Downregulation of m
Animals
Cell Line, Tumor
Cell Movement
/ genetics
Cell Proliferation
/ genetics
DNA Copy Number Variations
Deubiquitinating Enzyme CYLD
/ metabolism
Down-Regulation
Gene Expression Regulation, Neoplastic
Humans
Lung Neoplasms
/ genetics
Male
Mice
Mice, Inbred BALB C
Mice, Nude
NF-kappa B p50 Subunit
/ metabolism
RNA Helicases
/ genetics
Signal Transduction
Xenograft Model Antitumor Assays
CYLD
NF-κB pathway
YTHDC2
lung cancer
m6A RNA methylation
Journal
International journal of biological sciences
ISSN: 1449-2288
Titre abrégé: Int J Biol Sci
Pays: Australia
ID NLM: 101235568
Informations de publication
Date de publication:
2021
2021
Historique:
received:
03
02
2021
accepted:
08
06
2021
entrez:
30
7
2021
pubmed:
31
7
2021
medline:
17
3
2022
Statut:
epublish
Résumé
Lung cancer is one of the most common types of carcinoma worldwide. Cigarette smoking is considered the leading cause of lung cancer. Aberrant expression of several YT521-B homology (YTH) family proteins has been reported to be closely associated with multiple cancer types. The present study aims to evaluate the function and regulatory mechanisms of the N6-methyladenosine (m
Identifiants
pubmed: 34326699
doi: 10.7150/ijbs.58514
pii: ijbsv17p2633
pmc: PMC8315025
doi:
Substances chimiques
NF-kappa B p50 Subunit
0
NFKB1 protein, human
0
CYLD protein, human
EC 3.4.19.12
Deubiquitinating Enzyme CYLD
EC 3.4.19.12
RNA Helicases
EC 3.6.4.13
YTHDC2 protein, human
EC 3.6.4.13
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
2633-2651Informations de copyright
© The author(s).
Déclaration de conflit d'intérêts
Competing Interests: The authors have declared that no competing interest exists.
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