A Review of Techniques for Biodelivery of Nerve Growth Factor (NGF) to the Brain in Relation to Alzheimer's Disease.

Alzheimer’s disease (AD) Brain Delivery Methods Nerve growth factor (NGF) Techniques

Journal

Advances in experimental medicine and biology
ISSN: 0065-2598
Titre abrégé: Adv Exp Med Biol
Pays: United States
ID NLM: 0121103

Informations de publication

Date de publication:
2021
Historique:
entrez: 28 8 2021
pubmed: 29 8 2021
medline: 1 9 2021
Statut: ppublish

Résumé

Age-dependent progressive neurodegeneration and associated cognitive dysfunction represent a serious concern worldwide. Currently, dementia accounts for the fifth highest cause of death, among which Alzheimer's disease (AD) represents more than 60% of the cases. AD is associated with progressive cognitive dysfunction which affects daily life of the affected individual and associated family. The cognitive dysfunctions are at least partially due to the degeneration of a specific set of neurons (cholinergic neurons) whose cell bodies are situated in the basal forebrain region (basal forebrain cholinergic neurons, BFCNs) but innervate wide areas of the brain. It has been explicitly shown that the delivery of the neurotrophic protein nerve growth factor (NGF) can rescue BFCNs and restore cognitive dysfunction, making NGF interesting as a potential therapeutic substance for AD. Unfortunately, NGF cannot pass through the blood-brain barrier (BBB) and thus peripheral administration of NGF protein is not viable therapeutically. NGF must be delivered in a way which will allow its brain penetration and availability to the BFCNs to modulate BFCN activity and viability. Over the past few decades, various methodologies have been developed to deliver NGF to the brain tissue. In this chapter, NGF delivery methods are discussed in the context of AD.

Identifiants

pubmed: 34453298
doi: 10.1007/978-3-030-74046-7_11
doi:

Substances chimiques

Nerve Growth Factor 9061-61-4

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

167-191

Informations de copyright

© 2021. The Author(s), under exclusive license to Springer Nature Switzerland AG.

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Auteurs

Sumonto Mitra (S)

Division of Clinical Geriatrics, NVS Department, Karolinska Institutet, Stockholm, Sweden. sumonto.mitra@ki.se.

Ruchi Gera (R)

Division of Clinical Geriatrics, NVS Department, Karolinska Institutet, Stockholm, Sweden.

Bengt Linderoth (B)

Section of Neurosurgery, Department of Clinical Neuroscience, Karolinska Institutet, Stockholm, Sweden.

Göran Lind (G)

Section of Neurosurgery, Department of Clinical Neuroscience, Karolinska Institutet, Stockholm, Sweden.

Lars Wahlberg (L)

Gloriana Therapeutics Inc., Warren, RI, USA.

Per Almqvist (P)

Section of Neurosurgery, Department of Clinical Neuroscience, Karolinska Institutet, Stockholm, Sweden.

Homira Behbahani (H)

Division of Clinical Geriatrics, NVS Department, Karolinska Institutet, Stockholm, Sweden.
Karolinska Universitets laboratoriet (LNP5), Karolinska University Hospital, Stockholm, Sweden.

Maria Eriksdotter (M)

Division of Clinical Geriatrics, NVS Department, Karolinska Institutet, Stockholm, Sweden.
Theme Aging, Karolinska University Hospital, Huddinge, Sweden.

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