Distinct difference in tumor-infiltrating immune cells between Wilms' tumor gene 1 peptide vaccine and anti-programmed cell death-1 antibody therapies.

PD-1 WT1 cancer vaccine combination therapy glioblastoma immunotherapy

Journal

Neuro-oncology advances
ISSN: 2632-2498
Titre abrégé: Neurooncol Adv
Pays: England
ID NLM: 101755003

Informations de publication

Date de publication:
Historique:
entrez: 6 8 2021
pubmed: 7 8 2021
medline: 7 8 2021
Statut: epublish

Résumé

Wilms' tumor gene 1 (WT1) peptide vaccine and anti-programmed cell death-1 (anti-PD-1) antibody are expected as immunotherapies to improve the clinical outcome of glioblastoma. The aims of this study were to clarify how each immunotherapy affects tumor-infiltrating immune cells (TIIs) and to determine whether the combination of these two therapies could synergistically work. Mice were transplanted with WT1 and programmed cell death-ligand 1 doubly expressing glioblastoma cells into brain followed by treatment with WT1 peptide vaccine, anti-PD-1 antibody, or the combination of the two, and survival of each therapy was compared. CD45 Most mice seemed to be cured by the combination therapy with WT1 peptide vaccine and anti-PD-1 antibody, which was much better survival than each monotherapy. A large number of CD4 Our results clearly demonstrated that WT1 peptide vaccine and anti-PD-1 antibody therapies worked in the different steps of cancer-immunity cycle and that the combination of the two therapies could work synergistically against glioblastoma.

Sections du résumé

BACKGROUND BACKGROUND
Wilms' tumor gene 1 (WT1) peptide vaccine and anti-programmed cell death-1 (anti-PD-1) antibody are expected as immunotherapies to improve the clinical outcome of glioblastoma. The aims of this study were to clarify how each immunotherapy affects tumor-infiltrating immune cells (TIIs) and to determine whether the combination of these two therapies could synergistically work.
METHODS METHODS
Mice were transplanted with WT1 and programmed cell death-ligand 1 doubly expressing glioblastoma cells into brain followed by treatment with WT1 peptide vaccine, anti-PD-1 antibody, or the combination of the two, and survival of each therapy was compared. CD45
RESULTS RESULTS
Most mice seemed to be cured by the combination therapy with WT1 peptide vaccine and anti-PD-1 antibody, which was much better survival than each monotherapy. A large number of CD4
CONCLUSION CONCLUSIONS
Our results clearly demonstrated that WT1 peptide vaccine and anti-PD-1 antibody therapies worked in the different steps of cancer-immunity cycle and that the combination of the two therapies could work synergistically against glioblastoma.

Identifiants

pubmed: 34355173
doi: 10.1093/noajnl/vdab091
pii: vdab091
pmc: PMC8331049
doi:

Types de publication

Journal Article

Langues

eng

Pagination

vdab091

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press, the Society for Neuro-Oncology and the European Association of Neuro-Oncology.

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Auteurs

Chisato Yokota (C)

Department of Neurosurgery, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Jun Nakata (J)

Department of Clinical Laboratory and Biomedical Sciences, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Koji Takano (K)

Department of Neurosurgery, Osaka International Cancer Institute, Osaka, Osaka, Japan.

Hiroko Nakajima (H)

Department of Cancer Immunology, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Hiromu Hayashibara (H)

Department of Clinical Laboratory and Biomedical Sciences, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Hikaru Minagawa (H)

Department of Pediatrics, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Yasuyoshi Chiba (Y)

Department of Neurosurgery, Osaka Women's and Children's Hospital, Osaka, Izumi, Japan.

Ryuichi Hirayama (R)

Department of Neurosurgery, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Noriyuki Kijima (N)

Department of Neurosurgery, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Manabu Kinoshita (M)

Department of Neurosurgery, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Yoshiko Hashii (Y)

Department of Cancer Immunotherapy, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Akihiro Tsuboi (A)

Department of Cancer Immunotherapy, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Yoshihiro Oka (Y)

Department of Cancer Stem Cell Biology, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.
Department of Immunopathology, WP1 Immunology Frontier Research Center, Osaka University, Suita, Osaka, Japan.

Yusuke Oji (Y)

Department of Clinical Laboratory and Biomedical Sciences, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Atsushi Kumanogoh (A)

Department of Respiratory Medicine and Clinical Immunology, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.
Department of Immunopathology, WP1 Immunology Frontier Research Center, Osaka University, Suita, Osaka, Japan.

Haruo Sugiyama (H)

Department of Cancer Immunology, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Naoki Kagawa (N)

Department of Neurosurgery, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Haruhiko Kishima (H)

Department of Neurosurgery, Osaka University Graduate School of Medicine, Suita, Osaka, Japan.

Classifications MeSH