Attenuated infection by a Pteropine orthoreovirus isolated from an Egyptian fruit bat in Zambia.


Journal

PLoS neglected tropical diseases
ISSN: 1935-2735
Titre abrégé: PLoS Negl Trop Dis
Pays: United States
ID NLM: 101291488

Informations de publication

Date de publication:
09 2021
Historique:
received: 29 03 2021
accepted: 27 08 2021
revised: 17 09 2021
pubmed: 8 9 2021
medline: 15 12 2021
entrez: 7 9 2021
Statut: epublish

Résumé

Pteropine orthoreovirus (PRV) is an emerging bat-borne zoonotic virus that causes severe respiratory illness in humans. Although PRVs have been identified in fruit bats and humans in Australia and Asia, little is known about the prevalence of PRV infection in Africa. Therefore, this study performed an PRV surveillance in fruit bats in Zambia. Egyptian fruit bats (Rousettus aegyptiacus, n = 47) and straw-colored fruit bats (Eidolon helvum, n = 33) captured in Zambia in 2017-2018 were screened for PRV infection using RT-PCR and serum neutralization tests. The complete genome sequence of an isolated PRV strain was determined by next generation sequencing and subjected to BLAST and phylogenetic analyses. Replication capacity and pathogenicity of the strain were investigated using Vero E6 cell cultures and BALB/c mice, respectively. An PRV strain, tentatively named Nachunsulwe-57, was isolated from one Egyptian fruit bat. Serological assays demonstrated that 98% of sera (69/70) collected from Egyptian fruit bats (n = 37) and straw-colored fruit bats (n = 33) had neutralizing antibodies against PRV. Genetic analyses revealed that all 10 genome segments of Nachunsulwe-57 were closely related to a bat-derived Kasama strain found in Uganda. Nachunsulwe-57 showed less efficiency in viral growth and lower pathogenicity in mice than another PRV strain, Miyazaki-Bali/2007, isolated from a patient. A high proportion of Egyptian fruit bats and straw-colored fruit bats were found to be seropositive to PRV in Zambia. Importantly, a new PRV strain (Nachunsulwe-57) was isolated from an Egyptian fruit bat in Zambia, which had relatively weak pathogenicity in mice. Taken together, our findings provide new epidemiological insights about PRV infection in bats and indicate the first isolation of an PRV strain that may have low pathogenicity to humans.

Sections du résumé

BACKGROUND
Pteropine orthoreovirus (PRV) is an emerging bat-borne zoonotic virus that causes severe respiratory illness in humans. Although PRVs have been identified in fruit bats and humans in Australia and Asia, little is known about the prevalence of PRV infection in Africa. Therefore, this study performed an PRV surveillance in fruit bats in Zambia.
METHODS
Egyptian fruit bats (Rousettus aegyptiacus, n = 47) and straw-colored fruit bats (Eidolon helvum, n = 33) captured in Zambia in 2017-2018 were screened for PRV infection using RT-PCR and serum neutralization tests. The complete genome sequence of an isolated PRV strain was determined by next generation sequencing and subjected to BLAST and phylogenetic analyses. Replication capacity and pathogenicity of the strain were investigated using Vero E6 cell cultures and BALB/c mice, respectively.
RESULTS
An PRV strain, tentatively named Nachunsulwe-57, was isolated from one Egyptian fruit bat. Serological assays demonstrated that 98% of sera (69/70) collected from Egyptian fruit bats (n = 37) and straw-colored fruit bats (n = 33) had neutralizing antibodies against PRV. Genetic analyses revealed that all 10 genome segments of Nachunsulwe-57 were closely related to a bat-derived Kasama strain found in Uganda. Nachunsulwe-57 showed less efficiency in viral growth and lower pathogenicity in mice than another PRV strain, Miyazaki-Bali/2007, isolated from a patient.
CONCLUSIONS
A high proportion of Egyptian fruit bats and straw-colored fruit bats were found to be seropositive to PRV in Zambia. Importantly, a new PRV strain (Nachunsulwe-57) was isolated from an Egyptian fruit bat in Zambia, which had relatively weak pathogenicity in mice. Taken together, our findings provide new epidemiological insights about PRV infection in bats and indicate the first isolation of an PRV strain that may have low pathogenicity to humans.

Identifiants

pubmed: 34492038
doi: 10.1371/journal.pntd.0009768
pii: PNTD-D-21-00437
pmc: PMC8448348
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0009768

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Hayato Harima (H)

Hokudai Center for Zoonosis Control in Zambia, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Michihito Sasaki (M)

Division of Molecular Pathobiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Yasuko Orba (Y)

Division of Molecular Pathobiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
International Collaboration Unit, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Kosuke Okuya (K)

Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Yongjin Qiu (Y)

Hokudai Center for Zoonosis Control in Zambia, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Christida E Wastika (CE)

Division of Molecular Pathobiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Katendi Changula (K)

Department of Para-clinical Studies, School of Veterinary Medicine, the University of Zambia, Lusaka, Zambia.

Masahiro Kajihara (M)

Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Edgar Simulundu (E)

Department of Disease Control, School of Veterinary Medicine, the University of Zambia, Lusaka, Zambia.
Macha Research Trust, Choma, Zambia.

Tomoyuki Yamaguchi (T)

Division of Bioresources, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Yoshiki Eto (Y)

Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Akina Mori-Kajihara (A)

Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.

Akihiko Sato (A)

Division of Molecular Pathobiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Drug Discovery & Disease Research Laboratory, Shionogi & Co., Ltd., Osaka, Japan.

Satoshi Taniguchi (S)

Department of Virology I, National Institute of Infectious Diseases, Shinjuku, Tokyo, Japan.

Ayato Takada (A)

International Collaboration Unit, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Division of Global Epidemiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Department of Disease Control, School of Veterinary Medicine, the University of Zambia, Lusaka, Zambia.
Africa Center of Excellence for Infectious Diseases of Humans and Animals, the University of Zambia, Lusaka, Zambia.

Masayuki Saijo (M)

Department of Virology I, National Institute of Infectious Diseases, Shinjuku, Tokyo, Japan.

Bernard M Hang'ombe (BM)

Department of Para-clinical Studies, School of Veterinary Medicine, the University of Zambia, Lusaka, Zambia.
Africa Center of Excellence for Infectious Diseases of Humans and Animals, the University of Zambia, Lusaka, Zambia.

Hirofumi Sawa (H)

Division of Molecular Pathobiology, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
International Collaboration Unit, Research Center for Zoonosis Control, Hokkaido University, Sapporo, Japan.
Department of Disease Control, School of Veterinary Medicine, the University of Zambia, Lusaka, Zambia.
Africa Center of Excellence for Infectious Diseases of Humans and Animals, the University of Zambia, Lusaka, Zambia.
Global Virus Network, Baltimore, Maryland, United States of America.
One Health Research Center, Hokkaido University, Sapporo, Japan.

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