Efficacy and safety of dupilumab in patients with uncontrolled severe chronic rhinosinusitis with nasal polyps and a clinical diagnosis of NSAID-ERD: Results from two randomized placebo-controlled phase 3 trials.


Journal

Allergy
ISSN: 1398-9995
Titre abrégé: Allergy
Pays: Denmark
ID NLM: 7804028

Informations de publication

Date de publication:
04 2022
Historique:
received: 22 12 2020
accepted: 12 08 2021
pubmed: 31 8 2021
medline: 22 4 2022
entrez: 30 8 2021
Statut: ppublish

Résumé

About one-tenth of patients with difficult-to-treat chronic rhinosinusitis with nasal polyps (CRSwNP) have comorbid non-steroidal anti-inflammatory drug-exacerbated respiratory disease (NSAID-ERD). Dupilumab, a fully human monoclonal antibody that blocks the shared interleukin (IL)-4/IL-13 receptor component, is an approved add-on treatment in severe CRSwNP. This post hoc analysis evaluated dupilumab efficacy and safety in patients with CRSwNP with/without NSAID-ERD. Data were pooled from the phase 3 SINUS-24 and SINUS-52 studies in adults with uncontrolled severe CRSwNP who received dupilumab 300 mg or placebo every 2 weeks. CRSwNP, nasal airflow, lung function, and asthma control outcomes at Week 24 were evaluated, and treatment-subgroup interactions were assessed for patients with and without NSAID-ERD. Of 724 patients, 204 (28.2%) had a diagnosis of NSAID-ERD. At Week 24, least squares mean treatment differences demonstrated significant improvements in nasal polyp score, nasal congestion (NC), Lund-Mackay computed tomography, 22-item Sinonasal Outcome Test (SNOT-22), Total Symptom Score (TSS), rhinosinusitis severity visual analog scale, peak nasal inspiratory flow (PNIF), six-item Asthma Control Questionnaire score, and improvement in smell with dupilumab versus placebo (all p < .0001) in patients with NSAID-ERD. Treatment comparisons demonstrated significantly greater improvements with dupilumab in patients with versus without NSAID-ERD for NC (p = .0044), SNOT-22 (p = .0313), TSS (p = .0425), and PNIF (p = .0123). In patients with uncontrolled severe CRSwNP, dupilumab significantly improved objective measures and patient-reported symptoms to a greater extent in the presence of comorbid NSAID-ERD than without. Dupilumab was well tolerated in patients with/without NSAID-ERD.

Sections du résumé

BACKGROUND
About one-tenth of patients with difficult-to-treat chronic rhinosinusitis with nasal polyps (CRSwNP) have comorbid non-steroidal anti-inflammatory drug-exacerbated respiratory disease (NSAID-ERD). Dupilumab, a fully human monoclonal antibody that blocks the shared interleukin (IL)-4/IL-13 receptor component, is an approved add-on treatment in severe CRSwNP. This post hoc analysis evaluated dupilumab efficacy and safety in patients with CRSwNP with/without NSAID-ERD.
METHODS
Data were pooled from the phase 3 SINUS-24 and SINUS-52 studies in adults with uncontrolled severe CRSwNP who received dupilumab 300 mg or placebo every 2 weeks. CRSwNP, nasal airflow, lung function, and asthma control outcomes at Week 24 were evaluated, and treatment-subgroup interactions were assessed for patients with and without NSAID-ERD.
RESULTS
Of 724 patients, 204 (28.2%) had a diagnosis of NSAID-ERD. At Week 24, least squares mean treatment differences demonstrated significant improvements in nasal polyp score, nasal congestion (NC), Lund-Mackay computed tomography, 22-item Sinonasal Outcome Test (SNOT-22), Total Symptom Score (TSS), rhinosinusitis severity visual analog scale, peak nasal inspiratory flow (PNIF), six-item Asthma Control Questionnaire score, and improvement in smell with dupilumab versus placebo (all p < .0001) in patients with NSAID-ERD. Treatment comparisons demonstrated significantly greater improvements with dupilumab in patients with versus without NSAID-ERD for NC (p = .0044), SNOT-22 (p = .0313), TSS (p = .0425), and PNIF (p = .0123).
CONCLUSIONS
In patients with uncontrolled severe CRSwNP, dupilumab significantly improved objective measures and patient-reported symptoms to a greater extent in the presence of comorbid NSAID-ERD than without. Dupilumab was well tolerated in patients with/without NSAID-ERD.

Identifiants

pubmed: 34459002
doi: 10.1111/all.15067
pmc: PMC9292324
doi:

Substances chimiques

Anti-Inflammatory Agents, Non-Steroidal 0
Antibodies, Monoclonal, Humanized 0
dupilumab 420K487FSG

Banques de données

ClinicalTrials.gov
['NCT02912468', 'NCT02898454']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1231-1244

Informations de copyright

© 2021 Sanofi Genzyme. Allergy published by European Academy of Allergy and Clinical Immunology and John Wiley & Sons Ltd.

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Auteurs

Joaquim Mullol (J)

Rhinology Unit & Smell Clinic, ENT Department, Hospital Clínic, IDIBAPS, Universitat de Barcelona, CIBERES, Barcelona, Catalonia, Spain.

Tanya M Laidlaw (TM)

Division of Allergy and Clinical Immunology, Brigham and Women's Hospital, and Harvard Medical School, Boston, Massachusetts, USA.

Claus Bachert (C)

Upper Airways Research Laboratory and Department of Otorhinolaryngology, Ghent University, Ghent, Belgium.
Division of ENT Diseases, CLINTEX, Karolinska Institutet, Stockholm, Sweden.
First Affiliated Hosptial, Sun Yat-sen University, Guangzhou, China.

Leda P Mannent (LP)

Global Clinical Development, Sanofi, Chilly-Mazarin, France.

G Walter Canonica (GW)

Department of Biomedical Sciences, Humanitas University, Personalized Medicine Asthma & Allergy Unit-IRCCS Humanitas Research Hospital, Rozzano, Milan, Italy.

Joseph K Han (JK)

Department of Otolaryngology & Head and Neck Surgery, Eastern Virginia Medical School, Norfolk, Virginia, USA.

Jorge F Maspero (JF)

Allergy and Respiratory Medicine, Fundación CIDEA, Buenos Aires, Argentina.

Cesar Picado (C)

Clinical & Experimental Respiratory Immunoallergy, IDIBAPS, Universitat de Barcelona, CIBERES, Barcelona, Catalonia, Spain.

Nadia Daizadeh (N)

Biostatistics, Sanofi, Cambridge, Massachusetts, USA.

Benjamin Ortiz (B)

Immunology and Allergy Medical Affairs, Regeneron Pharmaceuticals, Inc., New York, USA.

Yongtao Li (Y)

Global Medical Affairs Respiratory, Sanofi, Bridgewater, New Jersey, USA.

Marcella Ruddy (M)

Clinical Sciences Global Development, Regeneron Pharmaceuticals, Inc., Tarrytown, New Jersey, USA.

Elizabeth Laws (E)

Immunology and Inflammation, Sanofi, Bridgewater, New Jersey, USA.

Nikhil Amin (N)

Clinical Sciences Global Development, Regeneron Pharmaceuticals, Inc., Tarrytown, New Jersey, USA.

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