Differences in the number of de novo mutations between individuals are due to small family-specific effects and stochasticity.


Journal

Genome research
ISSN: 1549-5469
Titre abrégé: Genome Res
Pays: United States
ID NLM: 9518021

Informations de publication

Date de publication:
09 2021
Historique:
received: 18 09 2020
accepted: 14 07 2021
pubmed: 25 7 2021
medline: 11 3 2022
entrez: 24 7 2021
Statut: ppublish

Résumé

The number of de novo mutations (DNMs) in the human germline is correlated with parental age at conception, but this explains only part of the observed variation. We investigated whether there is a family-specific contribution to the number of DNMs in offspring. The analysis of DNMs in 111 dizygotic twin pairs did not identify a substantial family-specific contribution. This result was corroborated by comparing DNMs of 1669 siblings to those of age-matched unrelated offspring following correction for parental age. In addition, by modeling DNM data from 1714 multi-offspring families, we estimated that the family-specific contribution explains ∼5.2% of the variation in DNM number. Furthermore, we found no substantial difference between the observed number of DNMs and those predicted by a stochastic Poisson process. We conclude that there is a small family-specific contribution to DNM number and that stochasticity explains a large proportion of variation in DNM counts.

Identifiants

pubmed: 34301630
pii: gr.271809.120
doi: 10.1101/gr.271809.120
pmc: PMC8415378
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1513-1518

Informations de copyright

© 2021 Goldmann et al.; Published by Cold Spring Harbor Laboratory Press.

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Auteurs

Jakob M Goldmann (JM)

Department of Human Genetics, Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, Nijmegen 6525 GA, The Netherlands.
Department of Human Genetics, Donders Centre for Neuroscience, Radboud University Medical Center, Nijmegen 6525 GA, The Netherlands.

Juliet E Hampstead (JE)

Department of Human Genetics, Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, Nijmegen 6525 GA, The Netherlands.
Department of Human Genetics, Donders Centre for Neuroscience, Radboud University Medical Center, Nijmegen 6525 GA, The Netherlands.

Wendy S W Wong (WSW)

Inova Translational Medicine Institute (ITMI), Inova Health Systems, Falls Church, Virginia 22042, USA.

Amy B Wilfert (AB)

Department of Genome Sciences, University of Washington School of Medicine, Seattle, Washington 98195, USA.

Tychele N Turner (TN)

Department of Genome Sciences, University of Washington School of Medicine, Seattle, Washington 98195, USA.

Marianne A Jonker (MA)

Department for Health Evidence, Radboud University Medical Center, Nijmegen 6525 GA, The Netherlands.

Raphael Bernier (R)

Department of Psychiatry and Behavioral Sciences, University of Washington, Seattle, Washington 98195, USA.

Martijn A Huynen (MA)

Centre for Molecular and Biomolecular Informatics, Radboud University Medical Centre, Nijmegen 6525 GA, The Netherlands.

Evan E Eichler (EE)

Department of Genome Sciences, University of Washington School of Medicine, Seattle, Washington 98195, USA.
Howard Hughes Medical Institute, University of Washington, Seattle, Washington 98195, USA.

Joris A Veltman (JA)

Biosciences Institute, Faculty of Medical Sciences, Newcastle University, Newcastle upon Tyne NE2 4HH, United Kingdom.

George L Maxwell (GL)

Department of Obstetrics and Gynecology, Inova Fairfax Department and Inova Schar Cancer Institute, Falls Church, Virginia 22042, USA.

Christian Gilissen (C)

Department of Human Genetics, Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, Nijmegen 6525 GA, The Netherlands.
Department of Human Genetics, Donders Centre for Neuroscience, Radboud University Medical Center, Nijmegen 6525 GA, The Netherlands.

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