Inositol serves as a natural inhibitor of mitochondrial fission by directly targeting AMPK.


Journal

Molecular cell
ISSN: 1097-4164
Titre abrégé: Mol Cell
Pays: United States
ID NLM: 9802571

Informations de publication

Date de publication:
16 09 2021
Historique:
received: 01 11 2020
revised: 01 06 2021
accepted: 19 08 2021
entrez: 21 9 2021
pubmed: 22 9 2021
medline: 16 10 2021
Statut: ppublish

Résumé

Mitochondrial dynamics regulated by mitochondrial fusion and fission maintain mitochondrial functions, whose alterations underline various human diseases. Here, we show that inositol is a critical metabolite directly restricting AMPK-dependent mitochondrial fission independently of its classical mode as a precursor for phosphoinositide generation. Inositol decline by IMPA1/2 deficiency elicits AMPK activation and mitochondrial fission without affecting ATP level, whereas inositol accumulation prevents AMPK-dependent mitochondrial fission. Metabolic stress or mitochondrial damage causes inositol decline in cells and mice to elicit AMPK-dependent mitochondrial fission. Inositol directly binds to AMPKγ and competes with AMP for AMPKγ binding, leading to restriction of AMPK activation and mitochondrial fission. Our study suggests that the AMP/inositol ratio is a critical determinant for AMPK activation and establishes a model in which AMPK activation requires inositol decline to release AMPKγ for AMP binding. Hence, AMPK is an inositol sensor, whose inactivation by inositol serves as a mechanism to restrict mitochondrial fission.

Identifiants

pubmed: 34547240
pii: S1097-2765(21)00692-4
doi: 10.1016/j.molcel.2021.08.025
pmc: PMC8462099
mid: NIHMS1737151
pii:
doi:

Substances chimiques

Inositol 4L6452S749
AMP-Activated Protein Kinases EC 2.7.11.31
Phosphoric Monoester Hydrolases EC 3.1.3.2
myo-inositol-1 (or 4)-monophosphatase EC 3.1.3.25

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3803-3819.e7

Subventions

Organisme : NCI NIH HHS
ID : P30 CA012197
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA182424
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA193813
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA248037
Pays : United States

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests The authors declare no competing interests.

Références

J Neurosci Methods. 2016 Mar 15;262:56-65
pubmed: 26777473
Cell Metab. 2013 Oct 1;18(4):556-66
pubmed: 24093679
Exp Eye Res. 2011 Oct;93(4):460-74
pubmed: 21722635
J Biol Chem. 1980 Dec 25;255(24):11710-2
pubmed: 7002927
Hepatology. 2011 Aug;54(2):452-62
pubmed: 21488074
Eur Rev Med Pharmacol Sci. 2011 Feb;15(2):129-34
pubmed: 21434479
Biochem Soc Trans. 2009 Oct;37(Pt 5):1099-103
pubmed: 19754460
Proc Natl Acad Sci U S A. 1993 Jul 1;90(13):5924-8
pubmed: 8392181
Science. 2012 Aug 10;337(6095):653-4
pubmed: 22879491
Eur J Obstet Gynecol Reprod Biol. 2011 Dec;159(2):267-72
pubmed: 21835536
Nat Rev Drug Discov. 2019 Jul;18(7):527-551
pubmed: 30867601
Dementia. 1993 Mar-Apr;4(2):81-6
pubmed: 8395284
J Biol Chem. 1967 Apr 10;242(7):1375-82
pubmed: 4290245
Biochim Biophys Acta. 2008 Sep;1777(9):1092-7
pubmed: 18519024
Gynecol Endocrinol. 2012 Jul;28(7):509-15
pubmed: 22296306
Science. 2012 Aug 10;337(6095):727-30
pubmed: 22722250
J Cell Biol. 2009 Dec 28;187(7):959-66
pubmed: 20038677
Annu Rev Nutr. 1986;6:563-97
pubmed: 2425833
J Clin Invest. 1995 Feb;95(2):542-6
pubmed: 7860736
Neuropsychopharmacology. 2008 Feb;33(3):674-84
pubmed: 17460611
Science. 2012 Aug 31;337(6098):1062-5
pubmed: 22936770
Cancer Cell. 2015 Feb 9;27(2):271-85
pubmed: 25640402
Cell. 2016 Jul 28;166(3):555-566
pubmed: 27471965
J Proteome Res. 2020 Jun 5;19(6):2367-2378
pubmed: 32397718
EMBO Rep. 2007 Oct;8(10):939-44
pubmed: 17721437
Cell. 2010 Jul 23;142(2):270-83
pubmed: 20655468
J Cell Biol. 2014 Feb 17;204(4):477-86
pubmed: 24515348
Science. 2016 Jan 15;351(6270):275-281
pubmed: 26816379
Nature. 2007 Sep 27;449(7161):496-500
pubmed: 17851531
Cell Res. 2018 Mar;28(3):265-280
pubmed: 29219147
J Cell Biol. 2010 Dec 13;191(6):1141-58
pubmed: 21149567
Genes Dev. 2011 Sep 15;25(18):1895-908
pubmed: 21937710
Mol Biol Cell. 2013 Mar;24(5):659-67
pubmed: 23283981
Semin Cancer Biol. 2021 Apr 17;:
pubmed: 33862221
ACS Omega. 2018 Oct 31;3(10):13876-13881
pubmed: 30411052
J Biol Chem. 2007 Jul 27;282(30):21583-7
pubmed: 17553808
Cell. 2019 Jun 27;178(1):176-189.e15
pubmed: 31155231
J Biol Chem. 2003 Mar 21;278(12):10602-12
pubmed: 12525476
Biochim Biophys Acta. 1997 Sep 4;1348(1-2):245-56
pubmed: 9370339
Cell Metab. 2012 Jul 3;16(1):9-17
pubmed: 22768835
Clin Cancer Res. 2019 Nov 1;25(21):6452-6462
pubmed: 31533931
J Cell Biol. 2016 Feb 15;212(4):379-87
pubmed: 26858267
Eur Rev Med Pharmacol Sci. 2011 May;15(5):509-14
pubmed: 21744744
Neuropsychopharmacology. 2014 Jan;39(2):319-28
pubmed: 23924600
Nat Cell Biol. 2011 Sep 02;13(9):1016-23
pubmed: 21892142
J Neurosci. 2011 Nov 2;31(44):15716-26
pubmed: 22049414
Cancer Lett. 2019 Oct 28;463:11-26
pubmed: 31404613
Mol Cell. 2015 Feb 5;57(3):537-51
pubmed: 25658205
J Biol Chem. 2014 Feb 28;289(9):6028-40
pubmed: 24429282
Nature. 2006 Oct 12;443(7112):651-7
pubmed: 17035995
Mol Psychiatry. 2016 Aug;21(8):1125-9
pubmed: 26416544
Trends Biochem Sci. 1992 Oct;17(10):414-7
pubmed: 1455509
J Biol Chem. 2014 Apr 11;289(15):10785-10796
pubmed: 24554717
Proc Natl Acad Sci U S A. 1999 Jul 20;96(15):8745-50
pubmed: 10411946
Cell Signal. 2015 May;27(5):978-88
pubmed: 25683918
Nat Rev Mol Cell Biol. 2005 Aug;6(8):657-63
pubmed: 16025099
Science. 1994 Aug 5;265(5173):813-5
pubmed: 8047889
Nature. 2015 Feb 19;518(7539):413-6
pubmed: 25487152
J Biol Chem. 2009 May 15;284(20):13843-13855
pubmed: 19279012
Nat Protoc. 2013 Nov;8(11):2281-2308
pubmed: 24157548
EMBO J. 2006 Jul 12;25(13):2966-77
pubmed: 16778770
N Engl J Med. 2013 Dec 5;369(23):2236-51
pubmed: 24304053
Mol Cell. 2005 Apr 29;18(3):283-93
pubmed: 15866171
Antioxid Redox Signal. 2011 May 15;14(10):1939-51
pubmed: 21128700
Annu Rev Biophys Biomol Struct. 2002;31:151-75
pubmed: 11988466
Cell. 2013 Sep 26;155(1):160-71
pubmed: 24055366

Auteurs

Che-Chia Hsu (CC)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Xian Zhang (X)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Guihua Wang (G)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Weina Zhang (W)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Zhen Cai (Z)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Bo-Syong Pan (BS)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Haiwei Gu (H)

Center for Metabolic and Vascular Biology, School of Nutrition and Health Promotion, College of Health Solutions, Arizona State University, Phoenix, AZ 85004, USA.

Chuan Xu (C)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Guoxiang Jin (G)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Xiangshang Xu (X)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Rajesh Kumar Manne (RK)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Yan Jin (Y)

Center for Metabolic and Vascular Biology, School of Nutrition and Health Promotion, College of Health Solutions, Arizona State University, Phoenix, AZ 85004, USA.

Wei Yan (W)

University of Arkansas for Medical Sciences, College of Pharmacy, Division of Pharmaceutical Science, 200 South Cedar, Little Rock, AR 72202, USA.

Jingwei Shao (J)

University of Arkansas for Medical Sciences, College of Pharmacy, Division of Pharmaceutical Science, 200 South Cedar, Little Rock, AR 72202, USA.

Tingjin Chen (T)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA.

Emily Lin (E)

Atkins High School, Winston-Salem, NC 27101, USA.

Amit Ketkar (A)

Department of Biochemistry and Molecular Biology, University of Arkansas for Medical Sciences, Little Rock, AR 72202, USA.

Robert Eoff (R)

Department of Biochemistry and Molecular Biology, University of Arkansas for Medical Sciences, Little Rock, AR 72202, USA.

Zhi-Gang Xu (ZG)

Chongqing Engineering Laboratory of Targeted and Innovative Therapeutics, Chongqing Key Laboratory of Kinase Modulators as Innovative Medicine, IATTI, Chongqing University of Arts and Sciences, Yongchuan, Chongqing 402160, China.

Zhong-Zhu Chen (ZZ)

Chongqing Engineering Laboratory of Targeted and Innovative Therapeutics, Chongqing Key Laboratory of Kinase Modulators as Innovative Medicine, IATTI, Chongqing University of Arts and Sciences, Yongchuan, Chongqing 402160, China.

Hong-Yu Li (HY)

University of Arkansas for Medical Sciences, College of Pharmacy, Division of Pharmaceutical Science, 200 South Cedar, Little Rock, AR 72202, USA.

Hui-Kuan Lin (HK)

Department of Cancer Biology, Wake Forest Baptist Medical Center, Wake Forest University, Winston-Salem, NC 27101, USA. Electronic address: hulin@wakehealth.edu.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH