A molecular sensor determines the ubiquitin substrate specificity of SARS-CoV-2 papain-like protease.


Journal

Cell reports
ISSN: 2211-1247
Titre abrégé: Cell Rep
Pays: United States
ID NLM: 101573691

Informations de publication

Date de publication:
28 09 2021
Historique:
received: 26 02 2021
revised: 15 07 2021
accepted: 02 09 2021
pubmed: 22 9 2021
medline: 13 10 2021
entrez: 21 9 2021
Statut: ppublish

Résumé

The SARS-CoV-2 papain-like protease (PLpro) is a target for antiviral drug development. It is essential for processing viral polyproteins for replication and functions in host immune evasion by cleaving ubiquitin (Ub) and ubiquitin-like protein (Ubl) conjugates. While highly conserved, SARS-CoV-2 and SARS-CoV PLpro have contrasting Ub/Ubl substrate preferences. Using a combination of structural analyses and functional assays, we identify a molecular sensor within the S1 Ub-binding site of PLpro that serves as a key determinant of substrate specificity. Variations within the S1 sensor specifically alter cleavage of Ub substrates but not of the Ubl interferon-stimulated gene 15 protein (ISG15). Significantly, a variant of concern associated with immune evasion carries a mutation in the S1 sensor that enhances PLpro activity on Ub substrates. Collectively, our data identify the S1 sensor region as a potential hotspot of variability that could alter host antiviral immune responses to newly emerging SARS-CoV-2 lineages.

Identifiants

pubmed: 34547223
pii: S2211-1247(21)01208-0
doi: 10.1016/j.celrep.2021.109754
pmc: PMC8423903
pii:
doi:

Substances chimiques

Ubiquitin 0
Ubiquitins 0
Viral Proteins 0
Peptide Hydrolases EC 3.4.-
Coronavirus Papain-Like Proteases EC 3.4.22.2
Papain EC 3.4.22.2
papain-like protease, SARS-CoV-2 EC 3.4.22.2

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

109754

Subventions

Organisme : NIGMS NIH HHS
ID : R35 GM139610
Pays : United States
Organisme : NIGMS NIH HHS
ID : P30 GM124165
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM128731
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA054174
Pays : United States
Organisme : NIEHS NIH HHS
ID : R01 ES025166
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM115568
Pays : United States
Organisme : NIH HHS
ID : S10 OD021527
Pays : United States

Informations de copyright

Copyright © 2021 The Author(s). Published by Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests M.B. is an employee of Arvinas, Inc, which was not involved in this study. T.T.H. is a member of the advisory board for Cell Reports. The remaining authors declare no competing interests.

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Auteurs

Stephanie Patchett (S)

Department of Biochemistry & Molecular Pharmacology, New York University School of Medicine, New York, NY 10016, USA.

Zongyang Lv (Z)

Department of Biochemistry & Structural Biology, University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA.

Wioletta Rut (W)

Department of Chemical Biology and Bioimaging, Wroclaw University of Science and Technology, Wyb. Wyspianskiego 27, 50-370 Wroclaw, Poland.

Miklos Békés (M)

Department of Biochemistry & Molecular Pharmacology, New York University School of Medicine, New York, NY 10016, USA.

Marcin Drag (M)

Department of Chemical Biology and Bioimaging, Wroclaw University of Science and Technology, Wyb. Wyspianskiego 27, 50-370 Wroclaw, Poland.

Shaun K Olsen (SK)

Department of Biochemistry & Structural Biology, University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, USA. Electronic address: olsens@uthscsa.edu.

Tony T Huang (TT)

Department of Biochemistry & Molecular Pharmacology, New York University School of Medicine, New York, NY 10016, USA. Electronic address: tony.huang@nyumc.org.

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Classifications MeSH