Machinery, regulation and pathophysiological implications of autophagosome maturation.


Journal

Nature reviews. Molecular cell biology
ISSN: 1471-0080
Titre abrégé: Nat Rev Mol Cell Biol
Pays: England
ID NLM: 100962782

Informations de publication

Date de publication:
11 2021
Historique:
accepted: 17 06 2021
pubmed: 25 7 2021
medline: 26 11 2021
entrez: 24 7 2021
Statut: ppublish

Résumé

Autophagy is a versatile degradation system for maintaining cellular homeostasis whereby cytosolic materials are sequestered in a double-membrane autophagosome and subsequently delivered to lysosomes, where they are broken down. In multicellular organisms, newly formed autophagosomes undergo a process called 'maturation', in which they fuse with vesicles originating from endolysosomal compartments, including early/late endosomes and lysosomes, to form amphisomes, which eventually become degradative autolysosomes. This fusion process requires the concerted actions of multiple regulators of membrane dynamics, including SNAREs, tethering proteins and RAB GTPases, and also transport of autophagosomes and late endosomes/lysosomes towards each other. Multiple mechanisms modulate autophagosome maturation, including post-translational modification of key components, spatial distribution of phosphoinositide lipid species on membranes, RAB protein dynamics, and biogenesis and function of lysosomes. Nutrient status and various stresses integrate into the autophagosome maturation machinery to coordinate the progression of autophagic flux. Impaired autophagosome maturation is linked to the pathogenesis of various human diseases, including neurodegenerative disorders, cancer and myopathies. Furthermore, invading pathogens exploit various strategies to block autophagosome maturation, thus evading destruction and even subverting autophagic vacuoles (autophagosomes, amphisomes and autolysosomes) for survival, growth and/or release. Here, we discuss the recent progress in our understanding of the machinery and regulation of autophagosome maturation, the relevance of these mechanisms to human pathophysiology and how they are harnessed by pathogens for their benefit. We also provide perspectives on targeting autophagosome maturation therapeutically.

Identifiants

pubmed: 34302147
doi: 10.1038/s41580-021-00392-4
pii: 10.1038/s41580-021-00392-4
pmc: PMC8300085
doi:

Substances chimiques

SNARE Proteins 0
rab GTP-Binding Proteins EC 3.6.5.2

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

733-750

Informations de copyright

© 2021. Springer Nature Limited.

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Auteurs

Yan G Zhao (YG)

Department of Biology, School of Life Sciences, Southern University of Science and Technology, Shenzhen, People's Republic of China.

Patrice Codogno (P)

Institut Necker-Enfants Malades, INSERM U1151-CNRS UMR 8253, Université de Paris, Paris, France. patrice.codogno@inserm.fr.

Hong Zhang (H)

National Laboratory of Biomacromolecules, CAS Center for Excellence in Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, People's Republic of China. hongzhang@ibp.ac.cn.
College of Life Sciences, University of Chinese Academy of Sciences, Beijing, People's Republic of China. hongzhang@ibp.ac.cn.

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