enabling the flexible HECT domain to reach protein targets presented by the various acceptor sites. Together, our data demonstrate how HUWE1 is regulated by its unique structure, adapting a promiscuous E3 ligase to selectively target unassembled orphan proteins.

Identifiants

pubmed: 34294896
doi: 10.1038/s41589-021-00831-5
pii: 10.1038/s41589-021-00831-5
pmc: PMC7611724
mid: EMS127426
doi:

Substances chimiques

Caenorhabditis elegans Proteins 0
Fungal Proteins 0
Ubiquitin-Protein Ligases EC 2.3.2.27

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1084-1092

Subventions

Organisme : European Research Council
ID : 694978
Pays : International
Organisme : Austrian Science Fund FWF
ID : F 7905
Pays : Austria

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer Nature America, Inc.

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Auteurs

Daniel B Grabarczyk (DB)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria. daniel.grabarczyk@imp.ac.at.

Olga A Petrova (OA)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Luiza Deszcz (L)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Robert Kurzbauer (R)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Paul Murphy (P)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Juraj Ahel (J)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Antonia Vogel (A)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Rebeca Gogova (R)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Victoria Faas (V)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Darja Kordic (D)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Alexander Schleiffer (A)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Anton Meinhart (A)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Richard Imre (R)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Anita Lehner (A)

Vienna Biocenter Core Facilities, Vienna BioCenter, Vienna, Austria.

Jana Neuhold (J)

Vienna Biocenter Core Facilities, Vienna BioCenter, Vienna, Austria.

Gerd Bader (G)

Boehringer Ingelheim RCV, Vienna, Austria.

Peggy Stolt-Bergner (P)

Boehringer Ingelheim RCV, Vienna, Austria.

Jark Böttcher (J)

Boehringer Ingelheim RCV, Vienna, Austria.

Bernhard Wolkerstorfer (B)

Boehringer Ingelheim RCV, Vienna, Austria.

Gerhard Fischer (G)

Boehringer Ingelheim RCV, Vienna, Austria.

Irina Grishkovskaya (I)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

David Haselbach (D)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria.

Dirk Kessler (D)

Boehringer Ingelheim RCV, Vienna, Austria.

Tim Clausen (T)

Research Institute of Molecular Pathology, Vienna BioCenter, Vienna, Austria. tim.clausen@imp.ac.at.

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