MAIT cell alterations in adults with recent-onset and long-term type 1 diabetes.


Journal

Diabetologia
ISSN: 1432-0428
Titre abrégé: Diabetologia
Pays: Germany
ID NLM: 0006777

Informations de publication

Date de publication:
10 2021
Historique:
received: 07 12 2020
accepted: 27 04 2021
pubmed: 6 8 2021
medline: 19 3 2022
entrez: 5 8 2021
Statut: ppublish

Résumé

Mucosal-associated invariant T (MAIT) cells are innate-like T lymphocytes expressing an αβ T cell antigen receptor that recognises the MHC-related 1 molecule. MAIT cells are altered in children at risk for and with type 1 diabetes, and mouse model studies have shown MAIT cell involvement in type 1 diabetes development. Since several studies support heterogeneity in type 1 diabetes physiopathology according to the age of individuals, we investigated whether MAIT cells were altered in adults with type 1 diabetes. MAIT cell frequency, phenotype and function were analysed by flow cytometry, using fresh peripheral blood from 21 adults with recent-onset type 1 diabetes (2-14 days after disease onset) and 47 adults with long-term disease (>2 years after diagnosis) compared with 55 healthy blood donors. We also separately analysed 17 women with long-term type 1 diabetes and an associated autoimmune disease, compared with 30 healthy women and 27 women with long-term type 1 diabetes. MAIT cells from adults with recent-onset type 1 diabetes, compared with healthy adult donors, harboured a strongly activated phenotype indicated by an elevated CD25 Alterations in MAIT cell frequency, phenotype and function were more pronounced in adults with long-term type 1 diabetes compared with adults with recent-onset type 1 diabetes. There were several correlations between MAIT cell variables and clinical characteristics. Moreover, the presence of another autoimmune disease in women with long-term type 1 diabetes further exacerbated MAIT cell alterations. Our results suggest that MAIT cell alterations in adults with type 1 diabetes could be associated with two aspects of the disease: impaired glucose homeostasis; and autoimmunity.

Identifiants

pubmed: 34350463
doi: 10.1007/s00125-021-05527-y
pii: 10.1007/s00125-021-05527-y
pmc: PMC8336671
doi:

Substances chimiques

Antigens, CD 0
Antigens, Differentiation, T-Lymphocyte 0
Biomarkers 0
CD69 antigen 0
Cytokines 0
IL2RA protein, human 0
Interleukin-2 Receptor alpha Subunit 0
Lectins, C-Type 0
PDCD1 protein, human 0
Programmed Cell Death 1 Receptor 0
Proto-Oncogene Proteins c-bcl-2 0

Banques de données

figshare
['10.6084/m9.figshare.c.5328767']

Types de publication

Comparative Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2306-2321

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.

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Auteurs

Isabelle Nel (I)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.

Lucie Beaudoin (L)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.

Zouriatou Gouda (Z)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.

Camille Rousseau (C)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.

Pauline Soulard (P)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.

Matthieu Rouland (M)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.

Léo Bertrand (L)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.

Christian Boitard (C)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.
Diabetology Department, Cochin Hospital, AP-HP Centre - Université de Paris, Paris, France.

Etienne Larger (E)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France.
Diabetology Department, Cochin Hospital, AP-HP Centre - Université de Paris, Paris, France.

Agnès Lehuen (A)

Institut Cochin, Inserm, CNRS, Laboratory of Excellence Inflamex, Université de Paris, Paris, France. agnes.lehuen@inserm.fr.

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