Toxicological effects of silver nanoparticles and cadmium chloride in macrophage cell line (RAW 264.7): An in vitro approach.


Journal

Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS)
ISSN: 1878-3252
Titre abrégé: J Trace Elem Med Biol
Pays: Germany
ID NLM: 9508274

Informations de publication

Date de publication:
Dec 2021
Historique:
received: 04 04 2021
revised: 19 08 2021
accepted: 31 08 2021
pubmed: 7 9 2021
medline: 17 2 2022
entrez: 6 9 2021
Statut: ppublish

Résumé

Silver nanoparticles (AgNP) are largely used in nanotechnological products, but the real risks for human and environment are still poorly understood if we consider the effects of mixtures of AgNP and environmental contaminants, such as non-essential metals. The aim of the present study was to investigate the cytotoxicity and toxicological interaction of AgNP (1-4 nm, 0.36 and 3.6 μg mL Effects were observed after a few hours (4 h) on reactive oxygen species (ROS) and became more pronounced after 24 h-exposure. Cell death occurred by apoptosis, and loss of cell viability (24 h-exposure) was preceded by increases of ROS levels and DNA repair foci, but not of NO levels. Co-exposure potentiated some effects (decrease of cell viability and increase of ROS and NO levels), indicating toxicological interaction. These effects are important findings that must be better investigated, since the interaction of Cd with AgNP from nanoproducts may impair the function of macrophages and represent a health risk for humans.

Sections du résumé

BACKGROUND BACKGROUND
Silver nanoparticles (AgNP) are largely used in nanotechnological products, but the real risks for human and environment are still poorly understood if we consider the effects of mixtures of AgNP and environmental contaminants, such as non-essential metals.
METHODS METHODS
The aim of the present study was to investigate the cytotoxicity and toxicological interaction of AgNP (1-4 nm, 0.36 and 3.6 μg mL
RESULTS RESULTS
Effects were observed after a few hours (4 h) on reactive oxygen species (ROS) and became more pronounced after 24 h-exposure. Cell death occurred by apoptosis, and loss of cell viability (24 h-exposure) was preceded by increases of ROS levels and DNA repair foci, but not of NO levels. Co-exposure potentiated some effects (decrease of cell viability and increase of ROS and NO levels), indicating toxicological interaction.
CONCLUSION CONCLUSIONS
These effects are important findings that must be better investigated, since the interaction of Cd with AgNP from nanoproducts may impair the function of macrophages and represent a health risk for humans.

Identifiants

pubmed: 34488184
pii: S0946-672X(21)00144-9
doi: 10.1016/j.jtemb.2021.126854
pii:
doi:

Substances chimiques

Reactive Oxygen Species 0
Cadmium 00BH33GNGH
Silver 3M4G523W1G
Cadmium Chloride J6K4F9V3BA

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

126854

Informations de copyright

Copyright © 2021 Elsevier GmbH. All rights reserved.

Auteurs

Andressa Glinski (A)

Laboratório de Toxicologia Celular, Departamento de Biologia Celular, Universidade Federal do Paraná, CEP 81.531-980, Curitiba, PR, Brazil.

Tugstênio Lima de Souza (T)

Laboratório de Toxicologia Celular, Departamento de Biologia Celular, Universidade Federal do Paraná, CEP 81.531-980, Curitiba, PR, Brazil.

Jessica Zablocki da Luz (J)

Laboratório de Toxicologia Celular, Departamento de Biologia Celular, Universidade Federal do Paraná, CEP 81.531-980, Curitiba, PR, Brazil.

Arandi Ginane Bezerra Junior (AG)

Laboratório Fotonanobio, Departamento Acadêmico de Física, Universidade Tecnológica Federal do Paraná, CEP 80.230-901, Curitiba, PR, Brazil.

Carolina Camargo de Oliveira (C)

Laboratório de Células Inflamatórias e Neoplásicas, Departamento de Biologia Celular, Universidade Federal do Paraná, CEP 81.531-980, Curitiba, PR, Brazil.

Ciro Alberto de Oliveira Ribeiro (CA)

Laboratório de Toxicologia Celular, Departamento de Biologia Celular, Universidade Federal do Paraná, CEP 81.531-980, Curitiba, PR, Brazil.

Francisco Filipak Neto (F)

Laboratório de Toxicologia Celular, Departamento de Biologia Celular, Universidade Federal do Paraná, CEP 81.531-980, Curitiba, PR, Brazil. Electronic address: filipak@ufpr.br.

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Classifications MeSH