The circadian clock component BMAL1 regulates SARS-CoV-2 entry and replication in lung epithelial cells.
Microbiology
Molecular biology
Transcriptomics
Virology
Journal
iScience
ISSN: 2589-0042
Titre abrégé: iScience
Pays: United States
ID NLM: 101724038
Informations de publication
Date de publication:
22 Oct 2021
22 Oct 2021
Historique:
received:
06
01
2021
revised:
11
09
2021
accepted:
13
09
2021
pubmed:
22
9
2021
medline:
22
9
2021
entrez:
21
9
2021
Statut:
ppublish
Résumé
The coronavirus disease 2019 pandemic, caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) coronavirus, is a global health issue with unprecedented challenges for public health. SARS-CoV-2 primarily infects cells of the respiratory tract via spike glycoprotein binding to angiotensin-converting enzyme (ACE2). Circadian rhythms coordinate an organism's response to its environment and can regulate host susceptibility to virus infection. We demonstrate that silencing the circadian regulator
Identifiants
pubmed: 34545347
doi: 10.1016/j.isci.2021.103144
pii: S2589-0042(21)01112-3
pmc: PMC8443536
doi:
Types de publication
Journal Article
Langues
eng
Pagination
103144Subventions
Organisme : Wellcome Trust
ID : 211050/Z/18/Z
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 200838/Z/16/Z
Pays : United Kingdom
Organisme : Biotechnology and Biological Sciences Research Council
ID : BBS/E/I/00007034
Pays : United Kingdom
Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Biotechnology and Biological Sciences Research Council
ID : BBS/E/I/COV07001
Pays : United Kingdom
Commentaires et corrections
Type : UpdateOf
Informations de copyright
© 2021 The Authors.
Déclaration de conflit d'intérêts
The other authors declare no financial interests.
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