The contribution of beta-amyloid to dementia in Lewy body diseases: a 1-year follow-up study.
PET/MRI
Parkinson’s disease
amyloid
cognitive dysfunction
dementia with Lewy bodies
Journal
Brain communications
ISSN: 2632-1297
Titre abrégé: Brain Commun
Pays: England
ID NLM: 101755125
Informations de publication
Date de publication:
2021
2021
Historique:
received:
25
05
2021
revised:
08
07
2021
accepted:
13
07
2021
entrez:
30
8
2021
pubmed:
31
8
2021
medline:
31
8
2021
Statut:
epublish
Résumé
Dementia in Lewy Body Diseases (Parkinson's disease and dementia with Lewy Bodies) affects progression of disabilities, quality of life and well-being. Understanding its pathogenetic mechanisms is critical to properly implement disease-modifying strategies. It has been hypothesized that synuclein- and amyloid-pathology act synergistically aggravating cognitive decline in elderly patients but their precise contribution to dementia is debated. In this study, we aimed at exploring if presence of amyloid deposits influences clinical, cognitive and neuroanatomical correlates of mental decline in a cohort of 40 Parkinson's disease patients with normal cognition (
Identifiants
pubmed: 34458730
doi: 10.1093/braincomms/fcab180
pii: fcab180
pmc: PMC8390473
doi:
Types de publication
Journal Article
Langues
eng
Pagination
fcab180Informations de copyright
© The Author(s) (2021). Published by Oxford University Press on behalf of the Guarantors of Brain.
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