Safety and Efficacy of Mepolizumab in Hypereosinophilic Syndrome: An Open-Label Extension Study.


Journal

The journal of allergy and clinical immunology. In practice
ISSN: 2213-2201
Titre abrégé: J Allergy Clin Immunol Pract
Pays: United States
ID NLM: 101597220

Informations de publication

Date de publication:
12 2021
Historique:
received: 25 05 2021
revised: 12 07 2021
accepted: 23 07 2021
pubmed: 15 8 2021
medline: 30 12 2021
entrez: 14 8 2021
Statut: ppublish

Résumé

A double-blind, placebo-controlled, phase III study (200622) showed that mepolizumab reduces disease flares for patients with uncontrolled FIP1-like-1-platelet-derived growth factor receptor α-negative hypereosinophilic syndrome (HES) and two or more flares in the previous year. To further characterize the safety, clinical benefit, and pharmacodynamics of mepolizumab. Eligible patients from both treatment arms of the double-blind study could enter an open-label extension study (205203; NCT03306043) to receive 4-weekly mepolizumab (300 mg subcutaneously) plus background therapy for 20 weeks. Primary end points were safety-based; other end points included flare rates and changes from baseline in mean daily oral corticosteroid (OCS) dose and blood eosinophil count. Of 104 patients who completed the double-blind study, 98% (previous placebo, n = 52; previous mepolizumab, n = 50) enrolled in the open-label extension. Overall, 66 of patients reported adverse events (AEs) (65%), 15 reported treatment-related AEs (15%), and nine reported serious AEs (9%). No events were fatal. The annualized flare rate (95% confidence interval) in the previous placebo and previous mepolizumab groups was 0.37 (0.16-0.86) and 0.14 (0.04-0.49) events/y, respectively. Of 72 patients receiving OCS during weeks 0 to 4, 20 (28%; previous placebo, n = 14; previous mepolizumab, n = 6) achieved 50% or greater reductions in mean daily dose during weeks 16 to 20. At week 20, blood eosinophil count was reduced by 89% in patients previously receiving placebo and remained reduced for those previously receiving mepolizumab. Extended mepolizumab treatment was associated with a positive benefit-risk profile. Continued control of disease flares and blood eosinophil counts, plus reductions in OCS use, were observed with mepolizumab in patients with FIP1-like-1-platelet-derived growth factor receptor α-negative HES.

Sections du résumé

BACKGROUND
A double-blind, placebo-controlled, phase III study (200622) showed that mepolizumab reduces disease flares for patients with uncontrolled FIP1-like-1-platelet-derived growth factor receptor α-negative hypereosinophilic syndrome (HES) and two or more flares in the previous year.
OBJECTIVE
To further characterize the safety, clinical benefit, and pharmacodynamics of mepolizumab.
METHODS
Eligible patients from both treatment arms of the double-blind study could enter an open-label extension study (205203; NCT03306043) to receive 4-weekly mepolizumab (300 mg subcutaneously) plus background therapy for 20 weeks. Primary end points were safety-based; other end points included flare rates and changes from baseline in mean daily oral corticosteroid (OCS) dose and blood eosinophil count.
RESULTS
Of 104 patients who completed the double-blind study, 98% (previous placebo, n = 52; previous mepolizumab, n = 50) enrolled in the open-label extension. Overall, 66 of patients reported adverse events (AEs) (65%), 15 reported treatment-related AEs (15%), and nine reported serious AEs (9%). No events were fatal. The annualized flare rate (95% confidence interval) in the previous placebo and previous mepolizumab groups was 0.37 (0.16-0.86) and 0.14 (0.04-0.49) events/y, respectively. Of 72 patients receiving OCS during weeks 0 to 4, 20 (28%; previous placebo, n = 14; previous mepolizumab, n = 6) achieved 50% or greater reductions in mean daily dose during weeks 16 to 20. At week 20, blood eosinophil count was reduced by 89% in patients previously receiving placebo and remained reduced for those previously receiving mepolizumab.
CONCLUSIONS
Extended mepolizumab treatment was associated with a positive benefit-risk profile. Continued control of disease flares and blood eosinophil counts, plus reductions in OCS use, were observed with mepolizumab in patients with FIP1-like-1-platelet-derived growth factor receptor α-negative HES.

Identifiants

pubmed: 34389506
pii: S2213-2198(21)00891-6
doi: 10.1016/j.jaip.2021.07.050
pii:
doi:

Substances chimiques

Adrenal Cortex Hormones 0
Antibodies, Monoclonal, Humanized 0
mepolizumab 90Z2UF0E52

Banques de données

ClinicalTrials.gov
['NCT03306043', 'NCT02836496']

Types de publication

Clinical Trial, Phase III Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

4431-4440.e1

Investigateurs

Gabriel Ricardo García (GR)
Pablo Pascale (P)
Luis Wehbe (L)
Daniël Blockmans (D)
Florence Roufosse (F)
Martti Anton Antila (MA)
Daniela Blanco (D)
Andreia Luisa Francisco Pez (AL)
Stanislas Faguer (S)
Jean-Emmanuel Kahn (JE)
Guillaume Lefévre (G)
Antoine Neel (A)
Peter M Kern (PM)
Andreas J Reiter (AJ)
Bastian Walz (B)
Tobias Welte (T)
Fabrizio Pane (F)
Alessandro M Vannucchi (AM)
Ruth Cerino-Javier (R)
Dante D Hernández-Colín (DD)
Héctor Glenn Valdéz-López (HG)
Izabela R Kupryś-Lipińska (IR)
Jacek Musial (J)
Eniko Mihaly (E)
Viola Maria Popov (VM)
Vladimir Ivanov (V)
Nikolay Tsyba (N)
Maria C Cid (MC)
Maria Laura Fox (ML)
Guillermo Sanz Santillana (GS)
Andrew J Wardlaw (AJ)
Praveen Akuthota (P)
Joseph H Butterfield (JH)
Geoffrey L Chupp (GL)
Gerald J Gleich (GJ)
Devi Jhaveri (D)
Marc E Rothenberg (ME)

Informations de copyright

Copyright © 2021 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Gerald J Gleich (GJ)

Department of Dermatology, School of Medicine, University of Utah, Salt Lake City, Utah; Department of Medicine, School of Medicine, University of Utah, Salt Lake City, Utah.

Florence Roufosse (F)

Department of Internal Medicine, Hôpital Erasme, Université Libre de Bruxelles, Brussels, Belgium. Electronic address: froufoss@ulb.ac.be.

Geoffrey Chupp (G)

Division of Pulmonary, Critical Care, and Sleep Medicine, Department of Internal Medicine, Yale School of Medicine, New Haven, Conn.

Stanislas Faguer (S)

Department of Nephrology and Transplantation of Organs, CHU de Toulouse, Toulouse, France.

Bastian Walz (B)

Department of Internal Medicine, Rheumatology, and Immunology, Medius Kliniken, University of Tübingen, Kirchheim-Teck, Germany.

Andreas Reiter (A)

Department of Hematology and Oncology, University Medical Centre Mannheim, Heidelberg University, Mannheim, Germany.

Steven W Yancey (SW)

Respiratory Therapeutic Area, GSK, Research Triangle Park, NC.

Jane H Bentley (JH)

Clinical Statistics, GSK, Brentford, Middlesex, UK.

Jonathan Steinfeld (J)

Respiratory Research and Development, GSK, Collegeville, Pa.

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