Enhanced antitumor immune response in melanoma tumor model by anti-PD-1 small interference RNA encapsulated in nanoliposomes.


Journal

Cancer gene therapy
ISSN: 1476-5500
Titre abrégé: Cancer Gene Ther
Pays: England
ID NLM: 9432230

Informations de publication

Date de publication:
06 2022
Historique:
received: 13 12 2020
accepted: 23 06 2021
revised: 23 05 2021
pubmed: 4 8 2021
medline: 23 6 2022
entrez: 3 8 2021
Statut: ppublish

Résumé

Programmed cell death protein-1 (PD-1), as an immune checkpoint molecule, attenuates T-cell activity and induces T-cell exhaustion. Although siRNA has a great potential in cancer immunotherapy, its delivery to target cells is the main limitation of using siRNA. This study aimed to prepare a liposomal formulation as a siRNA carrier to silence PD-1 expression in T cells and investigate it's in vivo antitumor efficacy. The liposomal siRNA was prepared and characterized by size, zeta potential, and biodistribution. Following that, the uptake assay and mRNA silencing were evaluated in vitro at mRNA and protein levels. siRNA-PD-1 (siPD-1)-loaded liposome nanoparticles were injected into B16F0 tumor-bearing mice to evaluate tumor growth, tumor-infiltrating lymphocytes, and survival rate. Liposomal siPD-1 efficiently silenced PD-1 mRNA expression in T cells (P < 0.0001), and siPD-1-loaded liposomal nanoparticles enhanced the infiltration of T-helper 1 (Th 1) and cytotoxic T lymphocytes into the tumor tissue (P < 0.0001). Liposome-PD-1 siRNA monotherapy and PD-1 siRNA-Doxil (liposomal doxorubicin) combination therapy improved the survival significantly, compared to the control treatment (P < 0.001). Overall, these findings suggest that immunotherapy with siPD-1-loaded liposomes by enhancing T-cell-mediated antitumor immune responses could be considered as a promising strategy for the treatment of melanoma cancer.

Identifiants

pubmed: 34341501
doi: 10.1038/s41417-021-00367-9
pii: 10.1038/s41417-021-00367-9
doi:

Substances chimiques

Liposomes 0
Programmed Cell Death 1 Receptor 0
RNA, Messenger 0
RNA, Small Interfering 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

814-824

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer Nature America, Inc.

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Auteurs

Mehdi Barati (M)

Allergy Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.
Department of Immunology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Farshad Mirzavi (F)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Amin Reza Nikpoor (AR)

Molecular Medicine Research Center, Hormozgan Health Institute, Hormozgan University of Medical Sciences, Bandar Abbas, Iran.

Mojtaba Sankian (M)

Immunobiochemistry Department, Immunology Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Hasan Namdar Ahmadabad (H)

Department of Pathobiology and medical laboratory science, North Khorasan University of Medical Sciences, Bojnurd, Iran.

Anvar Soleimani (A)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Mohammad Mashreghi (M)

Biotechnology Research Center, Pharmaceutical Technology Institute, Mashhad University of Medical Sciences, Mashhad, Iran.

Jalil Tavakol Afshar (J)

Immunology Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Mojgan Mohammadi (M)

Immunology Research Center, Mashhad University of Medical Sciences, Mashhad, Iran. mohammadimzh@mums.ac.ir.

Mahmoud Reza Jaafari (MR)

Biotechnology Research Center, Pharmaceutical Technology Institute, Mashhad University of Medical Sciences, Mashhad, Iran. JafariMR@mums.ac.ir.
Department of Pharmaceutical Nanotechnology, School of Pharmacy, Mashhad University of Medical Sciences, Mashhad, Iran. JafariMR@mums.ac.ir.

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