Renal-limited ANCA-associated vasculitis during erlotinib treatment for lung carcinoma.


Journal

CEN case reports
ISSN: 2192-4449
Titre abrégé: CEN Case Rep
Pays: Japan
ID NLM: 101636244

Informations de publication

Date de publication:
02 2022
Historique:
received: 27 03 2021
accepted: 20 07 2021
pubmed: 27 7 2021
medline: 22 4 2022
entrez: 26 7 2021
Statut: ppublish

Résumé

Epidermal growth factor receptor (EGFR) tyrosine kinase inhibitor (TKI) had clinical success in the treatment of non-small cell lung carcinoma (NSCLC). An effect of this drug on kidney has not been clarified and the occurrence of glomerulonephritis related to EGFR-TKI has rarely been reported. We present the case of a 71-year-old man with NSCLC who developed proteinuria and microscopic hematuria with the rise in a titer of MPO-ANCA, when 2 years and 3 months passed since the initiation of erlotinib, one of oral EGFR-TKI. Two serial biopsies support that ANCA-associated vasculitis may have been modified by the persistent use of erlotinib. We initiated intravenous pulse therapy with methylprednisolone followed by oral prednisone. The proteinuria has decreased and serum CRP was normalized. However, the serum creatinine level and hematuria did not change during the treatment period. While EGFR inhibition is implicated in protective control for glomerulonephritis, it may exacerbate vasculitis. Close monitoring of the kidney function and urinary findings is required during the use of EGFR inhibitors, such as erlotinib, because it may cause renal adverse events.

Identifiants

pubmed: 34309804
doi: 10.1007/s13730-021-00632-8
pii: 10.1007/s13730-021-00632-8
pmc: PMC8811088
doi:

Substances chimiques

Protein Kinase Inhibitors 0
Erlotinib Hydrochloride DA87705X9K
ErbB Receptors EC 2.7.10.1

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

67-72

Informations de copyright

© 2021. Japanese Society of Nephrology.

Références

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Auteurs

Rikako Oki (R)

Division of Nephrology and Endocrinology, The University of Tokyo Graduate School of Medicine, 7-3-1 Hongo, Bunkyo-ku, Tokyo, 113-8655, Japan.

Yosuke Hirakawa (Y)

Division of Nephrology and Endocrinology, The University of Tokyo Graduate School of Medicine, 7-3-1 Hongo, Bunkyo-ku, Tokyo, 113-8655, Japan. yhirakawa@g.ecc.u-tokyo.ac.jp.

Yasuhiro Oda (Y)

Division of Nephrology and Endocrinology, The University of Tokyo Graduate School of Medicine, 7-3-1 Hongo, Bunkyo-ku, Tokyo, 113-8655, Japan.

Motonobu Nakamura (M)

Division of Nephrology and Endocrinology, The University of Tokyo Graduate School of Medicine, 7-3-1 Hongo, Bunkyo-ku, Tokyo, 113-8655, Japan.

Kenjiro Honda (K)

Division of Nephrology and Endocrinology, The University of Tokyo Graduate School of Medicine, 7-3-1 Hongo, Bunkyo-ku, Tokyo, 113-8655, Japan.

Hiroyuki Abe (H)

Department of Pathology, The University of Tokyo Hospital, Tokyo, Japan.

Yukako Domoto (Y)

Department of Pathology, The University of Tokyo Hospital, Tokyo, Japan.

Naoya Miyashita (N)

Department of Respiratory Medicine, Graduate School of Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Takahide Nagase (T)

Department of Respiratory Medicine, Graduate School of Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Masaomi Nangaku (M)

Division of Nephrology and Endocrinology, The University of Tokyo Graduate School of Medicine, 7-3-1 Hongo, Bunkyo-ku, Tokyo, 113-8655, Japan.

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